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116 related items for PubMed ID: 27450018

  • 1. The study of the mechanism of arsenite toxicity in respiration-deficient cells reveals that NADPH oxidase-derived superoxide promotes the same downstream events mediated by mitochondrial superoxide in respiration-proficient cells.
    Guidarelli A, Fiorani M, Carloni S, Cerioni L, Balduini W, Cantoni O.
    Toxicol Appl Pharmacol; 2016 Sep 15; 307():35-44. PubMed ID: 27450018
    [Abstract] [Full Text] [Related]

  • 2. The compartmentalised nature of the mechanisms governing superoxide formation and scavenging in cells exposed to arsenite.
    Guidarelli A, Fiorani M, Cerioni L, Cantoni O.
    Toxicol Appl Pharmacol; 2019 Dec 01; 384():114766. PubMed ID: 31647942
    [Abstract] [Full Text] [Related]

  • 3. Mitochondrial ascorbic acid prevents mitochondrial O₂·⁻ formation, an event critical for U937 cell apoptosis induced by arsenite through both autophagic-dependent and independent mechanisms.
    Guidarelli A, Carloni S, Balduini W, Fiorani M, Cantoni O.
    Biofactors; 2016 Dec 01; 42(2):190-200. PubMed ID: 26893200
    [Abstract] [Full Text] [Related]

  • 4. Arsenite-Induced Mitochondrial Superoxide Formation: Time and Concentration Requirements for the Effects of the Metalloid on the Endoplasmic Reticulum and Mitochondria.
    Guidarelli A, Cerioni L, Fiorani M, Catalani A, Cantoni O.
    J Pharmacol Exp Ther; 2020 Apr 01; 373(1):62-71. PubMed ID: 31941719
    [Abstract] [Full Text] [Related]

  • 5. The dual role of mitochondrial superoxide in arsenite toxicity: Signaling at the boundary between apoptotic commitment and cytoprotection.
    Fiorani M, Guidarelli A, Capellacci V, Cerioni L, Crinelli R, Cantoni O.
    Toxicol Appl Pharmacol; 2018 Apr 15; 345():26-35. PubMed ID: 29526526
    [Abstract] [Full Text] [Related]

  • 6. RETRACTED: Thapsigargin triggers cardiac contractile dysfunction via NADPH oxidase-mediated mitochondrial dysfunction: Role of Akt dephosphorylation.
    Zhang Y, Ren J.
    Free Radic Biol Med; 2011 Dec 15; 51(12):2172-2184. PubMed ID: 21996563
    [Abstract] [Full Text] [Related]

  • 7. Mitochondrial respiration scavenges extramitochondrial superoxide anion via a nonenzymatic mechanism.
    Guidot DM, Repine JE, Kitlowski AD, Flores SC, Nelson SK, Wright RM, McCord JM.
    J Clin Invest; 1995 Aug 15; 96(2):1131-6. PubMed ID: 7635949
    [Abstract] [Full Text] [Related]

  • 8. Calcium signals between the ryanodine receptor- and mitochondria critically regulate the effects of arsenite on mitochondrial superoxide formation and on the ensuing survival vs apoptotic signaling.
    Guidarelli A, Fiorani M, Cerioni L, Cantoni O.
    Redox Biol; 2019 Jan 15; 20():285-295. PubMed ID: 30388683
    [Abstract] [Full Text] [Related]

  • 9. ERO1α primes the ryanodine receptor to respond to arsenite with concentration dependent Ca2+ release sequentially triggering two different mechanisms of ROS formation.
    Guidarelli A, Spina A, Buffi G, Blandino G, Fiorani M, Cantoni O.
    Chem Biol Interact; 2023 Sep 25; 383():110694. PubMed ID: 37659621
    [Abstract] [Full Text] [Related]

  • 10. Functional organization of the endoplasmic reticulum dictates the susceptibility of target cells to arsenite-induced mitochondrial superoxide formation, mitochondrial dysfunction and apoptosis.
    Guidarelli A, Catalani A, Spina A, Varone E, Fumagalli S, Zito E, Fiorani M, Cantoni O.
    Food Chem Toxicol; 2021 Oct 25; 156():112523. PubMed ID: 34453993
    [Abstract] [Full Text] [Related]

  • 11. Arsenite impinges on endoplasmic reticulum-mitochondria crosstalk to elicit mitochondrial ROS formation and downstream toxicity.
    Cantoni O, Zito E, Fiorani M, Guidarelli A.
    Semin Cancer Biol; 2021 Nov 25; 76():132-138. PubMed ID: 34089843
    [Abstract] [Full Text] [Related]

  • 12. Arsenite-induced endoplasmic reticulum-dependent apoptosis through disturbance of calcium homeostasis in HBE cell line.
    Chen C, Gu S, Jiang X, Zhang Z.
    Environ Toxicol; 2017 Jan 25; 32(1):197-216. PubMed ID: 26677073
    [Abstract] [Full Text] [Related]

  • 13. Arsenite induces DNA damage via mitochondrial ROS and induction of mitochondrial permeability transition.
    Guidarelli A, Fiorani M, Cerioni L, Scotti M, Cantoni O.
    Biofactors; 2017 Sep 10; 43(5):673-684. PubMed ID: 28703385
    [Abstract] [Full Text] [Related]

  • 14. Inhibition of activity/expression, or genetic deletion, of ERO1α blunts arsenite geno- and cyto-toxicity.
    Guidarelli A, Spina A, Fiorani M, Zito E, Cantoni O.
    Food Chem Toxicol; 2022 Oct 10; 168():113360. PubMed ID: 35964836
    [Abstract] [Full Text] [Related]

  • 15. U937 cell apoptosis induced by arsenite is prevented by low concentrations of mitochondrial ascorbic acid with hardly any effect mediated by the cytosolic fraction of the vitamin.
    Guidarelli A, Fiorani M, Azzolini C, Cerioni L, Scotti M, Cantoni O.
    Biofactors; 2015 Oct 10; 41(2):101-10. PubMed ID: 25809564
    [Abstract] [Full Text] [Related]

  • 16. Taurine resumed neuronal differentiation in arsenite-treated N2a cells through reducing oxidative stress, endoplasmic reticulum stress, and mitochondrial dysfunction.
    Chou CT, Lin HT, Hwang PA, Wang ST, Hsieh CH, Hwang DF.
    Amino Acids; 2015 Apr 10; 47(4):735-44. PubMed ID: 25547999
    [Abstract] [Full Text] [Related]

  • 17. Mitochondrial superoxide mediates mitochondrial and endoplasmic reticulum dysfunctions in TRAIL-induced apoptosis in Jurkat cells.
    Inoue T, Suzuki-Karasaki Y.
    Free Radic Biol Med; 2013 Aug 10; 61():273-84. PubMed ID: 23608466
    [Abstract] [Full Text] [Related]

  • 18. NADPH oxidase-mitochondria axis-derived ROS mediate arsenite-induced HIF-1α stabilization by inhibiting prolyl hydroxylases activity.
    Li YN, Xi MM, Guo Y, Hai CX, Yang WL, Qin XJ.
    Toxicol Lett; 2014 Jan 13; 224(2):165-74. PubMed ID: 24188932
    [Abstract] [Full Text] [Related]

  • 19. Low Concentrations of Arsenite Target the Intraluminal Inositol 1, 4, 5-Trisphosphate Receptor/Ryanodine Receptor Crosstalk to Significantly Elevate Intracellular Ca2.
    Guidarelli A, Fiorani M, Cantoni O.
    J Pharmacol Exp Ther; 2018 Oct 13; 367(1):184-193. PubMed ID: 30068729
    [Abstract] [Full Text] [Related]

  • 20. Crosstalk between ERO1α and ryanodine receptor in arsenite-dependent mitochondrial ROS formation.
    Spina A, Guidarelli A, Fiorani M, Varone E, Catalani A, Zito E, Cantoni O.
    Biochem Pharmacol; 2022 Apr 13; 198():114973. PubMed ID: 35189109
    [Abstract] [Full Text] [Related]

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