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Journal Abstract Search

1449 related items for PubMed ID: 27020329

  • 1. Rifampicin is a candidate preventive medicine against amyloid-β and tau oligomers.
    Umeda T, Ono K, Sakai A, Yamashita M, Mizuguchi M, Klein WL, Yamada M, Mori H, Tomiyama T.
    Brain; 2016 May; 139(Pt 5):1568-86. PubMed ID: 27020329
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  • 2. Neurofibrillary tangle formation by introducing wild-type human tau into APP transgenic mice.
    Umeda T, Maekawa S, Kimura T, Takashima A, Tomiyama T, Mori H.
    Acta Neuropathol; 2014 May; 127(5):685-98. PubMed ID: 24531886
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  • 6. Pyruvate prevents the development of age-dependent cognitive deficits in a mouse model of Alzheimer's disease without reducing amyloid and tau pathology.
    Isopi E, Granzotto A, Corona C, Bomba M, Ciavardelli D, Curcio M, Canzoniero LM, Navarra R, Lattanzio R, Piantelli M, Sensi SL.
    Neurobiol Dis; 2015 Sep; 81():214-24. PubMed ID: 25434488
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  • 7. Tau passive immunization inhibits not only tau but also Aβ pathology.
    Dai CL, Tung YC, Liu F, Gong CX, Iqbal K.
    Alzheimers Res Ther; 2017 Jan 10; 9(1):1. PubMed ID: 28073379
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  • 9. Liraglutide can reverse memory impairment, synaptic loss and reduce plaque load in aged APP/PS1 mice, a model of Alzheimer's disease.
    McClean PL, Hölscher C.
    Neuropharmacology; 2014 Jan 10; 76 Pt A():57-67. PubMed ID: 23973293
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  • 10. L-3-n-butylphthalide reduces tau phosphorylation and improves cognitive deficits in AβPP/PS1-Alzheimer's transgenic mice.
    Peng Y, Hu Y, Xu S, Li P, Li J, Lu L, Yang H, Feng N, Wang L, Wang X.
    J Alzheimers Dis; 2012 Jan 10; 29(2):379-91. PubMed ID: 22233765
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  • 11. Green tea epigallocatechin-3-gallate (EGCG) reduces beta-amyloid mediated cognitive impairment and modulates tau pathology in Alzheimer transgenic mice.
    Rezai-Zadeh K, Arendash GW, Hou H, Fernandez F, Jensen M, Runfeldt M, Shytle RD, Tan J.
    Brain Res; 2008 Jun 12; 1214():177-87. PubMed ID: 18457818
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  • 13. SLOH, a carbazole-based fluorophore, mitigates neuropathology and behavioral impairment in the triple-transgenic mouse model of Alzheimer's disease.
    Wu X, Kosaraju J, Zhou W, Tam KY.
    Neuropharmacology; 2018 Mar 15; 131():351-363. PubMed ID: 29309769
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  • 14. Targeting CCR3 to Reduce Amyloid-β Production, Tau Hyperphosphorylation, and Synaptic Loss in a Mouse Model of Alzheimer's Disease.
    Zhu C, Xu B, Sun X, Zhu Q, Sui Y.
    Mol Neurobiol; 2017 Dec 15; 54(10):7964-7978. PubMed ID: 27878757
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  • 15. Soluble Conformers of Aβ and Tau Alter Selective Proteins Governing Axonal Transport.
    Sherman MA, LaCroix M, Amar F, Larson ME, Forster C, Aguzzi A, Bennett DA, Ramsden M, Lesné SE.
    J Neurosci; 2016 Sep 14; 36(37):9647-58. PubMed ID: 27629715
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  • 17. Mutation-induced loss of APP function causes GABAergic depletion in recessive familial Alzheimer's disease: analysis of Osaka mutation-knockin mice.
    Umeda T, Kimura T, Yoshida K, Takao K, Fujita Y, Matsuyama S, Sakai A, Yamashita M, Yamashita Y, Ohnishi K, Suzuki M, Takuma H, Miyakawa T, Takashima A, Morita T, Mori H, Tomiyama T.
    Acta Neuropathol Commun; 2017 Jul 31; 5(1):59. PubMed ID: 28760161
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  • 18. The GABAergic septohippocampal connection is impaired in a mouse model of tauopathy.
    Soler H, Dorca-Arévalo J, González M, Rubio SE, Ávila J, Soriano E, Pascual M.
    Neurobiol Aging; 2017 Jan 31; 49():40-51. PubMed ID: 27743524
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