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275 related items for PubMed ID: 28550361
1. Prolyl-hydroxylase inhibition induces SDF-1 associated with increased CXCR4+/CD11b+ subpopulations and cardiac repair. Ghadge SK, Messner M, Van Pham T, Doppelhammer M, Petry A, Görlach A, Husse B, Franz WM, Zaruba MM. J Mol Med (Berl); 2017 Aug; 95(8):825-837. PubMed ID: 28550361 [Abstract] [Full Text] [Related]
2. Early upregulation of myocardial CXCR4 expression is critical for dimethyloxalylglycine-induced cardiac improvement in acute myocardial infarction. Mayorga M, Kiedrowski M, Shamhart P, Forudi F, Weber K, Chilian WM, Penn MS, Dong F. Am J Physiol Heart Circ Physiol; 2016 Jan 01; 310(1):H20-8. PubMed ID: 26519029 [Abstract] [Full Text] [Related]
3. Preconditioning of bone marrow mesenchymal stem cells by prolyl hydroxylase inhibition enhances cell survival and angiogenesis in vitro and after transplantation into the ischemic heart of rats. Liu XB, Wang JA, Ji XY, Yu SP, Wei L. Stem Cell Res Ther; 2014 Sep 25; 5(5):111. PubMed ID: 25257482 [Abstract] [Full Text] [Related]
4. Systemic preconditioning by a prolyl hydroxylase inhibitor promotes prevention of skin flap necrosis via HIF-1-induced bone marrow-derived cells. Takaku M, Tomita S, Kurobe H, Kihira Y, Morimoto A, Higashida M, Ikeda Y, Ushiyama A, Hashimoto I, Nakanishi H, Tamaki T. PLoS One; 2012 Sep 25; 7(8):e42964. PubMed ID: 22880134 [Abstract] [Full Text] [Related]
8. A critical role of Src family kinase in SDF-1/CXCR4-mediated bone-marrow progenitor cell recruitment to the ischemic heart. Cheng M, Huang K, Zhou J, Yan D, Tang YL, Zhao TC, Miller RJ, Kishore R, Losordo DW, Qin G. J Mol Cell Cardiol; 2015 Apr 01; 81():49-53. PubMed ID: 25655934 [Abstract] [Full Text] [Related]
9. DPP-4 inhibition has beneficial effects on the heart after myocardial infarction. Kubota A, Takano H, Wang H, Hasegawa H, Tadokoro H, Hirose M, Kobara Y, Yamada-Inagawa T, Komuro I, Kobayashi Y. J Mol Cell Cardiol; 2016 Feb 01; 91():72-80. PubMed ID: 26739213 [Abstract] [Full Text] [Related]
10. VEGF/SDF-1 promotes cardiac stem cell mobilization and myocardial repair in the infarcted heart. Tang JM, Wang JN, Zhang L, Zheng F, Yang JY, Kong X, Guo LY, Chen L, Huang YZ, Wan Y, Chen SY. Cardiovasc Res; 2011 Aug 01; 91(3):402-11. PubMed ID: 21345805 [Abstract] [Full Text] [Related]
11. Inhibition of Prolyl Hydroxylase Attenuates Fas Ligand-Induced Apoptosis and Lung Injury in Mice. Nagamine Y, Tojo K, Yazawa T, Takaki S, Baba Y, Goto T, Kurahashi K. Am J Respir Cell Mol Biol; 2016 Dec 01; 55(6):878-888. PubMed ID: 27494234 [Abstract] [Full Text] [Related]
12. HIF-1 activation attenuates postischemic myocardial injury: role for heme oxygenase-1 in modulating microvascular chemokine generation. Ockaili R, Natarajan R, Salloum F, Fisher BJ, Jones D, Fowler AA, Kukreja RC. Am J Physiol Heart Circ Physiol; 2005 Aug 01; 289(2):H542-8. PubMed ID: 15805230 [Abstract] [Full Text] [Related]
13. Inhibition of endogenous HIF inactivation induces angiogenesis in ischaemic skeletal muscles of mice. Milkiewicz M, Pugh CW, Egginton S. J Physiol; 2004 Oct 01; 560(Pt 1):21-6. PubMed ID: 15319416 [Abstract] [Full Text] [Related]
14. Danshensu accelerates angiogenesis after myocardial infarction in rats and promotes the functions of endothelial progenitor cells through SDF-1α/CXCR4 axis. Yin Y, Duan J, Guo C, Wei G, Wang Y, Guan Y, Mu F, Yao M, Xi M, Wen A. Eur J Pharmacol; 2017 Nov 05; 814():274-282. PubMed ID: 28864209 [Abstract] [Full Text] [Related]
15. Dimethyloxalylglycine prevents bone loss in ovariectomized C57BL/6J mice through enhanced angiogenesis and osteogenesis. Peng J, Lai ZG, Fang ZL, Xing S, Hui K, Hao C, Jin Q, Qi Z, Shen WJ, Dong QN, Bing ZH, Fu DL. PLoS One; 2014 Nov 05; 9(11):e112744. PubMed ID: 25394221 [Abstract] [Full Text] [Related]
16. Hydroxysafflor yellow A promotes neovascularization and cardiac function recovery through HO-1/VEGF-A/SDF-1α cascade. Wei G, Yin Y, Duan J, Guo C, Zhu Y, Wang Y, Xi M, Wen A. Biomed Pharmacother; 2017 Apr 05; 88():409-420. PubMed ID: 28122306 [Abstract] [Full Text] [Related]
17. Heme oxygenase-1 and carbon monoxide promote neovascularization after myocardial infarction by modulating the expression of HIF-1alpha, SDF-1alpha and VEGF-B. Lakkisto P, Kytö V, Forsten H, Siren JM, Segersvärd H, Voipio-Pulkki LM, Laine M, Pulkki K, Tikkanen I. Eur J Pharmacol; 2010 Jun 10; 635(1-3):156-64. PubMed ID: 20303947 [Abstract] [Full Text] [Related]
18. SDF-1α as a therapeutic stem cell homing factor in myocardial infarction. Ghadge SK, Mühlstedt S, Ozcelik C, Bader M. Pharmacol Ther; 2011 Jan 10; 129(1):97-108. PubMed ID: 20965212 [Abstract] [Full Text] [Related]
19. Prolyl-hydroxylase inhibition preserves endothelial cell function in a rat model of vascular ischemia reperfusion injury. Barnucz E, Veres G, Hegedűs P, Klein S, Zöller R, Radovits T, Korkmaz S, Horkay F, Merkely B, Karck M, Szabó G. J Pharmacol Exp Ther; 2013 Apr 10; 345(1):25-31. PubMed ID: 23388095 [Abstract] [Full Text] [Related]
20. The prolyl hydroxylase inhibitor dimethyloxalylglycine enhances dentin sialophoshoprotein expression through VEGF-induced Runx2 stabilization. Rahman SU, Lee MS, Baek JH, Ryoo HM, Woo KM. PLoS One; 2014 Apr 10; 9(11):e112078. PubMed ID: 25369078 [Abstract] [Full Text] [Related] Page: [Next] [New Search]