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PUBMED FOR HANDHELDS

Journal Abstract Search


113 related items for PubMed ID: 28466810

  • 1. Oleanolic acid (OA) regulates inflammation and cellular dedifferentiation of chondrocytes via MAPK signaling pathways.
    Phull AR, Eo SH, Kim SJ.
    Cell Mol Biol (Noisy-le-grand); 2017 Mar 31; 63(3):12-17. PubMed ID: 28466810
    [Abstract] [Full Text] [Related]

  • 2. The thymoquinone-induced production of reactive oxygen species promotes dedifferentiation through the ERK pathway and inflammation through the p38 and PI3K pathways in rabbit articular chondrocytes.
    Yu SM, Kim SJ.
    Int J Mol Med; 2015 Feb 31; 35(2):325-32. PubMed ID: 25435376
    [Abstract] [Full Text] [Related]

  • 3. PEP-1-SIRT2 causes dedifferentiation and COX-2 expression via the MAPK pathways in rabbit articular chondrocytes.
    Eo SH, Kim DW, Choi SY, Kim HA, Kim SJ.
    Exp Cell Res; 2015 Dec 10; 339(2):351-9. PubMed ID: 26358233
    [Abstract] [Full Text] [Related]

  • 4. Production of reactive oxygen species by withaferin A causes loss of type collagen expression and COX-2 expression through the PI3K/Akt, p38, and JNK pathways in rabbit articular chondrocytes.
    Yu SM, Kim SJ.
    Exp Cell Res; 2013 Nov 01; 319(18):2822-34. PubMed ID: 24016823
    [Abstract] [Full Text] [Related]

  • 5. Simvastatin induces differentiation of rabbit articular chondrocytes via the ERK-1/2 and p38 kinase pathways.
    Han Y, Kim SJ.
    Exp Cell Res; 2016 Aug 15; 346(2):198-205. PubMed ID: 27475840
    [Abstract] [Full Text] [Related]

  • 6. Rosmarinic acid induces rabbit articular chondrocyte differentiation by decreases matrix metalloproteinase-13 and inflammation by upregulating cyclooxygenase-2 expression.
    Eo SH, Kim SJ.
    J Biomed Sci; 2017 Sep 18; 24(1):75. PubMed ID: 28923043
    [Abstract] [Full Text] [Related]

  • 7. Salinomycin causes dedifferentiation via the extracellular signal-regulated kinase (ERK) pathway in rabbit articular chondrocytes.
    Yu SM, Kim SJ.
    J Pharmacol Sci; 2015 Feb 18; 127(2):196-202. PubMed ID: 25727957
    [Abstract] [Full Text] [Related]

  • 8. Cytokine-induced apoptosis inhibitor-1 causes dedifferentiation of rabbit articular chondrocytes via the ERK-1/2 and p38 kinase pathways.
    Yu SM, Yeo HJ, Choi SY, Kim SJ.
    Int J Biochem Cell Biol; 2016 Nov 18; 80():10-18. PubMed ID: 27644154
    [Abstract] [Full Text] [Related]

  • 9. Inhibition of p38 pathway leads to OA-like changes in a rat animal model.
    Prasadam I, Mao X, Wang Y, Shi W, Crawford R, Xiao Y.
    Rheumatology (Oxford); 2012 May 18; 51(5):813-23. PubMed ID: 22240502
    [Abstract] [Full Text] [Related]

  • 10. Gallotannin causes differentiation and inflammation via ERK‑1/‑2 and p38 kinase pathways in rabbit articular chondrocytes.
    Lee WK, Chung KW, Kim GH, Kim SJ.
    Mol Med Rep; 2013 Feb 18; 7(2):701-7. PubMed ID: 23229854
    [Abstract] [Full Text] [Related]

  • 11. ERK-1/2 and p38 in the regulation of hypertrophic changes of normal articular cartilage chondrocytes induced by osteoarthritic subchondral osteoblasts.
    Prasadam I, van Gennip S, Friis T, Shi W, Crawford R, Xiao Y.
    Arthritis Rheum; 2010 May 18; 62(5):1349-60. PubMed ID: 20155832
    [Abstract] [Full Text] [Related]

  • 12. ERK-1/-2 and p38 kinase oppositely regulate 15-deoxy-delta(12,14)-prostaglandinJ(2)-Induced PPAR-gamma activation that mediates dedifferentiation but not cyclooxygenase-2 expression in articular chondrocytes.
    Yoon EK, Lee WK, Lee JH, Yu SM, Hwang SG, Kim SJ.
    J Korean Med Sci; 2007 Dec 18; 22(6):1015-21. PubMed ID: 18162716
    [Abstract] [Full Text] [Related]

  • 13. PEP-1-SIRT2-induced matrix metalloproteinase-1 and -13 modulates type II collagen expression via ERK signaling in rabbit articular chondrocytes.
    Eo SH, Choi SY, Kim SJ.
    Exp Cell Res; 2016 Nov 01; 348(2):201-208. PubMed ID: 27697532
    [Abstract] [Full Text] [Related]

  • 14. E3 Ubiquitin Ligase Constitutive Photomorphogenic 1 Regulates Differentiation and Inflammation via MAPK Signaling Pathway in Rabbit Articular Chondrocytes.
    Eom YS, Ko BS, Shah FH, Kim SJ.
    DNA Cell Biol; 2023 May 01; 42(5):239-247. PubMed ID: 36940307
    [Abstract] [Full Text] [Related]

  • 15. Sphingosine-1-phosphate attenuates proteoglycan aggrecan expression via production of prostaglandin E2 from human articular chondrocytes.
    Masuko K, Murata M, Nakamura H, Yudoh K, Nishioka K, Kato T.
    BMC Musculoskelet Disord; 2007 Mar 20; 8():29. PubMed ID: 17374154
    [Abstract] [Full Text] [Related]

  • 16. Sphingomyelinase decreases type II collagen expression in bovine articular cartilage chondrocytes via the ERK signaling pathway.
    Gilbert SJ, Blain EJ, Duance VC, Mason DJ.
    Arthritis Rheum; 2008 Jan 20; 58(1):209-20. PubMed ID: 18163502
    [Abstract] [Full Text] [Related]

  • 17. PEP-1-glutaredoxin-1 induces dedifferentiation of rabbit articular chondrocytes by the endoplasmic reticulum stress-dependent ERK-1/2 pathway and the endoplasmic reticulum stress-independent p38 kinase and PI-3 kinase pathways.
    Yu SM, Choi YJ, Kim SJ.
    Int J Biol Macromol; 2018 May 20; 111():1059-1066. PubMed ID: 29366902
    [Abstract] [Full Text] [Related]

  • 18. Low-Intensity Pulsed Ultrasound Affects Chondrocyte Extracellular Matrix Production via an Integrin-Mediated p38 MAPK Signaling Pathway.
    Xia P, Ren S, Lin Q, Cheng K, Shen S, Gao M, Li X.
    Ultrasound Med Biol; 2015 Jun 20; 41(6):1690-700. PubMed ID: 25736607
    [Abstract] [Full Text] [Related]

  • 19. Simvastatin-dependent actin cytoskeleton rearrangement regulates differentiation via the extracellular signal-regulated kinase-1/2 and p38 kinase pathways in rabbit articular chondrocytes.
    Han Y, Kim SJ.
    Eur J Pharmacol; 2018 Sep 05; 834():197-205. PubMed ID: 30009811
    [Abstract] [Full Text] [Related]

  • 20. Berberine induces dedifferentiation by actin cytoskeleton reorganization via phosphoinositide 3-kinase/Akt and p38 kinase pathways in rabbit articular chondrocytes.
    Yu SM, Cho H, Kim GH, Chung KW, Seo SY, Kim SJ.
    Exp Biol Med (Maywood); 2016 Apr 05; 241(8):800-7. PubMed ID: 26851252
    [Abstract] [Full Text] [Related]


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