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PUBMED FOR HANDHELDS

Journal Abstract Search


401 related items for PubMed ID: 27206451

  • 1. Inhibition of ER stress and unfolding protein response pathways causes skeletal muscle wasting during cancer cachexia.
    Bohnert KR, Gallot YS, Sato S, Xiong G, Hindi SM, Kumar A.
    FASEB J; 2016 Sep; 30(9):3053-68. PubMed ID: 27206451
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  • 3. The Toll-Like Receptor/MyD88/XBP1 Signaling Axis Mediates Skeletal Muscle Wasting during Cancer Cachexia.
    Bohnert KR, Goli P, Roy A, Sharma AK, Xiong G, Gallot YS, Kumar A.
    Mol Cell Biol; 2019 Aug 01; 39(15):. PubMed ID: 31138662
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  • 6. Muscle mTORC1 suppression by IL-6 during cancer cachexia: a role for AMPK.
    White JP, Puppa MJ, Gao S, Sato S, Welle SL, Carson JA.
    Am J Physiol Endocrinol Metab; 2013 May 15; 304(10):E1042-52. PubMed ID: 23531613
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  • 11. Effects of muscular dystrophy, exercise and blocking activin receptor IIB ligands on the unfolded protein response and oxidative stress.
    Hulmi JJ, Hentilä J, DeRuisseau KC, Oliveira BM, Papaioannou KG, Autio R, Kujala UM, Ritvos O, Kainulainen H, Korkmaz A, Atalay M.
    Free Radic Biol Med; 2016 Oct 15; 99():308-322. PubMed ID: 27554968
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  • 12. Reduced sucrose nonfermenting AMPK-related kinase (SNARK) activity aggravates cancer-induced skeletal muscle wasting.
    Alves CRR, MacDonald TL, Nigro P, Pathak P, Hirshman MF, Goodyear LJ, Lessard SJ.
    Biomed Pharmacother; 2019 Sep 15; 117():109197. PubMed ID: 31387190
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  • 14. C/EBPβ mediates tumour-induced ubiquitin ligase atrogin1/MAFbx upregulation and muscle wasting.
    Zhang G, Jin B, Li YP.
    EMBO J; 2011 Aug 16; 30(20):4323-35. PubMed ID: 21847090
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  • 16. VEGF Signals through ATF6 and PERK to promote endothelial cell survival and angiogenesis in the absence of ER stress.
    Karali E, Bellou S, Stellas D, Klinakis A, Murphy C, Fotsis T.
    Mol Cell; 2014 May 22; 54(4):559-72. PubMed ID: 24746698
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