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2. The stepwise acquisition of fluconazole resistance mutations causes a gradual loss of fitness in Candida albicans. Sasse C, Dunkel N, Schäfer T, Schneider S, Dierolf F, Ohlsen K, Morschhäuser J. Mol Microbiol; 2012 Nov; 86(3):539-56. PubMed ID: 22924823 [Abstract] [Full Text] [Related]
3. The transcription factor Ndt80 does not contribute to Mrr1-, Tac1-, and Upc2-mediated fluconazole resistance in Candida albicans. Sasse C, Schillig R, Dierolf F, Weyler M, Schneider S, Mogavero S, Rogers PD, Morschhäuser J. PLoS One; 2011 Nov; 6(9):e25623. PubMed ID: 21980509 [Abstract] [Full Text] [Related]
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5. Induction of Candida albicans drug resistance genes by hybrid zinc cluster transcription factors. Schneider S, Morschhäuser J. Antimicrob Agents Chemother; 2015 Jan; 59(1):558-69. PubMed ID: 25385116 [Abstract] [Full Text] [Related]
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8. An A643T mutation in the transcription factor Upc2p causes constitutive ERG11 upregulation and increased fluconazole resistance in Candida albicans. Heilmann CJ, Schneider S, Barker KS, Rogers PD, Morschhäuser J. Antimicrob Agents Chemother; 2010 Jan; 54(1):353-9. PubMed ID: 19884367 [Abstract] [Full Text] [Related]
9. An acquired mechanism of antifungal drug resistance simultaneously enables Candida albicans to escape from intrinsic host defenses. Hampe IAI, Friedman J, Edgerton M, Morschhäuser J. PLoS Pathog; 2017 Sep; 13(9):e1006655. PubMed ID: 28953977 [Abstract] [Full Text] [Related]
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