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Journal Abstract Search


1251 related items for PubMed ID: 27038603

  • 1. Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Clark JA, Southam KA, Blizzard CA, King AE, Dickson TC.
    J Chem Neuroanat; 2016 Oct; 76(Pt A):35-47. PubMed ID: 27038603
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  • 4. AAV-NRIP gene therapy ameliorates motor neuron degeneration and muscle atrophy in ALS model mice.
    Chen HH, Yeo HT, Huang YH, Tsai LK, Lai HJ, Tsao YP, Chen SL.
    Skelet Muscle; 2024 Jul 24; 14(1):17. PubMed ID: 39044305
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  • 5. Opposite Synaptic Alterations at the Neuromuscular Junction in an ALS Mouse Model: When Motor Units Matter.
    Tremblay E, Martineau É, Robitaille R.
    J Neurosci; 2017 Sep 13; 37(37):8901-8918. PubMed ID: 28821658
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  • 6. Electroacupuncture alleviates motor dysfunction by regulating neuromuscular junction disruption and neuronal degeneration in SOD1G93A mice.
    Liu J, Zhao W, Guo J, Kang K, Li H, Yang X, Li J, Wang Q, Qiao H.
    Brain Res Bull; 2024 Oct 01; 216():111036. PubMed ID: 39084570
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  • 7. Adipose-derived stem cell conditioned medium impacts asymptomatic peripheral neuromuscular denervation in the mutant superoxide dismutase (G93A) transgenic mouse model of amyotrophic lateral sclerosis.
    Walker CL, Meadows RM, Merfeld-Clauss S, Du Y, March KL, Jones KJ.
    Restor Neurol Neurosci; 2018 Oct 01; 36(5):621-627. PubMed ID: 30010155
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  • 8. Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS).
    Van Dyke JM, Smit-Oistad IM, Macrander C, Krakora D, Meyer MG, Suzuki M.
    Exp Neurol; 2016 Mar 01; 277():275-282. PubMed ID: 26775178
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  • 10. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
    Hegedus J, Putman CT, Gordon T.
    Neurobiol Dis; 2007 Nov 01; 28(2):154-64. PubMed ID: 17766128
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  • 11. Neuromuscular junction denervation and terminal Schwann cell loss in the hTDP-43 overexpression mouse model of amyotrophic lateral sclerosis.
    Alhindi A, Shand M, Smith HL, Leite AS, Huang YT, van der Hoorn D, Ridgway Z, Faller KME, Jones RA, Gillingwater TH, Chaytow H.
    Neuropathol Appl Neurobiol; 2023 Aug 01; 49(4):e12925. PubMed ID: 37465879
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  • 12. Caspase 6 has a protective role in SOD1(G93A) transgenic mice.
    Hogg MC, Mitchem MR, König HG, Prehn JH.
    Biochim Biophys Acta; 2016 Jun 01; 1862(6):1063-73. PubMed ID: 26976329
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  • 13. Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.
    Kobayakawa Y, Sakumi K, Kajitani K, Kadoya T, Horie H, Kira J, Nakabeppu Y.
    Neuropathol Appl Neurobiol; 2015 Feb 01; 41(2):227-44. PubMed ID: 24707896
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  • 14. Neuromuscular effects of G93A-SOD1 expression in zebrafish.
    Sakowski SA, Lunn JS, Busta AS, Oh SS, Zamora-Berridi G, Palmer M, Rosenberg AA, Philip SG, Dowling JJ, Feldman EL.
    Mol Neurodegener; 2012 Aug 31; 7():44. PubMed ID: 22938571
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  • 15. Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Gordon T, Tyreman N, Li S, Putman CT, Hegedus J.
    Neurobiol Dis; 2010 Feb 31; 37(2):412-22. PubMed ID: 19879358
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  • 16. Early changes of neuromuscular transmission in the SOD1(G93A) mice model of ALS start long before motor symptoms onset.
    Rocha MC, Pousinha PA, Correia AM, Sebastião AM, Ribeiro JA.
    PLoS One; 2013 Feb 31; 8(9):e73846. PubMed ID: 24040091
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  • 17. Progressive Degeneration and Inhibition of Peripheral Nerve Regeneration in the SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis.
    Deng B, Lv W, Duan W, Liu Y, Li Z, Ma Y, Zhang G, Song X, Cui C, Qi X, Li Y, Li C.
    Cell Physiol Biochem; 2018 Feb 31; 46(6):2358-2372. PubMed ID: 29742495
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  • 18. Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice.
    Wong M, Martin LJ.
    Hum Mol Genet; 2010 Jun 01; 19(11):2284-302. PubMed ID: 20223753
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  • 19. Muscle-derived but not centrally derived transgene GDNF is neuroprotective in G93A-SOD1 mouse model of ALS.
    Li W, Brakefield D, Pan Y, Hunter D, Myckatyn TM, Parsadanian A.
    Exp Neurol; 2007 Feb 01; 203(2):457-71. PubMed ID: 17034790
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  • 20. Delayed disease onset and extended survival in the SOD1G93A rat model of amyotrophic lateral sclerosis after suppression of mutant SOD1 in the motor cortex.
    Thomsen GM, Gowing G, Latter J, Chen M, Vit JP, Staggenborg K, Avalos P, Alkaslasi M, Ferraiuolo L, Likhite S, Kaspar BK, Svendsen CN.
    J Neurosci; 2014 Nov 19; 34(47):15587-600. PubMed ID: 25411487
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