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Journal Abstract Search

362 related items for PubMed ID: 27459671

  • 1. Tau Protein Mediates APP Intracellular Domain (AICD)-Induced Alzheimer's-Like Pathological Features in Mice.
    Ghosal K, Fan Q, Dawson HN, Pimplikar SW.
    PLoS One; 2016; 11(7):e0159435. PubMed ID: 27459671
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  • 2. The apolipoprotein-E-mimetic COG112 protects amyloid precursor protein intracellular domain-overexpressing animals from Alzheimer's disease-like pathological features.
    Ghosal K, Stathopoulos A, Thomas D, Phenis D, Vitek MP, Pimplikar SW.
    Neurodegener Dis; 2013; 12(1):51-8. PubMed ID: 22965147
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  • 3. APP intracellular domain impairs adult neurogenesis in transgenic mice by inducing neuroinflammation.
    Ghosal K, Stathopoulos A, Pimplikar SW.
    PLoS One; 2010 Jul 30; 5(7):e11866. PubMed ID: 20689579
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  • 4. Alzheimer's disease-like pathological features in transgenic mice expressing the APP intracellular domain.
    Ghosal K, Vogt DL, Liang M, Shen Y, Lamb BT, Pimplikar SW.
    Proc Natl Acad Sci U S A; 2009 Oct 27; 106(43):18367-72. PubMed ID: 19837693
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  • 5. Modeling Alzheimer's disease in mouse without mutant protein overexpression: cooperative and independent effects of Aβ and tau.
    Guo Q, Li H, Cole AL, Hur JY, Li Y, Zheng H.
    PLoS One; 2013 Oct 27; 8(11):e80706. PubMed ID: 24278307
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  • 7. JNK-interacting protein 1 mediates Alzheimer's-like pathological features in AICD-transgenic mice.
    Margevicius DR, Bastian C, Fan Q, Davis RJ, Pimplikar SW.
    Neurobiol Aging; 2015 Aug 27; 36(8):2370-9. PubMed ID: 26022769
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  • 8. Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimer's disease mouse model.
    Roberson ED, Scearce-Levie K, Palop JJ, Yan F, Cheng IH, Wu T, Gerstein H, Yu GQ, Mucke L.
    Science; 2007 May 04; 316(5825):750-4. PubMed ID: 17478722
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  • 13. Aging and excitotoxic stress exacerbate neural circuit reorganization in amyloid precursor protein intracellular domain transgenic mice.
    Ghosal K, Pimplikar SW.
    Neurobiol Aging; 2011 Dec 04; 32(12):2320.e1-9. PubMed ID: 20493588
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  • 14. Physiological and pathophysiological control of synaptic GluN2B-NMDA receptors by the C-terminal domain of amyloid precursor protein.
    Pousinha PA, Mouska X, Raymond EF, Gwizdek C, Dhib G, Poupon G, Zaragosi LE, Giudici C, Bethus I, Pacary E, Willem M, Marie H.
    Elife; 2017 Jul 06; 6():. PubMed ID: 28682239
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  • 15. Ca2+-permeable TRPV1 pain receptor knockout rescues memory deficits and reduces amyloid-β and tau in a mouse model of Alzheimer's disease.
    Kim J, Lee S, Kim J, Ham S, Park JHY, Han S, Jung YK, Shim I, Han JS, Lee KW, Kim J.
    Hum Mol Genet; 2020 Jan 15; 29(2):228-237. PubMed ID: 31814000
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  • 18. Tau passive immunization inhibits not only tau but also Aβ pathology.
    Dai CL, Tung YC, Liu F, Gong CX, Iqbal K.
    Alzheimers Res Ther; 2017 Jan 10; 9(1):1. PubMed ID: 28073379
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  • 20. Alzheimer's disease pathological lesions activate the spleen tyrosine kinase.
    Schweig JE, Yao H, Beaulieu-Abdelahad D, Ait-Ghezala G, Mouzon B, Crawford F, Mullan M, Paris D.
    Acta Neuropathol Commun; 2017 Sep 06; 5(1):69. PubMed ID: 28877763
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