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Journal Abstract Search

401 related items for PubMed ID: 2804076

  • 1. Glucose oxidation rates in fatty acid-perfused isolated working hearts from diabetic rats.
    Wall SR, Lopaschuk GD.
    Biochim Biophys Acta; 1989 Nov 06; 1006(1):97-103. PubMed ID: 2804076
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  • 2. Response of isolated working hearts to fatty acids and carnitine palmitoyltransferase I inhibition during reduction of coronary flow in acutely and chronically diabetic rats.
    Lopaschuk GD, Spafford M.
    Circ Res; 1989 Aug 06; 65(2):378-87. PubMed ID: 2526694
    [Abstract] [Full Text] [Related]

  • 3. Glucose oxidation is stimulated in reperfused ischemic hearts with the carnitine palmitoyltransferase 1 inhibitor, Etomoxir.
    Lopaschuk GD, McNeil GF, McVeigh JJ.
    Mol Cell Biochem; 1989 Aug 06; 88(1-2):175-9. PubMed ID: 2779537
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  • 4. Carnitine stimulation of glucose oxidation in the fatty acid perfused isolated working rat heart.
    Broderick TL, Quinney HA, Lopaschuk GD.
    J Biol Chem; 1992 Feb 25; 267(6):3758-63. PubMed ID: 1740427
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  • 5. Glucose and palmitate oxidation in isolated working rat hearts reperfused after a period of transient global ischemia.
    Lopaschuk GD, Spafford MA, Davies NJ, Wall SR.
    Circ Res; 1990 Feb 25; 66(2):546-53. PubMed ID: 2297817
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  • 6. Etomoxir, a carnitine palmitoyltransferase I inhibitor, protects hearts from fatty acid-induced ischemic injury independent of changes in long chain acylcarnitine.
    Lopaschuk GD, Wall SR, Olley PM, Davies NJ.
    Circ Res; 1988 Dec 25; 63(6):1036-43. PubMed ID: 3197271
    [Abstract] [Full Text] [Related]

  • 7. Triacylglycerol turnover in isolated working hearts of acutely diabetic rats.
    Saddik M, Lopaschuk GD.
    Can J Physiol Pharmacol; 1994 Oct 25; 72(10):1110-9. PubMed ID: 7882174
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  • 8. Myocardial triglyceride turnover and contribution to energy substrate utilization in isolated working rat hearts.
    Saddik M, Lopaschuk GD.
    J Biol Chem; 1991 May 05; 266(13):8162-70. PubMed ID: 1902472
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  • 11. Contribution of malonyl-CoA decarboxylase to the high fatty acid oxidation rates seen in the diabetic heart.
    Sakamoto J, Barr RL, Kavanagh KM, Lopaschuk GD.
    Am J Physiol Heart Circ Physiol; 2000 Apr 05; 278(4):H1196-204. PubMed ID: 10749714
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  • 12. Fatty acid metabolism in hearts containing elevated levels of CoA.
    Lopaschuk GD, Hansen CA, Neely JR.
    Am J Physiol; 1986 Mar 05; 250(3 Pt 2):H351-9. PubMed ID: 3953832
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  • 13. Inhibition of carnitine palmitoyltransferase 1 by phenylalkyloxiranecarboxylic acid and its influence on lipolysis and glucose metabolism in isolated, perfused hearts of streptozotocin-diabetic rats.
    Rösen P, Reinauer H.
    Metabolism; 1984 Feb 05; 33(2):177-85. PubMed ID: 6694560
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  • 14. Fatty acid oxidation and cardiac function in the sodium pivalate model of secondary carnitine deficiency.
    Broderick TL, Christos SC, Wolf BA, DiDomenico D, Shug AL, Paulson DJ.
    Metabolism; 1995 Apr 05; 44(4):499-505. PubMed ID: 7723673
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  • 15. Calcium regulation of glycolysis, glucose oxidation, and fatty acid oxidation in the aerobic and ischemic heart.
    Schönekess BO, Brindley PG, Lopaschuk GD.
    Can J Physiol Pharmacol; 1995 Nov 05; 73(11):1632-40. PubMed ID: 8789418
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  • 18. Propionyl-L-carnitine effects on postischemic recovery of heart function and substrate oxidation in the diabetic rat.
    Broderick TL, Driedzic W, Paulson DJ.
    Mol Cell Biochem; 2000 Mar 05; 206(1-2):151-7. PubMed ID: 10839205
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  • 19. Effects of high levels of fatty acids on functional recovery of ischemic hearts from diabetic rats.
    Lopaschuk GD, Saddik M, Barr R, Huang L, Barker CC, Muzyka RA.
    Am J Physiol; 1992 Dec 05; 263(6):E1046-53. PubMed ID: 1476176
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