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Journal Abstract Search

565 related items for PubMed ID: 28400540

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  • 2. Increased CD56(bright) NK cells in HIV-HCV co-infection and HCV mono-infection are associated with distinctive alterations of their phenotype.
    Bhardwaj S, Ahmad F, Wedemeyer H, Cornberg M, Schulze Zur Wiesch J, van Lunzen J, Sarin SK, Schmidt RE, Meyer-Olson D.
    Virol J; 2016 Apr 18; 13():67. PubMed ID: 27091211
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  • 4. CD56(+dim) and CD56(+bright) cell activation and apoptosis in hepatitis C virus infection.
    Lin AW, Gonzalez SA, Cunningham-Rundles S, Dorante G, Marshall S, Tignor A, Ha C, Jacobson IM, Talal AH.
    Clin Exp Immunol; 2004 Aug 18; 137(2):408-16. PubMed ID: 15270860
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  • 5. Distinct natural killer cells in HIV-exposed seronegative subjects with effector cytotoxic CD56(dim) and CD56(bright) cells and memory-like CD57⁺NKG2C⁺CD56(dim) cells.
    Lima JF, Oliveira LM, Pereira NZ, Mitsunari GE, Duarte AJ, Sato MN.
    J Acquir Immune Defic Syndr; 2014 Dec 15; 67(5):463-71. PubMed ID: 25230289
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  • 6. Deficient IL-2 Produced by Activated CD56+ T Cells Contributes to Impaired NK Cell-Mediated ADCC Function in Chronic HIV-1 Infection.
    Xie Z, Zheng J, Wang Y, Li D, Maermaer T, Li Y, Tu J, Xu Q, Liang H, Cai W, Shen T.
    Front Immunol; 2019 Dec 15; 10():1647. PubMed ID: 31379845
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  • 10. Expansion of CD56- NK cells in chronic HCV/HIV-1 co-infection: reversion by antiviral treatment with pegylated IFNalpha and ribavirin.
    Gonzalez VD, Falconer K, Michaëlsson J, Moll M, Reichard O, Alaeus A, Sandberg JK.
    Clin Immunol; 2008 Jul 15; 128(1):46-56. PubMed ID: 18495540
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  • 11. Expansion of functionally skewed CD56-negative NK cells in chronic hepatitis C virus infection: correlation with outcome of pegylated IFN-alpha and ribavirin treatment.
    Gonzalez VD, Falconer K, Björkström NK, Blom KG, Weiland O, Ljunggren HG, Alaeus A, Sandberg JK.
    J Immunol; 2009 Nov 15; 183(10):6612-8. PubMed ID: 19846870
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  • 13. Altered natural killer cell subset distributions in resolved and persistent hepatitis C virus infection following single source exposure.
    Golden-Mason L, Madrigal-Estebas L, McGrath E, Conroy MJ, Ryan EJ, Hegarty JE, O'Farrelly C, Doherty DG.
    Gut; 2008 Aug 15; 57(8):1121-8. PubMed ID: 18372499
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  • 15. NK cells of HIV-1-infected patients with poor CD4+ T-cell reconstitution despite suppressive HAART show reduced IFN-γ production and high frequency of autoreactive CD56bright cells.
    Giuliani E, Vassena L, Di Cesare S, Malagnino V, Desimio MG, Andreoni M, Barnaba V, Doria M.
    Immunol Lett; 2017 Oct 15; 190():185-193. PubMed ID: 28826739
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  • 18. CD3(+)CD56(+) Natural Killer-Like T Cells Display Anti-HCV Activity but Are Functionally Impaired in HIV(+) Patients With Acute Hepatitis C.
    Kokordelis P, Krämer B, Boesecke C, Voigt E, Ingiliz P, Glässner A, Wolter F, Srassburg CP, Spengler U, Rockstroh JK, Nattermann J.
    J Acquir Immune Defic Syndr; 2015 Dec 01; 70(4):338-46. PubMed ID: 26509931
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  • 19. Cytotoxicity of CD56(bright) NK cells towards autologous activated CD4+ T cells is mediated through NKG2D, LFA-1 and TRAIL and dampened via CD94/NKG2A.
    Nielsen N, Ødum N, Ursø B, Lanier LL, Spee P.
    PLoS One; 2012 Dec 01; 7(2):e31959. PubMed ID: 22384114
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  • 20. Terminal Differentiation of CD56(dim)CD16(+) Natural Killer Cells Is Associated with Increase in Natural Killer Cell Frequencies After Antiretroviral Treatment in HIV-1 Infection.
    Ahmad F, Tufa DM, Mishra N, Jacobs R, Schmidt RE.
    AIDS Res Hum Retroviruses; 2015 Dec 01; 31(12):1206-12. PubMed ID: 26352913
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