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366 related items for PubMed ID: 28062479
1. Loss of Macrophage Wnt Secretion Improves Remodeling and Function After Myocardial Infarction in Mice. Palevski D, Levin-Kotler LP, Kain D, Naftali-Shani N, Landa N, Ben-Mordechai T, Konfino T, Holbova R, Molotski N, Rosin-Arbesfeld R, Lang RA, Leor J. J Am Heart Assoc; 2017 Jan 06; 6(1):. PubMed ID: 28062479 [Abstract] [Full Text] [Related]
2. Targeting and modulating infarct macrophages with hemin formulated in designed lipid-based particles improves cardiac remodeling and function. Ben-Mordechai T, Kain D, Holbova R, Landa N, Levin LP, Elron-Gross I, Glucksam-Galnoy Y, Feinberg MS, Margalit R, Leor J. J Control Release; 2017 Jul 10; 257():21-31. PubMed ID: 28065861 [Abstract] [Full Text] [Related]
5. Cardiac CD68+ and stabilin-1+ macrophages in wound healing following myocardial infarction: From experiment to clinic. Ryabov V, Gombozhapova A, Rogovskaya Y, Kzhyshkowska J, Rebenkova M, Karpov R. Immunobiology; 2018 Jul 10; 223(4-5):413-421. PubMed ID: 29179985 [Abstract] [Full Text] [Related]
7. Long-Term Administration of Eicosapentaenoic Acid Improves Post-Myocardial Infarction Cardiac Remodeling in Mice by Regulating Macrophage Polarization. Takamura M, Kurokawa K, Ootsuji H, Inoue O, Okada H, Nomura A, Kaneko S, Usui S. J Am Heart Assoc; 2017 Feb 21; 6(2):. PubMed ID: 28223437 [Abstract] [Full Text] [Related]
8. HDAC inhibition helps post-MI healing by modulating macrophage polarization. Kimbrough D, Wang SH, Wright LH, Mani SK, Kasiganesan H, LaRue AC, Cheng Q, Nadig SN, Atkinson C, Menick DR. J Mol Cell Cardiol; 2018 Jun 21; 119():51-63. PubMed ID: 29680681 [Abstract] [Full Text] [Related]
9. Lp-PLA2 Antagonizes Left Ventricular Healing After Myocardial Infarction by Impairing the Appearance of Reparative Macrophages. He S, Chousterman BG, Fenn A, Anzai A, Nairz M, Brandt M, Hilgendorf I, Sun Y, Ye YX, Iwamoto Y, Tricot B, Weissleder R, Macphee C, Libby P, Nahrendorf M, Swirski FK. Circ Heart Fail; 2015 Sep 21; 8(5):980-7. PubMed ID: 26232205 [Abstract] [Full Text] [Related]
10. MAIR-II deficiency ameliorates cardiac remodelling post-myocardial infarction by suppressing TLR9-mediated macrophage activation. Yonebayashi S, Tajiri K, Murakoshi N, Xu D, Li S, Feng D, Okabe Y, Yuan Z, Song Z, Aonuma K, Shibuya A, Aonuma K, Ieda M. J Cell Mol Med; 2020 Dec 21; 24(24):14481-14490. PubMed ID: 33140535 [Abstract] [Full Text] [Related]
11. Cardiac overexpression of monocyte chemoattractant protein-1 in transgenic mice prevents cardiac dysfunction and remodeling after myocardial infarction. Morimoto H, Takahashi M, Izawa A, Ise H, Hongo M, Kolattukudy PE, Ikeda U. Circ Res; 2006 Oct 13; 99(8):891-9. PubMed ID: 16990567 [Abstract] [Full Text] [Related]
12. IL33 attenuates ventricular remodeling after myocardial infarction through inducing alternatively activated macrophages ethical standards statement. Li J, Shen D, Tang J, Wang Y, Wang B, Xiao Y, Cao C, Shi X, Liu HM, Zhao W, Zhang J. Eur J Pharmacol; 2019 Jul 05; 854():307-319. PubMed ID: 31039343 [Abstract] [Full Text] [Related]
13. Azithromycin therapy reduces cardiac inflammation and mitigates adverse cardiac remodeling after myocardial infarction: Potential therapeutic targets in ischemic heart disease. Al-Darraji A, Haydar D, Chelvarajan L, Tripathi H, Levitan B, Gao E, Venditto VJ, Gensel JC, Feola DJ, Abdel-Latif A. PLoS One; 2018 Jul 05; 13(7):e0200474. PubMed ID: 30001416 [Abstract] [Full Text] [Related]
14. Class A scavenger receptor attenuates myocardial infarction-induced cardiomyocyte necrosis through suppressing M1 macrophage subset polarization. Hu Y, Zhang H, Lu Y, Bai H, Xu Y, Zhu X, Zhou R, Ben J, Xu Y, Chen Q. Basic Res Cardiol; 2011 Nov 05; 106(6):1311-28. PubMed ID: 21769674 [Abstract] [Full Text] [Related]
15. Interleukin-13 deficiency aggravates healing and remodeling in male mice after experimental myocardial infarction. Hofmann U, Knorr S, Vogel B, Weirather J, Frey A, Ertl G, Frantz S. Circ Heart Fail; 2014 Sep 05; 7(5):822-30. PubMed ID: 24970469 [Abstract] [Full Text] [Related]
16. The cardiac microenvironment uses non-canonical WNT signaling to activate monocytes after myocardial infarction. Meyer IS, Jungmann A, Dieterich C, Zhang M, Lasitschka F, Werkmeister S, Haas J, Müller OJ, Boutros M, Nahrendorf M, Katus HA, Hardt SE, Leuschner F. EMBO Mol Med; 2017 Sep 05; 9(9):1279-1293. PubMed ID: 28774883 [Abstract] [Full Text] [Related]
17. KDM3A inhibition modulates macrophage polarization to aggravate post-MI injuries and accelerates adverse ventricular remodeling via an IRF4 signaling pathway. Liu X, Chen J, Zhang B, Liu G, Zhao H, Hu Q. Cell Signal; 2019 Dec 05; 64():109415. PubMed ID: 31513837 [Abstract] [Full Text] [Related]
18. Splenic Ly6Chi monocytes contribute to adverse late post-ischemic left ventricular remodeling in heme oxygenase-1 deficient mice. Tomczyk M, Kraszewska I, Szade K, Bukowska-Strakova K, Meloni M, Jozkowicz A, Dulak J, Jazwa A. Basic Res Cardiol; 2017 Jul 05; 112(4):39. PubMed ID: 28534119 [Abstract] [Full Text] [Related]
20. The apical and basolateral secretion of Wnt11 and Wnt3a in polarized epithelial cells is regulated by different mechanisms. Yamamoto H, Awada C, Hanaki H, Sakane H, Tsujimoto I, Takahashi Y, Takao T, Kikuchi A. J Cell Sci; 2013 Jul 01; 126(Pt 13):2931-43. PubMed ID: 23613470 [Abstract] [Full Text] [Related] Page: [Next] [New Search]