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Journal Abstract Search
255 related items for PubMed ID: 27999940
1. FHF2 isoforms differentially regulate Nav1.6-mediated resurgent sodium currents in dorsal root ganglion neurons. Barbosa C, Xiao Y, Johnson AJ, Xie W, Strong JA, Zhang JM, Cummins TR. Pflugers Arch; 2017 Feb; 469(2):195-212. PubMed ID: 27999940 [Abstract] [Full Text] [Related]
5. Human Nav1.6 Channels Generate Larger Resurgent Currents than Human Nav1.1 Channels, but the Navβ4 Peptide Does Not Protect Either Isoform from Use-Dependent Reduction. Patel RR, Barbosa C, Xiao Y, Cummins TR. PLoS One; 2015 Nov 01; 10(7):e0133485. PubMed ID: 26182346 [Abstract] [Full Text] [Related]
14. Role of NaV1.6 and NaVβ4 Sodium Channel Subunits in a Rat Model of Low Back Pain Induced by Compression of the Dorsal Root Ganglia. Xie W, Zhang J, Strong JA, Zhang JM. Neuroscience; 2019 Mar 15; 402():51-65. PubMed ID: 30699332 [Abstract] [Full Text] [Related]
15. Interactions among DIV voltage-sensor movement, fast inactivation, and resurgent Na current induced by the NaVβ4 open-channel blocking peptide. Lewis AH, Raman IM. J Gen Physiol; 2013 Sep 15; 142(3):191-206. PubMed ID: 23940261 [Abstract] [Full Text] [Related]
17. Production of resurgent current in NaV1.6-null Purkinje neurons by slowing sodium channel inactivation with beta-pompilidotoxin. Grieco TM, Raman IM. J Neurosci; 2004 Jan 07; 24(1):35-42. PubMed ID: 14715935 [Abstract] [Full Text] [Related]