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Journal Abstract Search

263 related items for PubMed ID: 28500788

  • 1. Unraveling the importance of the malaria parasite helicases.
    Tuteja R.
    FEBS J; 2017 Aug; 284(16):2592-2603. PubMed ID: 28500788
    [Abstract] [Full Text] [Related]

  • 2. Genome wide identification of Plasmodium falciparum helicases: a comparison with human host.
    Tuteja R.
    Cell Cycle; 2010 Jan 01; 9(1):104-20. PubMed ID: 20016272
    [Abstract] [Full Text] [Related]

  • 3. Plasmodium falciparum UvrD activities are downregulated by DNA-interacting compounds and its dsRNA inhibits malaria parasite growth.
    Tarique M, Tabassum F, Ahmad M, Tuteja R, Malaria Group.
    BMC Biochem; 2014 Apr 03; 15():9. PubMed ID: 24707807
    [Abstract] [Full Text] [Related]

  • 4. Unraveling the 'DEAD-box' helicases of Plasmodium falciparum.
    Tuteja R, Pradhan A.
    Gene; 2006 Jul 05; 376(1):1-12. PubMed ID: 16713133
    [Abstract] [Full Text] [Related]

  • 5. Helicases - feasible antimalarial drug target for Plasmodium falciparum.
    Tuteja R.
    FEBS J; 2007 Sep 05; 274(18):4699-704. PubMed ID: 17824956
    [Abstract] [Full Text] [Related]

  • 6. Emerging importance of mismatch repair components including UvrD helicase and their cross-talk with the development of drug resistance in malaria parasite.
    Ahmad M, Tuteja R.
    Mutat Res; 2014 Dec 05; 770():54-60. PubMed ID: 25771870
    [Abstract] [Full Text] [Related]

  • 7. Plasmodium falciparum Bloom homologue, a nucleocytoplasmic protein, translocates in 3' to 5' direction and is essential for parasite growth.
    Rahman F, Tarique M, Tuteja R.
    Biochim Biophys Acta; 2016 May 05; 1864(5):594-608. PubMed ID: 26917473
    [Abstract] [Full Text] [Related]

  • 8. ATPase activity of Plasmodium falciparum MLH is inhibited by DNA-interacting ligands and dsRNAs of MLH along with UvrD curtail malaria parasite growth.
    Tarique M, Chauhan M, Tuteja R.
    Protoplasma; 2017 May 05; 254(3):1295-1305. PubMed ID: 27624787
    [Abstract] [Full Text] [Related]

  • 9. UvrD helicase of Plasmodium falciparum.
    Shankar J, Tuteja R.
    Gene; 2008 Mar 15; 410(2):223-33. PubMed ID: 18242886
    [Abstract] [Full Text] [Related]

  • 10. A method to inhibit the growth of Plasmodium falciparum by double-stranded RNA-mediated gene silencing of helicases.
    Tuteja R.
    Methods Mol Biol; 2010 Mar 15; 587():389-99. PubMed ID: 20225164
    [Abstract] [Full Text] [Related]

  • 11. Characterization of Plasmodium falciparum ATP-dependent DNA helicase RuvB3.
    Limudomporn P, Moonsom S, Leartsakulpanich U, Suntornthiticharoen P, Petmitr S, Weinfeld M, Chavalitshewinkoon-Petmitr P.
    Malar J; 2016 Nov 03; 15(1):526. PubMed ID: 27809838
    [Abstract] [Full Text] [Related]

  • 12. 'DEAD-box' helicase from Plasmodium falciparum is active at wide pH and is schizont stage-specific.
    Pradhan A, Chauhan VS, Tuteja R.
    J Vector Borne Dis; 2007 Mar 03; 44(1):12-22. PubMed ID: 17378213
    [Abstract] [Full Text] [Related]

  • 13. Plasmodium falciparum DDX3X is a nucleocytoplasmic protein and requires N-terminal for DNA helicase activity.
    Sourabh S, Yasmin R, Tuteja R.
    Parasitol Int; 2021 Dec 03; 85():102420. PubMed ID: 34265466
    [Abstract] [Full Text] [Related]

  • 14. Plasmodium falciparum specific helicase 3 is nucleocytoplasmic protein and unwinds DNA duplex in 3' to 5' direction.
    Chauhan M, Tarique M, Tuteja R.
    Sci Rep; 2017 Oct 13; 7(1):13146. PubMed ID: 29030567
    [Abstract] [Full Text] [Related]

  • 15. Biochemical characterization of Plasmodium falciparum parasite specific helicase 1 (PfPSH1).
    Chauhan M, Sourabh S, Yasmin R, Pahuja I, Tuteja R.
    FEBS Open Bio; 2019 Nov 13; 9(11):1909-1927. PubMed ID: 31469232
    [Abstract] [Full Text] [Related]

  • 16. Plasmodium falciparum Werner homologue is a nuclear protein and its biochemical activities reside in the N-terminal region.
    Rahman F, Tarique M, Ahmad M, Tuteja R.
    Protoplasma; 2016 Jan 13; 253(1):45-60. PubMed ID: 25824666
    [Abstract] [Full Text] [Related]

  • 17. Functional genomic technologies applied to the control of the human malaria parasite, Plasmodium falciparum.
    Carucci DJ.
    Pharmacogenomics; 2001 May 13; 2(2):137-42. PubMed ID: 11368752
    [Abstract] [Full Text] [Related]

  • 18. Plasmodium falciparum RuvB2 translocates in 5'-3' direction, relocalizes during schizont stage and its enzymatic activities are up regulated by RuvB3 of the same complex.
    Ahmad M, Tuteja R.
    Biochim Biophys Acta; 2013 Dec 13; 1834(12):2795-811. PubMed ID: 24161468
    [Abstract] [Full Text] [Related]

  • 19. Emerging functions of helicases in regulation of stress survival in malaria parasite Plasmodium falciparum and their comparison with human host.
    Tuteja R.
    Parasitol Int; 2016 Dec 13; 65(6 Pt A):645-664. PubMed ID: 27586396
    [Abstract] [Full Text] [Related]

  • 20. RecQ helicases in the malaria parasite Plasmodium falciparum affect genome stability, gene expression patterns and DNA replication dynamics.
    Claessens A, Harris LM, Stanojcic S, Chappell L, Stanton A, Kuk N, Veneziano-Broccia P, Sterkers Y, Rayner JC, Merrick CJ.
    PLoS Genet; 2018 Jul 13; 14(7):e1007490. PubMed ID: 29965959
    [Abstract] [Full Text] [Related]

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