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Journal Abstract Search

358 related items for PubMed ID: 28338646

  • 21. Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system.
    Backert S, Tegtmeyer N, Fischer W.
    Future Microbiol; 2015; 10(6):955-65. PubMed ID: 26059619
    [Abstract] [Full Text] [Related]

  • 22. Signal transduction of Helicobacter pylori during interaction with host cell protein receptors of epithelial and immune cells.
    Pachathundikandi SK, Tegtmeyer N, Backert S.
    Gut Microbes; 2013; 4(6):454-74. PubMed ID: 24280762
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  • 25. Helicobacter pylori CagL Y58/E59 mutation turns-off type IV secretion-dependent delivery of CagA into host cells.
    Tegtmeyer N, Lind J, Schmid B, Backert S.
    PLoS One; 2014; 9(6):e97782. PubMed ID: 24893039
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  • 26. Phosphorylation of Helicobacter pylori CagA by c-Abl leads to cell motility.
    Poppe M, Feller SM, Römer G, Wessler S.
    Oncogene; 2007 May 24; 26(24):3462-72. PubMed ID: 17160020
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  • 27. HopQ impacts the integrin α5β1-independent NF-κB activation by Helicobacter pylori in CEACAM expressing cells.
    Feige MH, Sokolova O, Pickenhahn A, Maubach G, Naumann M.
    Int J Med Microbiol; 2018 Jul 24; 308(5):527-533. PubMed ID: 29779861
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  • 28. Helicobacter pylori CagA interacts with E-cadherin and deregulates the beta-catenin signal that promotes intestinal transdifferentiation in gastric epithelial cells.
    Murata-Kamiya N, Kurashima Y, Teishikata Y, Yamahashi Y, Saito Y, Higashi H, Aburatani H, Akiyama T, Peek RM, Azuma T, Hatakeyama M.
    Oncogene; 2007 Jul 12; 26(32):4617-26. PubMed ID: 17237808
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  • 29. Structural Insights into Helicobacter pylori Cag Protein Interactions with Host Cell Factors.
    Bergé C, Terradot L.
    Curr Top Microbiol Immunol; 2017 Jul 12; 400():129-147. PubMed ID: 28124152
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  • 30. Integrin but not CEACAM receptors are dispensable for Helicobacter pylori CagA translocation.
    Zhao Q, Busch B, Jiménez-Soto LF, Ishikawa-Ankerhold H, Massberg S, Terradot L, Fischer W, Haas R.
    PLoS Pathog; 2018 Oct 12; 14(10):e1007359. PubMed ID: 30365569
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  • 31. Outer inflammatory protein a (OipA) of Helicobacter pylori is regulated by host cell contact and mediates CagA translocation and interleukin-8 response only in the presence of a functional cag pathogenicity island type IV secretion system.
    Horridge DN, Begley AA, Kim J, Aravindan N, Fan K, Forsyth MH.
    Pathog Dis; 2017 Nov 30; 75(8):. PubMed ID: 29040466
    [Abstract] [Full Text] [Related]

  • 32. Cortactin Is Required for Efficient FAK, Src and Abl Tyrosine Kinase Activation and Phosphorylation of Helicobacter pylori CagA.
    Knorr J, Sharafutdinov I, Fiedler F, Soltan Esmaeili D, Rohde M, Rottner K, Backert S, Tegtmeyer N.
    Int J Mol Sci; 2021 Jun 03; 22(11):. PubMed ID: 34205064
    [Abstract] [Full Text] [Related]

  • 33. The versatility of Helicobacter pylori CagA effector protein functions: The master key hypothesis.
    Backert S, Tegtmeyer N, Selbach M.
    Helicobacter; 2010 Jun 03; 15(3):163-76. PubMed ID: 20557357
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  • 34. Attenuation of Helicobacter pylori CagA x SHP-2 signaling by interaction between CagA and C-terminal Src kinase.
    Tsutsumi R, Higashi H, Higuchi M, Okada M, Hatakeyama M.
    J Biol Chem; 2003 Feb 07; 278(6):3664-70. PubMed ID: 12446738
    [Abstract] [Full Text] [Related]

  • 35. The Middle Fragment of Helicobacter pylori CagA Induces Actin Rearrangement and Triggers Its Own Uptake into Gastric Epithelial Cells.
    Tohidpour A, Gorrell RJ, Roujeinikova A, Kwok T.
    Toxins (Basel); 2017 Jul 28; 9(8):. PubMed ID: 28788072
    [Abstract] [Full Text] [Related]

  • 36. CagI is an essential component of the Helicobacter pylori Cag type IV secretion system and forms a complex with CagL.
    Pham KT, Weiss E, Jiménez Soto LF, Breithaupt U, Haas R, Fischer W.
    PLoS One; 2012 Jul 28; 7(4):e35341. PubMed ID: 22493745
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  • 37. The Helicobacter pylori CagA protein induces cortactin dephosphorylation and actin rearrangement by c-Src inactivation.
    Selbach M, Moese S, Hurwitz R, Hauck CR, Meyer TF, Backert S.
    EMBO J; 2003 Feb 03; 22(3):515-28. PubMed ID: 12554652
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  • 38. Host SHP1 phosphatase antagonizes Helicobacter pylori CagA and can be downregulated by Epstein-Barr virus.
    Saju P, Murata-Kamiya N, Hayashi T, Senda Y, Nagase L, Noda S, Matsusaka K, Funata S, Kunita A, Urabe M, Seto Y, Fukayama M, Kaneda A, Hatakeyama M.
    Nat Microbiol; 2016 Mar 14; 1():16026. PubMed ID: 27572445
    [Abstract] [Full Text] [Related]

  • 39. A novel NOD1- and CagA-independent pathway of interleukin-8 induction mediated by the Helicobacter pylori type IV secretion system.
    Gorrell RJ, Guan J, Xin Y, Tafreshi MA, Hutton ML, McGuckin MA, Ferrero RL, Kwok T.
    Cell Microbiol; 2013 Apr 14; 15(4):554-70. PubMed ID: 23107019
    [Abstract] [Full Text] [Related]

  • 40. Focal adhesion kinase is a substrate and downstream effector of SHP-2 complexed with Helicobacter pylori CagA.
    Tsutsumi R, Takahashi A, Azuma T, Higashi H, Hatakeyama M.
    Mol Cell Biol; 2006 Jan 14; 26(1):261-76. PubMed ID: 16354697
    [Abstract] [Full Text] [Related]

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