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170 related items for PubMed ID: 27789289

  • 1. MTUS1 silencing promotes E-selectin production through p38 MAPK-dependent CREB ubiquitination in endothelial cells.
    Wang Y, Dai X, Liu Y, Li J, Liu Z, Yin P, Chen J, Wang Y, Wang N, Zhang P.
    J Mol Cell Cardiol; 2016 Dec; 101():1-10. PubMed ID: 27789289
    [Abstract] [Full Text] [Related]

  • 2. The inhibition of TNF-alpha-induced E-selectin expression in endothelial cells via the JNK/NF-kappaB pathways by highly N-acetylated chitooligosaccharides.
    Lin CW, Chen LJ, Lee PL, Lee CI, Lin JC, Chiu JJ.
    Biomaterials; 2007 Mar; 28(7):1355-66. PubMed ID: 17126899
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  • 3. CAMP-response element-binding protein mediates tumor necrosis factor-alpha-induced vascular cell adhesion molecule-1 expression in endothelial cells.
    Ono H, Ichiki T, Ohtsubo H, Fukuyama K, Imayama I, Iino N, Masuda S, Hashiguchi Y, Takeshita A, Sunagawa K.
    Hypertens Res; 2006 Jan; 29(1):39-47. PubMed ID: 16715652
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  • 4. Unfractionated heparin inhibits lipopolysaccharide-induced inflammatory response through blocking p38 MAPK and NF-κB activation on endothelial cell.
    Li X, Zheng Z, Li X, Ma X.
    Cytokine; 2012 Oct; 60(1):114-21. PubMed ID: 22763042
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  • 5. Cyclic AMP inhibits p38 activation via CREB-induced dynein light chain.
    Zhang J, Bui TN, Xiang J, Lin A.
    Mol Cell Biol; 2006 Feb; 26(4):1223-34. PubMed ID: 16449637
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  • 13. Chitosan oligosaccharides downregulate the expression of E-selectin and ICAM-1 induced by LPS in endothelial cells by inhibiting MAP kinase signaling.
    Li Y, Xu Q, Wei P, Cheng L, Peng Q, Li S, Yin H, Du Y.
    Int J Mol Med; 2014 Feb; 33(2):392-400. PubMed ID: 24336934
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  • 20. Hyperosmotic stress strongly potentiates serum response factor (SRF)-dependent transcriptional activity in Ehrlich Lettré Ascites cells through a mechanism involving p38 mitogen-activated protein kinase.
    Gorbatenko A, Wiwel M, Klingberg H, Nielsen AB, Kapus A, Pedersen SF.
    J Cell Physiol; 2011 Nov; 226(11):2857-68. PubMed ID: 21302281
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