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Journal Abstract Search


366 related items for PubMed ID: 27707967

  • 1. Neuronal Dystroglycan Is Necessary for Formation and Maintenance of Functional CCK-Positive Basket Cell Terminals on Pyramidal Cells.
    Früh S, Romanos J, Panzanelli P, Bürgisser D, Tyagarajan SK, Campbell KP, Santello M, Fritschy JM.
    J Neurosci; 2016 Oct 05; 36(40):10296-10313. PubMed ID: 27707967
    [Abstract] [Full Text] [Related]

  • 2. Neuronal Dystroglycan regulates postnatal development of CCK/cannabinoid receptor-1 interneurons.
    Miller DS, Wright KM.
    Neural Dev; 2021 Aug 06; 16(1):4. PubMed ID: 34362433
    [Abstract] [Full Text] [Related]

  • 3. Differential surface density and modulatory effects of presynaptic GABAB receptors in hippocampal cholecystokinin and parvalbumin basket cells.
    Booker SA, Althof D, Degro CE, Watanabe M, Kulik Á, Vida I.
    Brain Struct Funct; 2017 Nov 06; 222(8):3677-3690. PubMed ID: 28466358
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  • 4. Differential role of GABAA receptors and neuroligin 2 for perisomatic GABAergic synapse formation in the hippocampus.
    Panzanelli P, Früh S, Fritschy JM.
    Brain Struct Funct; 2017 Dec 06; 222(9):4149-4161. PubMed ID: 28643105
    [Abstract] [Full Text] [Related]

  • 5. Selective reduction of cholecystokinin-positive basket cell innervation in a model of temporal lobe epilepsy.
    Wyeth MS, Zhang N, Mody I, Houser CR.
    J Neurosci; 2010 Jun 30; 30(26):8993-9006. PubMed ID: 20592220
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  • 6. Postsynaptic origin of CB1-dependent tonic inhibition of GABA release at cholecystokinin-positive basket cell to pyramidal cell synapses in the CA1 region of the rat hippocampus.
    Neu A, Földy C, Soltesz I.
    J Physiol; 2007 Jan 01; 578(Pt 1):233-47. PubMed ID: 17053036
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  • 8. Synapsin II Regulation of GABAergic Synaptic Transmission Is Dependent on Interneuron Subtype.
    Feliciano P, Matos H, Andrade R, Bykhovskaia M.
    J Neurosci; 2017 Feb 15; 37(7):1757-1771. PubMed ID: 28087765
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  • 9. Anatomically heterogeneous populations of CB1 cannabinoid receptor-expressing interneurons in the CA3 region of the hippocampus show homogeneous input-output characteristics.
    Szabó GG, Papp OI, Máté Z, Szabó G, Hájos N.
    Hippocampus; 2014 Dec 15; 24(12):1506-23. PubMed ID: 25044969
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  • 10. Properties and dynamics of inhibitory synaptic communication within the CA3 microcircuits of pyramidal cells and interneurons expressing parvalbumin or cholecystokinin.
    Kohus Z, Káli S, Rovira-Esteban L, Schlingloff D, Papp O, Freund TF, Hájos N, Gulyás AI.
    J Physiol; 2016 Jul 01; 594(13):3745-74. PubMed ID: 27038232
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  • 11. Increased cholecystokinin labeling in the hippocampus of a mouse model of epilepsy maps to spines and glutamatergic terminals.
    Wyeth MS, Zhang N, Houser CR.
    Neuroscience; 2012 Jan 27; 202():371-83. PubMed ID: 22155653
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  • 12. Cholecystokinin-immunopositive basket and Schaffer collateral-associated interneurones target different domains of pyramidal cells in the CA1 area of the rat hippocampus.
    Cope DW, Maccaferri G, Márton LF, Roberts JD, Cobden PM, Somogyi P.
    Neuroscience; 2002 Jan 27; 109(1):63-80. PubMed ID: 11784700
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  • 13. Dystroglycan is selectively associated with inhibitory GABAergic synapses but is dispensable for their differentiation.
    Lévi S, Grady RM, Henry MD, Campbell KP, Sanes JR, Craig AM.
    J Neurosci; 2002 Jun 01; 22(11):4274-85. PubMed ID: 12040032
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  • 14. Experience and activity-dependent maturation of perisomatic GABAergic innervation in primary visual cortex during a postnatal critical period.
    Chattopadhyaya B, Di Cristo G, Higashiyama H, Knott GW, Kuhlman SJ, Welker E, Huang ZJ.
    J Neurosci; 2004 Oct 27; 24(43):9598-611. PubMed ID: 15509747
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  • 15. Presynaptic kainate receptor activation preserves asynchronous GABA release despite the reduction in synchronous release from hippocampal cholecystokinin interneurons.
    Daw MI, Pelkey KA, Chittajallu R, McBain CJ.
    J Neurosci; 2010 Aug 18; 30(33):11202-9. PubMed ID: 20720128
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  • 18. Distinct mechanisms regulate GABAA receptor and gephyrin clustering at perisomatic and axo-axonic synapses on CA1 pyramidal cells.
    Panzanelli P, Gunn BG, Schlatter MC, Benke D, Tyagarajan SK, Scheiffele P, Belelli D, Lambert JJ, Rudolph U, Fritschy JM.
    J Physiol; 2011 Oct 15; 589(Pt 20):4959-80. PubMed ID: 21825022
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  • 19. GABAergic terminals are required for postsynaptic clustering of dystrophin but not of GABA(A) receptors and gephyrin.
    Brünig I, Suter A, Knuesel I, Lüscher B, Fritschy JM.
    J Neurosci; 2002 Jun 15; 22(12):4805-13. PubMed ID: 12077177
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  • 20. Loss of cholecystokinin-containing terminals in temporal lobe epilepsy.
    Sun C, Sun J, Erisir A, Kapur J.
    Neurobiol Dis; 2014 Feb 15; 62():44-55. PubMed ID: 24051276
    [Abstract] [Full Text] [Related]


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