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336 related items for PubMed ID: 27006390
1. Disturbed sialic acid recognition on endothelial cells and platelets in complement attack causes atypical hemolytic uremic syndrome. Hyvärinen S, Meri S, Jokiranta TS. Blood; 2016 Jun 02; 127(22):2701-10. PubMed ID: 27006390 [Abstract] [Full Text] [Related]
2. Mutations of factor H impair regulation of surface-bound C3b by three mechanisms in atypical hemolytic uremic syndrome. Lehtinen MJ, Rops AL, Isenman DE, van der Vlag J, Jokiranta TS. J Biol Chem; 2009 Jun 05; 284(23):15650-8. PubMed ID: 19351878 [Abstract] [Full Text] [Related]
3. Deregulation of Factor H by Factor H-Related Protein 1 Depends on Sialylation of Host Surfaces. Dopler A, Stibitzky S, Hevey R, Mannes M, Guariento M, Höchsmann B, Schrezenmeier H, Ricklin D, Schmidt CQ. Front Immunol; 2021 Jun 05; 12():615748. PubMed ID: 33732239 [Abstract] [Full Text] [Related]
4. Recognition of malondialdehyde-modified proteins by the C terminus of complement factor H is mediated via the polyanion binding site and impaired by mutations found in atypical hemolytic uremic syndrome. Hyvärinen S, Uchida K, Varjosalo M, Jokela R, Jokiranta TS. J Biol Chem; 2014 Feb 14; 289(7):4295-306. PubMed ID: 24344133 [Abstract] [Full Text] [Related]
6. Mutations in Complement Factor H Impair Alternative Pathway Regulation on Mouse Glomerular Endothelial Cells in Vitro. Loeven MA, Rops AL, Lehtinen MJ, van Kuppevelt TH, Daha MR, Smith RJ, Bakker M, Berden JH, Rabelink TJ, Jokiranta TS, van der Vlag J. J Biol Chem; 2016 Mar 04; 291(10):4974-81. PubMed ID: 26728463 [Abstract] [Full Text] [Related]
7. Both domain 19 and domain 20 of factor H are involved in binding to complement C3b and C3d. Bhattacharjee A, Lehtinen MJ, Kajander T, Goldman A, Jokiranta TS. Mol Immunol; 2010 May 04; 47(9):1686-91. PubMed ID: 20378178 [Abstract] [Full Text] [Related]
9. Structure of complement factor H carboxyl-terminus reveals molecular basis of atypical haemolytic uremic syndrome. Jokiranta TS, Jaakola VP, Lehtinen MJ, Pärepalo M, Meri S, Goldman A. EMBO J; 2006 Apr 19; 25(8):1784-94. PubMed ID: 16601698 [Abstract] [Full Text] [Related]
10. Comparison of surface recognition and C3b binding properties of mouse and human complement factor H. Cheng ZZ, Hellwage J, Seeberger H, Zipfel PF, Meri S, Jokiranta TS. Mol Immunol; 2006 Mar 19; 43(7):972-9. PubMed ID: 16023208 [Abstract] [Full Text] [Related]
11. The binding of factor H to a complex of physiological polyanions and C3b on cells is impaired in atypical hemolytic uremic syndrome. Ferreira VP, Herbert AP, Cortés C, McKee KA, Blaum BS, Esswein ST, Uhrín D, Barlow PN, Pangburn MK, Kavanagh D. J Immunol; 2009 Jun 01; 182(11):7009-18. PubMed ID: 19454698 [Abstract] [Full Text] [Related]
14. Unraveling the Effect of a Potentiating Anti-Factor H Antibody on Atypical Hemolytic Uremic Syndrome-Associated Factor H Variants. Dekkers G, Brouwer MC, Jeremiasse J, Kamp A, Biggs RM, van Mierlo G, Lauder S, Katti S, Kuijpers TW, Rispens T, Jongerius I. J Immunol; 2020 Oct 01; 205(7):1778-1786. PubMed ID: 32848031 [Abstract] [Full Text] [Related]
15. Factor H Competitor Generated by Gene Conversion Events Associates with Atypical Hemolytic Uremic Syndrome. Goicoechea de Jorge E, Tortajada A, García SP, Gastoldi S, Merinero HM, García-Fernández J, Arjona E, Cao M, Remuzzi G, Noris M, Rodríguez de Córdoba S. J Am Soc Nephrol; 2018 Jan 01; 29(1):240-249. PubMed ID: 28993505 [Abstract] [Full Text] [Related]
16. Selective Binding of Heparin/Heparan Sulfate Oligosaccharides to Factor H and Factor H-Related Proteins: Therapeutic Potential for C3 Glomerulopathies. Loeven MA, Maciej-Hulme ML, Yanginlar C, Hubers MC, Kellenbach E, de Graaf M, van Kuppevelt TH, Wetzels J, Rabelink TJ, Smith RJH, van der Vlag J. Front Immunol; 2021 Jan 01; 12():676662. PubMed ID: 34489931 [Abstract] [Full Text] [Related]
17. Structural basis for sialic acid-mediated self-recognition by complement factor H. Blaum BS, Hannan JP, Herbert AP, Kavanagh D, Uhrín D, Stehle T. Nat Chem Biol; 2015 Jan 01; 11(1):77-82. PubMed ID: 25402769 [Abstract] [Full Text] [Related]
18. The major autoantibody epitope on factor H in atypical hemolytic uremic syndrome is structurally different from its homologous site in factor H-related protein 1, supporting a novel model for induction of autoimmunity in this disease. Bhattacharjee A, Reuter S, Trojnár E, Kolodziejczyk R, Seeberger H, Hyvärinen S, Uzonyi B, Szilágyi Á, Prohászka Z, Goldman A, Józsi M, Jokiranta TS. J Biol Chem; 2015 Apr 10; 290(15):9500-10. PubMed ID: 25659429 [Abstract] [Full Text] [Related]
19. Dual interaction of factor H with C3d and glycosaminoglycans in host-nonhost discrimination by complement. Kajander T, Lehtinen MJ, Hyvärinen S, Bhattacharjee A, Leung E, Isenman DE, Meri S, Goldman A, Jokiranta TS. Proc Natl Acad Sci U S A; 2011 Feb 15; 108(7):2897-902. PubMed ID: 21285368 [Abstract] [Full Text] [Related]
20. Factor H dysfunction in patients with atypical hemolytic uremic syndrome contributes to complement deposition on platelets and their activation. Ståhl AL, Vaziri-Sani F, Heinen S, Kristoffersson AC, Gydell KH, Raafat R, Gutierrez A, Beringer O, Zipfel PF, Karpman D. Blood; 2008 Jun 01; 111(11):5307-15. PubMed ID: 18268093 [Abstract] [Full Text] [Related] Page: [Next] [New Search]