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206 related items for PubMed ID: 2839621

  • 1. Is inositol bisphosphate the product of A23187 and carbachol-mediated polyphosphoinositide breakdown in synaptosomes?
    Brammer MJ, Hajimohammadreza I, Sardiwal S, Weaver K.
    J Neurochem; 1988 Aug; 51(2):514-21. PubMed ID: 2839621
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  • 2. Kinetic analysis of A23187-mediated polyphosphoinositide breakdown in rat cortical synaptosomes suggests that inositol bisphosphate does not arise primarily by degradation of inositol trisphosphate.
    Brammer M, Weaver K.
    J Neurochem; 1989 Aug; 53(2):399-407. PubMed ID: 2545817
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  • 3. Carbachol causes rapid phosphodiesteratic cleavage of phosphatidylinositol 4,5-bisphosphate and accumulation of inositol phosphates in rabbit iris smooth muscle; prazosin inhibits noradrenaline- and ionophore A23187-stimulated accumulation of inositol phosphates.
    Akhtar RA, Abdel-Latif AA.
    Biochem J; 1984 Nov 15; 224(1):291-300. PubMed ID: 6095818
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  • 9. Effects of in vitro hypoxia on depolarization-stimulated accumulation of inositol phosphates in synaptosomes.
    Huang HM, Gibson GE.
    Life Sci; 1989 Nov 15; 45(16):1443-9. PubMed ID: 2811601
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  • 10. Potassium depolarisation markedly enhances muscarinic receptor stimulated inositol tetrakisphosphate accumulation in rat cerebral cortical slices.
    Baird JG, Nahorski SR.
    Biochem Biophys Res Commun; 1986 Dec 30; 141(3):1130-7. PubMed ID: 3814117
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  • 12. Inositol 1,3,4,5-tetrakisphosphate and not phosphatidylinositol 3,4-bisphosphate is the probable precursor of inositol 1,3,4-trisphosphate in agonist-stimulated parotid gland.
    Downes CP, Hawkins PT, Irvine RF.
    Biochem J; 1986 Sep 01; 238(2):501-6. PubMed ID: 2432882
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  • 13. Gonadotropin-releasing hormone activates a rapid Ca2+-independent phosphodiester hydrolysis of polyphosphoinositides in pituitary gonadotrophs.
    Naor Z, Azrad A, Limor R, Zakut H, Lotan M.
    J Biol Chem; 1986 Sep 25; 261(27):12506-12. PubMed ID: 3017978
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  • 14. Metabolism of inositol phosphates in parotid cells: implications for the pathway of the phosphoinositide effect and for the possible messenger role of inositol trisphosphate.
    Aub DL, Putney JW.
    Life Sci; 1984 Apr 02; 34(14):1347-55. PubMed ID: 6323902
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  • 15. Inositol 1,2-cyclic 4,5-trisphosphate is not a product of muscarinic receptor-stimulated phosphatidylinositol 4,5-bisphosphate hydrolysis in rat parotid glands.
    Hawkins PT, Berrie CP, Morris AJ, Downes CP.
    Biochem J; 1987 Apr 01; 243(1):211-8. PubMed ID: 3038079
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  • 16. Inositol phospholipid hydrolysis in rat cerebral cortical slices: II. Calcium requirement.
    Kendall DA, Nahorski SR.
    J Neurochem; 1984 May 01; 42(5):1388-94. PubMed ID: 6423773
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  • 17. Carbamoylcholine-induced accumulation of inositol mono-, bis-, tris- and tetrakisphosphates in isolated cardiac myocytes from adult rats.
    Berg I, Guse AH, Gercken G.
    Biochim Biophys Acta; 1989 Jan 17; 1010(1):100-7. PubMed ID: 2909244
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  • 18. Enhancement by potassium of carbachol-stimulated inositol phospholipid breakdown in rat cerebral cortical miniprisms: comparison with other depolarising agents.
    Tiger G, Björklund PE, Cowburn RF, Fowler CJ.
    J Neurochem; 1989 Jun 17; 52(6):1843-53. PubMed ID: 2470855
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  • 19. A specific transduction mechanism for the glutamate action on phosphoinositide metabolism via the quisqualate metabotropic receptor in rat brain synaptoneurosomes: II. Calcium dependency, cadmium inhibition.
    Guiramand J, Vignes M, Récasens M.
    J Neurochem; 1991 Nov 17; 57(5):1501-9. PubMed ID: 1681030
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  • 20. Source of 3H-labeled inositol bis- and monophosphates in agonist-activated rat parotid acinar cells.
    Hughes AR, Putney JW.
    J Biol Chem; 1989 Jun 05; 264(16):9400-7. PubMed ID: 2542308
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