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258 related items for PubMed ID: 28365586

  • 1. ENaC and ROMK activity are inhibited in the DCT2/CNT of TgWnk4PHAII mice.
    Zhang C, Wang L, Su XT, Zhang J, Lin DH, Wang WH.
    Am J Physiol Renal Physiol; 2017 Apr 01; 312(4):F682-F688. PubMed ID: 28365586
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  • 2. Role of WNK4 and kidney-specific WNK1 in mediating the effect of high dietary K+ intake on ROMK channel in the distal convoluted tubule.
    Wu P, Gao ZX, Su XT, Ellison DH, Hadchouel J, Teulon J, Wang WH.
    Am J Physiol Renal Physiol; 2018 Aug 01; 315(2):F223-F230. PubMed ID: 29667910
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  • 3. High baseline ROMK activity in the mouse late distal convoluted and early connecting tubule probably contributes to aldosterone-independent K+ secretion.
    Nesterov V, Bertog M, Korbmacher C.
    Am J Physiol Renal Physiol; 2022 Jan 01; 322(1):F42-F54. PubMed ID: 34843658
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  • 5. The effect of high-dietary K+ (HK) on Kir4.1/Kir5.1 and ROMK in the distal convoluted tubule (DCT) is not affected by gender and Cl- content of the diet.
    Meng XX, Zhang H, Meng GL, Jiang SP, Duan XP, Wang WH, Wang MX.
    Front Physiol; 2022 Jan 01; 13():1039029. PubMed ID: 36439248
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  • 6. Wnk4 controls blood pressure and potassium homeostasis via regulation of mass and activity of the distal convoluted tubule.
    Lalioti MD, Zhang J, Volkman HM, Kahle KT, Hoffmann KE, Toka HR, Nelson-Williams C, Ellison DH, Flavell R, Booth CJ, Lu Y, Geller DS, Lifton RP.
    Nat Genet; 2006 Oct 01; 38(10):1124-32. PubMed ID: 16964266
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  • 7. Effect of Angiotensin II on ENaC in the Distal Convoluted Tubule and in the Cortical Collecting Duct of Mineralocorticoid Receptor Deficient Mice.
    Wu P, Gao ZX, Zhang DD, Duan XP, Terker AS, Lin DH, Ellison DH, Wang WH.
    J Am Heart Assoc; 2020 Apr 07; 9(7):e014996. PubMed ID: 32208832
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  • 11. Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules.
    Grimm PR, Coleman R, Delpire E, Welling PA.
    J Am Soc Nephrol; 2017 Sep 07; 28(9):2597-2606. PubMed ID: 28442491
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  • 13. Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS.
    Zhang DD, Duan XP, Xiao Y, Wu P, Gao ZX, Wang WH, Lin DH.
    Am J Physiol Renal Physiol; 2021 May 01; 320(5):F883-F896. PubMed ID: 33818128
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  • 14. In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron.
    Nesterov V, Krueger B, Bertog M, Dahlmann A, Palmisano R, Korbmacher C.
    Hypertension; 2016 Jun 01; 67(6):1256-62. PubMed ID: 27170740
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  • 16. Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting duct.
    Su XT, Zhang C, Wang L, Gu R, Lin DH, Wang WH.
    Am J Physiol Renal Physiol; 2016 May 01; 310(10):F985-93. PubMed ID: 26887833
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  • 18. Mouse model of type II Bartter's syndrome. II. Altered expression of renal sodium- and water-transporting proteins.
    Wagner CA, Loffing-Cueni D, Yan Q, Schulz N, Fakitsas P, Carrel M, Wang T, Verrey F, Geibel JP, Giebisch G, Hebert SC, Loffing J.
    Am J Physiol Renal Physiol; 2008 Jun 01; 294(6):F1373-80. PubMed ID: 18322017
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  • 19. Deletion of renal Nedd4-2 abolishes the effect of high K+ intake on Kir4.1/Kir5.1 and NCC activity in the distal convoluted tubule.
    Xiao Y, Duan XP, Zhang DD, Wang WH, Lin DH.
    Am J Physiol Renal Physiol; 2021 Jul 01; 321(1):F1-F11. PubMed ID: 34029145
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  • 20. Molecular pathogenesis of pseudohypoaldosteronism type II: generation and analysis of a Wnk4(D561A/+) knockin mouse model.
    Yang SS, Morimoto T, Rai T, Chiga M, Sohara E, Ohno M, Uchida K, Lin SH, Moriguchi T, Shibuya H, Kondo Y, Sasaki S, Uchida S.
    Cell Metab; 2007 May 01; 5(5):331-44. PubMed ID: 17488636
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