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Journal Abstract Search

219 related items for PubMed ID: 27150392

  • 1. Type 1 Deiodinase Regulates ApoA-I Gene Expression and ApoA-I Synthesis Independent of Thyroid Hormone Signaling.
    Liu J, Hernandez-Ono A, Graham MJ, Galton VA, Ginsberg HN.
    Arterioscler Thromb Vasc Biol; 2016 Jul; 36(7):1356-66. PubMed ID: 27150392
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  • 2. Effect of 3,5,3'-Triiodothyronine (T3) administration on dio1 gene expression and T3 metabolism in normal and type 1 deiodinase-deficient mice.
    Maia AL, Kieffer JD, Harney JW, Larsen PR.
    Endocrinology; 1995 Nov; 136(11):4842-9. PubMed ID: 7588215
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  • 3. A novel compound 4010B-30 upregulates apolipoprotein A-I gene expression through activation of PPARγ in HepG2 cells.
    Du Y, Wang L, Si S, Yang Y, Hong B.
    Atherosclerosis; 2015 Apr; 239(2):589-98. PubMed ID: 25734982
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  • 5. Dietary cholic acid lowers plasma levels of mouse and human apolipoprotein A-I primarily via a transcriptional mechanism.
    Srivastava RA, Srivastava N, Averna M.
    Eur J Biochem; 2000 Jul; 267(13):4272-80. PubMed ID: 10866832
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  • 6. NFkappaB regulates plasma apolipoprotein A-I and high density lipoprotein cholesterol through inhibition of peroxisome proliferator-activated receptor alpha.
    Morishima A, Ohkubo N, Maeda N, Miki T, Mitsuda N.
    J Biol Chem; 2003 Oct 03; 278(40):38188-93. PubMed ID: 12882972
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  • 7. Bile acid-activated nuclear receptor FXR suppresses apolipoprotein A-I transcription via a negative FXR response element.
    Claudel T, Sturm E, Duez H, Torra IP, Sirvent A, Kosykh V, Fruchart JC, Dallongeville J, Hum DW, Kuipers F, Staels B.
    J Clin Invest; 2002 Apr 03; 109(7):961-71. PubMed ID: 11927623
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  • 8. Human Type 1 Iodothyronine Deiodinase (DIO1) Mutations Cause Abnormal Thyroid Hormone Metabolism.
    França MM, German A, Fernandes GW, Liao XH, Bianco AC, Refetoff S, Dumitrescu AM.
    Thyroid; 2021 Feb 03; 31(2):202-207. PubMed ID: 32718224
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  • 10. LCAT can rescue the abnormal phenotype produced by the natural ApoA-I mutations (Leu141Arg)Pisa and (Leu159Arg)FIN.
    Koukos G, Chroni A, Duka A, Kardassis D, Zannis VI.
    Biochemistry; 2007 Sep 18; 46(37):10713-21. PubMed ID: 17711302
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  • 11. ApoA-I knockout mice: characterization of HDL metabolism in homozygotes and identification of a post-RNA mechanism of apoA-I up-regulation in heterozygotes.
    Plump AS, Azrolan N, Odaka H, Wu L, Jiang X, Tall A, Eisenberg S, Breslow JL.
    J Lipid Res; 1997 May 18; 38(5):1033-47. PubMed ID: 9186920
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  • 13. AAV gene therapy as a means to increase apolipoprotein (Apo) A-I and high-density lipoprotein-cholesterol levels: correction of murine ApoA-I deficiency.
    Vaessen SF, Veldman RJ, Comijn EM, Snapper J, Sierts JA, van den Oever K, Beattie SG, Twisk J, Kuivenhoven JA.
    J Gene Med; 2009 Aug 18; 11(8):697-707. PubMed ID: 19431216
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  • 15. Increased high density lipoprotein (HDL), defective hepatic catabolism of ApoA-I and ApoA-II, and decreased ApoA-I mRNA in ob/ob mice. Possible role of leptin in stimulation of HDL turnover.
    Silver DL, Jiang XC, Tall AR.
    J Biol Chem; 1999 Feb 12; 274(7):4140-6. PubMed ID: 9933608
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  • 16. Deletions of helices 2 and 3 of human apoA-I are associated with severe dyslipidemia following adenovirus-mediated gene transfer in apoA-I-deficient mice.
    Chroni A, Kan HY, Shkodrani A, Liu T, Zannis VI.
    Biochemistry; 2005 Mar 15; 44(10):4108-17. PubMed ID: 15751988
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  • 18. Hepatocyte nuclear factor 4alpha contributes to thyroid hormone homeostasis by cooperatively regulating the type 1 iodothyronine deiodinase gene with GATA4 and Kruppel-like transcription factor 9.
    Ohguchi H, Tanaka T, Uchida A, Magoori K, Kudo H, Kim I, Daigo K, Sakakibara I, Okamura M, Harigae H, Sasaki T, Osborne TF, Gonzalez FJ, Hamakubo T, Kodama T, Sakai J.
    Mol Cell Biol; 2008 Jun 15; 28(12):3917-31. PubMed ID: 18426912
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