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127 related items for PubMed ID: 27826738
1. Transcription factor PEX1 modulates extracellular matrix turnover through regulation of MMP-9 expression. Jurado Acosta A, Rysä J, Szabo Z, Moilanen AM, Komati H, Nemer M, Ruskoaho H. Cell Tissue Res; 2017 Feb; 367(2):369-385. PubMed ID: 27826738 [Abstract] [Full Text] [Related]
2. (Pro)renin receptor triggers distinct angiotensin II-independent extracellular matrix remodeling and deterioration of cardiac function. Moilanen AM, Rysä J, Serpi R, Mustonen E, Szabò Z, Aro J, Näpänkangas J, Tenhunen O, Sutinen M, Salo T, Ruskoaho H. PLoS One; 2012 Feb; 7(7):e41404. PubMed ID: 22911790 [Abstract] [Full Text] [Related]
3. Changes in extracellular matrix and in transforming growth factor beta isoforms after coronary artery ligation in rats. Deten A, Hölzl A, Leicht M, Barth W, Zimmer HG. J Mol Cell Cardiol; 2001 Jun; 33(6):1191-207. PubMed ID: 11444923 [Abstract] [Full Text] [Related]
4. The zinc finger-only protein Zfp260 is a novel cardiac regulator and a nuclear effector of alpha1-adrenergic signaling. Debrus S, Rahbani L, Marttila M, Delorme B, Paradis P, Nemer M. Mol Cell Biol; 2005 Oct; 25(19):8669-82. PubMed ID: 16166646 [Abstract] [Full Text] [Related]
5. Adiponectin attenuates profibrotic extracellular matrix remodeling following cardiac injury by up-regulating matrix metalloproteinase 9 expression in mice. Jenke A, Schur R, Röger C, Karadeniz Z, Grüger M, Holzhauser L, Savvatis K, Poller W, Schultheiss HP, Landmesser U, Skurk C. Physiol Rep; 2017 Dec; 5(24):. PubMed ID: 29263115 [Abstract] [Full Text] [Related]
6. Single-target RNA interference for the blockade of multiple interacting proinflammatory and profibrotic pathways in cardiac fibroblasts. Tank J, Lindner D, Wang X, Stroux A, Gilke L, Gast M, Zietsch C, Skurk C, Scheibenbogen C, Klingel K, Lassner D, Kühl U, Schultheiss HP, Westermann D, Poller W. J Mol Cell Cardiol; 2014 Jan; 66():141-56. PubMed ID: 24239602 [Abstract] [Full Text] [Related]
7. A functional activating protein 1 (AP-1) site regulates matrix metalloproteinase 2 (MMP-2) transcription by cardiac cells through interactions with JunB-Fra1 and JunB-FosB heterodimers. Bergman MR, Cheng S, Honbo N, Piacentini L, Karliner JS, Lovett DH. Biochem J; 2003 Feb 01; 369(Pt 3):485-96. PubMed ID: 12371906 [Abstract] [Full Text] [Related]
8. Direct actions of urotensin II on the heart: implications for cardiac fibrosis and hypertrophy. Tzanidis A, Hannan RD, Thomas WG, Onan D, Autelitano DJ, See F, Kelly DJ, Gilbert RE, Krum H. Circ Res; 2003 Aug 08; 93(3):246-53. PubMed ID: 12842917 [Abstract] [Full Text] [Related]
9. A-kinase anchoring protein-Lbc promotes pro-fibrotic signaling in cardiac fibroblasts. Cavin S, Maric D, Diviani D. Biochim Biophys Acta; 2014 Feb 08; 1843(2):335-45. PubMed ID: 24269843 [Abstract] [Full Text] [Related]
10. Angiotensin type 2 receptor stimulation ameliorates left ventricular fibrosis and dysfunction via regulation of tissue inhibitor of matrix metalloproteinase 1/matrix metalloproteinase 9 axis and transforming growth factor β1 in the rat heart. Lauer D, Slavic S, Sommerfeld M, Thöne-Reineke C, Sharkovska Y, Hallberg A, Dahlöf B, Kintscher U, Unger T, Steckelings UM, Kaschina E. Hypertension; 2014 Mar 08; 63(3):e60-7. PubMed ID: 24379181 [Abstract] [Full Text] [Related]
11. Interplay of matrix metalloproteinases, tissue inhibitors of metalloproteinases and their regulators in cardiac matrix remodeling. Li YY, McTiernan CF, Feldman AM. Cardiovasc Res; 2000 May 08; 46(2):214-24. PubMed ID: 10773225 [Abstract] [Full Text] [Related]
12. Regulation of fibronectin gene expression in cardiac fibroblasts by scleraxis. Bagchi RA, Lin J, Wang R, Czubryt MP. Cell Tissue Res; 2016 Nov 08; 366(2):381-391. PubMed ID: 27324126 [Abstract] [Full Text] [Related]
13. miR-133 and miR-30 regulate connective tissue growth factor: implications for a role of microRNAs in myocardial matrix remodeling. Duisters RF, Tijsen AJ, Schroen B, Leenders JJ, Lentink V, van der Made I, Herias V, van Leeuwen RE, Schellings MW, Barenbrug P, Maessen JG, Heymans S, Pinto YM, Creemers EE. Circ Res; 2009 Jan 30; 104(2):170-8, 6p following 178. PubMed ID: 19096030 [Abstract] [Full Text] [Related]
14. Intramyocardial BNP gene delivery improves cardiac function through distinct context-dependent mechanisms. Moilanen AM, Rysä J, Mustonen E, Serpi R, Aro J, Tokola H, Leskinen H, Manninen A, Levijoki J, Vuolteenaho O, Ruskoaho H. Circ Heart Fail; 2011 Jul 30; 4(4):483-95. PubMed ID: 21558448 [Abstract] [Full Text] [Related]
15. Fibroblast growth factor 23 is induced by an activated renin-angiotensin-aldosterone system in cardiac myocytes and promotes the pro-fibrotic crosstalk between cardiac myocytes and fibroblasts. Leifheit-Nestler M, Kirchhoff F, Nespor J, Richter B, Soetje B, Klintschar M, Heineke J, Haffner D. Nephrol Dial Transplant; 2018 Oct 01; 33(10):1722-1734. PubMed ID: 29425341 [Abstract] [Full Text] [Related]
16. Regulation of matrix metalloproteinase MT1-MMP/MMP-2 in cardiac fibroblasts by TGF-beta1 involves furin-convertase. Stawowy P, Margeta C, Kallisch H, Seidah NG, Chrétien M, Fleck E, Graf K. Cardiovasc Res; 2004 Jul 01; 63(1):87-97. PubMed ID: 15194465 [Abstract] [Full Text] [Related]
17. Notch3 Ameliorates Cardiac Fibrosis After Myocardial Infarction by Inhibiting the TGF-β1/Smad3 Pathway. Zhang M, Pan X, Zou Q, Xia Y, Chen J, Hao Q, Wang H, Sun D. Cardiovasc Toxicol; 2016 Oct 01; 16(4):316-24. PubMed ID: 26487518 [Abstract] [Full Text] [Related]
18. Phosphorylation of GATA4 at serine 105 is required for left ventricular remodelling process in angiotensin II-induced hypertension in rats. Jurado Acosta A, Rysä J, Szabo Z, Moilanen AM, Serpi R, Ruskoaho H. Basic Clin Pharmacol Toxicol; 2020 Sep 01; 127(3):178-195. PubMed ID: 32060996 [Abstract] [Full Text] [Related]
19. CTRP3 attenuates post-infarct cardiac fibrosis by targeting Smad3 activation and inhibiting myofibroblast differentiation. Wu D, Lei H, Wang JY, Zhang CL, Feng H, Fu FY, Li L, Wu LL. J Mol Med (Berl); 2015 Dec 01; 93(12):1311-25. PubMed ID: 26138247 [Abstract] [Full Text] [Related]
20. Peroxisome proliferator-activated receptor alpha-independent actions of fenofibrate exacerbates left ventricular dilation and fibrosis in chronic pressure overload. Duhaney TA, Cui L, Rude MK, Lebrasseur NK, Ngoy S, De Silva DS, Siwik DA, Liao R, Sam F. Hypertension; 2007 May 01; 49(5):1084-94. PubMed ID: 17353509 [Abstract] [Full Text] [Related] Page: [Next] [New Search]