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Journal Abstract Search

94 related items for PubMed ID: 2851459

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  • 3. (Na+,K+)-ATPase and noradrenergic function: effects of chronic ethanol.
    Swann AC.
    Eur J Pharmacol; 1987 Feb 10; 134(2):145-53. PubMed ID: 3032651
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  • 8. Norepinephrine and (Na+, K+)-ATPase: evidence for stabilization by lithium or imipramine.
    Swann AC.
    Neuropharmacology; 1988 Mar 10; 27(3):261-7. PubMed ID: 2836750
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  • 9. Noradrenaline and thyroid function regulate (Na+,K+)-adenosine triphosphatase independently in vivo.
    Swann AC.
    Eur J Pharmacol; 1989 Oct 10; 169(2-3):275-83. PubMed ID: 2553449
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  • 12. Effects of adrenal steroids on the concentration of Na(+)-K+ pumps in rat skeletal muscle.
    Dørup I, Clausen T.
    J Endocrinol; 1997 Jan 10; 152(1):49-57. PubMed ID: 9014839
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  • 13. Brain (Na+, K+)-ATPase and noradrenergic activity: effects of hyperinnervation and denervation on high-affinity ouabain binding.
    Swann AC, Grant SJ, Maas JW.
    J Neurochem; 1982 Mar 10; 38(3):836-9. PubMed ID: 6173466
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  • 15. Modulation of renal sodium-potassium-adenosine triphosphatase by aldosterone. Effect of high physiologic levels on enzyme activity in isolated rat and rabbit tubules.
    Mujais SK, Chekal MA, Jones WJ, Hayslett JP, Katz AI.
    J Clin Invest; 1985 Jul 10; 76(1):170-6. PubMed ID: 2991336
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  • 16. Glucocorticoid regulation of mRNA encoding (Na+K) ATPase alpha 3 and beta 1 subunits in rat brain measured by in situ hybridization.
    Grillo C, Piroli G, González SL, Angulo J, McEwen BS, De Nicola AF.
    Brain Res; 1994 Sep 19; 657(1-2):83-91. PubMed ID: 7820647
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  • 17. Brain Na+,K+-ATPase: alteration of ligand affinities and conformation by chronic ethanol and noradrenergic stimulation in vivo.
    Swann AC.
    J Neurochem; 1986 Sep 19; 47(3):707-14. PubMed ID: 3016182
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  • 19. In vitro stimulation of Na-K-ATPase in rat thick ascending limb by dexamethasone.
    Doucet A, Hus-Citharel A, Morel F.
    Am J Physiol; 1986 Nov 19; 251(5 Pt 2):F851-7. PubMed ID: 3022604
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  • 20. Brain Na+,K(+)-ATPase regulation in vivo: reduction in activity and response to sodium by intracerebroventricular tetrodotoxin.
    Swann AC.
    Brain Res; 1991 Mar 15; 543(2):251-5. PubMed ID: 1647832
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