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Journal Abstract Search


227 related items for PubMed ID: 27555555

  • 1. Eliminating Nox2 reactive oxygen species production protects dystrophic skeletal muscle from pathological calcium influx assessed in vivo by manganese-enhanced magnetic resonance imaging.
    Loehr JA, Stinnett GR, Hernández-Rivera M, Roten WT, Wilson LJ, Pautler RG, Rodney GG.
    J Physiol; 2016 Nov 01; 594(21):6395-6405. PubMed ID: 27555555
    [Abstract] [Full Text] [Related]

  • 2. Microtubules underlie dysfunction in duchenne muscular dystrophy.
    Khairallah RJ, Shi G, Sbrana F, Prosser BL, Borroto C, Mazaitis MJ, Hoffman EP, Mahurkar A, Sachs F, Sun Y, Chen YW, Raiteri R, Lederer WJ, Dorsey SG, Ward CW.
    Sci Signal; 2012 Aug 07; 5(236):ra56. PubMed ID: 22871609
    [Abstract] [Full Text] [Related]

  • 3. Nox4 - RyR1 - Nox2: Regulators of micro-domain signaling in skeletal muscle.
    Cully TR, Rodney GG.
    Redox Biol; 2020 Sep 07; 36():101557. PubMed ID: 32506037
    [Abstract] [Full Text] [Related]

  • 4. Reciprocal amplification of ROS and Ca(2+) signals in stressed mdx dystrophic skeletal muscle fibers.
    Shkryl VM, Martins AS, Ullrich ND, Nowycky MC, Niggli E, Shirokova N.
    Pflugers Arch; 2009 Sep 07; 458(5):915-28. PubMed ID: 19387681
    [Abstract] [Full Text] [Related]

  • 5. Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells.
    Henríquez-Olguín C, Altamirano F, Valladares D, López JR, Allen PD, Jaimovich E.
    Biochim Biophys Acta; 2015 Jul 07; 1852(7):1410-9. PubMed ID: 25857619
    [Abstract] [Full Text] [Related]

  • 6. Hypochlorous acid exposure impairs skeletal muscle function and Ca2+ signalling: implications for Duchenne muscular dystrophy pathology.
    Lea TA, Panizza PM, Arthur PG, Bakker AJ, Pinniger GJ.
    J Physiol; 2023 Dec 07; 601(23):5257-5275. PubMed ID: 37864413
    [Abstract] [Full Text] [Related]

  • 7. NADPH oxidase mediates microtubule alterations and diaphragm dysfunction in dystrophic mice.
    Loehr JA, Wang S, Cully TR, Pal R, Larina IV, Larin KV, Rodney GG.
    Elife; 2018 Jan 30; 7():. PubMed ID: 29381135
    [Abstract] [Full Text] [Related]

  • 8. Diapocynin, a dimer of the NADPH oxidase inhibitor apocynin, reduces ROS production and prevents force loss in eccentrically contracting dystrophic muscle.
    Ismail HM, Scapozza L, Ruegg UT, Dorchies OM.
    PLoS One; 2014 Jan 30; 9(10):e110708. PubMed ID: 25329652
    [Abstract] [Full Text] [Related]

  • 9. Muscle damage in mdx (dystrophic) mice: role of calcium and reactive oxygen species.
    Whitehead NP, Yeung EW, Allen DG.
    Clin Exp Pharmacol Physiol; 2006 Jul 30; 33(7):657-62. PubMed ID: 16789936
    [Abstract] [Full Text] [Related]

  • 10. BETs inhibition attenuates oxidative stress and preserves muscle integrity in Duchenne muscular dystrophy.
    Segatto M, Szokoll R, Fittipaldi R, Bottino C, Nevi L, Mamchaoui K, Filippakopoulos P, Caretti G.
    Nat Commun; 2020 Nov 30; 11(1):6108. PubMed ID: 33257646
    [Abstract] [Full Text] [Related]

  • 11. Alterations in Notch signalling in skeletal muscles from mdx and dko dystrophic mice and patients with Duchenne muscular dystrophy.
    Church JE, Trieu J, Chee A, Naim T, Gehrig SM, Lamon S, Angelini C, Russell AP, Lynch GS.
    Exp Physiol; 2014 Apr 30; 99(4):675-87. PubMed ID: 24443351
    [Abstract] [Full Text] [Related]

  • 12. NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.
    Gonzalez DR, Treuer AV, Lamirault G, Mayo V, Cao Y, Dulce RA, Hare JM.
    Am J Physiol Heart Circ Physiol; 2014 Sep 01; 307(5):H710-21. PubMed ID: 25015966
    [Abstract] [Full Text] [Related]

  • 13. Pivotal role of miR-448 in the development of ROS-induced cardiomyopathy.
    Kyrychenko S, Kyrychenko V, Badr MA, Ikeda Y, Sadoshima J, Shirokova N.
    Cardiovasc Res; 2015 Dec 01; 108(3):324-34. PubMed ID: 26503985
    [Abstract] [Full Text] [Related]

  • 14. Coenzyme Q10 supplementation acts as antioxidant on dystrophic muscle cells.
    Mizobuti DS, Fogaça AR, Moraes FDSR, Moraes LHR, Mâncio RD, Hermes TA, Macedo AB, Valduga AH, de Lourenço CC, Pereira ECL, Minatel E.
    Cell Stress Chaperones; 2019 Nov 01; 24(6):1175-1185. PubMed ID: 31620981
    [Abstract] [Full Text] [Related]

  • 15. Dysregulation of Intracellular Ca2+ in Dystrophic Cortical and Hippocampal Neurons.
    Lopez JR, Kolster J, Uryash A, Estève E, Altamirano F, Adams JA.
    Mol Neurobiol; 2018 Jan 01; 55(1):603-618. PubMed ID: 27975174
    [Abstract] [Full Text] [Related]

  • 16. X-ROS signaling: rapid mechano-chemo transduction in heart.
    Prosser BL, Ward CW, Lederer WJ.
    Science; 2011 Sep 09; 333(6048):1440-5. PubMed ID: 21903813
    [Abstract] [Full Text] [Related]

  • 17. Proteomic analysis of the sarcolemma-enriched fraction from dystrophic mdx-4cv skeletal muscle.
    Murphy S, Zweyer M, Henry M, Meleady P, Mundegar RR, Swandulla D, Ohlendieck K.
    J Proteomics; 2019 Jan 16; 191():212-227. PubMed ID: 29408692
    [Abstract] [Full Text] [Related]

  • 18. The role of reactive oxygen species in the hearts of dystrophin-deficient mdx mice.
    Williams IA, Allen DG.
    Am J Physiol Heart Circ Physiol; 2007 Sep 16; 293(3):H1969-77. PubMed ID: 17573457
    [Abstract] [Full Text] [Related]

  • 19. Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.
    Altamirano F, Valladares D, Henríquez-Olguín C, Casas M, López JR, Allen PD, Jaimovich E.
    PLoS One; 2013 Sep 16; 8(12):e81222. PubMed ID: 24349043
    [Abstract] [Full Text] [Related]

  • 20. Is the normal content of sulfhydryl groups attributable to sparing from dystrophic pathology in dystrophin-deficient muscles?
    Niebrój-Dobosz I, Fidziańska A, Glinka Z, Hausmanowa-Petrusewicz I.
    Folia Neuropathol; 2002 Sep 16; 40(3):143-50. PubMed ID: 12572920
    [Abstract] [Full Text] [Related]


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