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Journal Abstract Search

530 related items for PubMed ID: 27439514

  • 1. Lack of IL-1 Receptor-Associated Kinase-4 Leads to Defective Th1 Cell Responses and Renders Mice Susceptible to Mycobacterial Infection.
    Marinho FV, Fahel JS, Scanga CA, Gomes MT, Guimarães G, Carvalho GR, Morales SV, Báfica A, Oliveira SC.
    J Immunol; 2016 Sep 01; 197(5):1852-63. PubMed ID: 27439514
    [Abstract] [Full Text] [Related]

  • 2. Complement C5a anaphylatoxin is an innate determinant of dendritic cell-induced Th1 immunity to Mycobacterium bovis BCG infection in mice.
    Moulton RA, Mashruwala MA, Smith AK, Lindsey DR, Wetsel RA, Haviland DL, Hunter RL, Jagannath C.
    J Leukoc Biol; 2007 Oct 01; 82(4):956-67. PubMed ID: 17675563
    [Abstract] [Full Text] [Related]

  • 3. Role of apoptosis-regulating signal kinase 1 in innate immune responses by Mycobacterium bovis bacillus Calmette-Guérin.
    Yuk JM, Shin DM, Yang CS, Kim KH, An SJ, Rho J, Park JK, Jo EK.
    Immunol Cell Biol; 2009 Jan 01; 87(1):100-7. PubMed ID: 18852704
    [Abstract] [Full Text] [Related]

  • 4. Severe impairment of interleukin-1 and Toll-like receptor signalling in mice lacking IRAK-4.
    Suzuki N, Suzuki S, Duncan GS, Millar DG, Wada T, Mirtsos C, Takada H, Wakeham A, Itie A, Li S, Penninger JM, Wesche H, Ohashi PS, Mak TW, Yeh WC.
    Nature; 2002 Apr 18; 416(6882):750-6. PubMed ID: 11923871
    [Abstract] [Full Text] [Related]

  • 5. IRAK-M alters the polarity of macrophages to facilitate the survival of Mycobacterium tuberculosis.
    Shen P, Li Q, Ma J, Tian M, Hong F, Zhai X, Li J, Huang H, Shi C.
    BMC Microbiol; 2017 Aug 23; 17(1):185. PubMed ID: 28835201
    [Abstract] [Full Text] [Related]

  • 6. De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.
    Stüve P, Minarrieta L, Erdmann H, Arnold-Schrauf C, Swallow M, Guderian M, Krull F, Hölscher A, Ghorbani P, Behrends J, Abraham WR, Hölscher C, Sparwasser TD, Berod L.
    Front Immunol; 2018 Aug 23; 9():495. PubMed ID: 29675017
    [Abstract] [Full Text] [Related]

  • 7. Transient membrane recruitment of IRAK-1 in response to LPS and IL-1beta requires TNF R1.
    Lockett A, Goebl MG, Harrington MA.
    Am J Physiol Cell Physiol; 2008 Aug 23; 295(2):C313-23. PubMed ID: 18562480
    [Abstract] [Full Text] [Related]

  • 8. PPE57 induces activation of macrophages and drives Th1-type immune responses through TLR2.
    Xu Y, Yang E, Huang Q, Ni W, Kong C, Liu G, Li G, Su H, Wang H.
    J Mol Med (Berl); 2015 Jun 23; 93(6):645-62. PubMed ID: 25586105
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  • 10. Toll-like receptor 6 senses Mycobacterium avium and is required for efficient control of mycobacterial infection.
    Marinho FA, de Paula RR, Mendes AC, de Almeida LA, Gomes MT, Carvalho NB, Oliveira FS, Caliari MV, Oliveira SC.
    Eur J Immunol; 2013 Sep 23; 43(9):2373-85. PubMed ID: 23716075
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  • 17. Procyanidin dimer B2-mediated IRAK-M induction negatively regulates TLR4 signaling in macrophages.
    Sung NY, Yang MS, Song DS, Kim JK, Park JH, Song BS, Park SH, Lee JW, Park HJ, Kim JH, Byun EB, Byun EH.
    Biochem Biophys Res Commun; 2013 Aug 16; 438(1):122-8. PubMed ID: 23872113
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  • 18. The Mycobacterium tuberculosis glycoprotein Rv1016c protein inhibits dendritic cell maturation, and impairs Th1 /Th17 responses during mycobacteria infection.
    Su H, Peng B, Zhang Z, Liu Z, Zhang Z.
    Mol Immunol; 2019 May 16; 109():58-70. PubMed ID: 30856410
    [Abstract] [Full Text] [Related]

  • 19. IL-1 receptor-associated kinase modulates host responsiveness to endotoxin.
    Swantek JL, Tsen MF, Cobb MH, Thomas JA.
    J Immunol; 2000 Apr 15; 164(8):4301-6. PubMed ID: 10754329
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  • 20. IL-1R-associated kinase 4 is required for lipopolysaccharide-induced activation of APC.
    Suzuki N, Suzuki S, Eriksson U, Hara H, Mirtosis C, Chen NJ, Wada T, Bouchard D, Hwang I, Takeda K, Fujita T, Der S, Penninger JM, Akira S, Saito T, Yeh WC.
    J Immunol; 2003 Dec 01; 171(11):6065-71. PubMed ID: 14634120
    [Abstract] [Full Text] [Related]

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