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Journal Abstract Search

290 related items for PubMed ID: 28618997

  • 1. VDAC1 as a Player in Mitochondria-Mediated Apoptosis and Target for Modulating Apoptosis.
    Shoshan-Barmatz V, Krelin Y, Chen Q.
    Curr Med Chem; 2017; 24(40):4435-4446. PubMed ID: 28618997
    [Abstract] [Full Text] [Related]

  • 2. VDAC1-interacting anion transport inhibitors inhibit VDAC1 oligomerization and apoptosis.
    Ben-Hail D, Shoshan-Barmatz V.
    Biochim Biophys Acta; 2016 Jul; 1863(7 Pt A):1612-23. PubMed ID: 27064145
    [Abstract] [Full Text] [Related]

  • 3. The mitochondrial voltage-dependent anion channel 1 in tumor cells.
    Shoshan-Barmatz V, Ben-Hail D, Admoni L, Krelin Y, Tripathi SS.
    Biochim Biophys Acta; 2015 Oct; 1848(10 Pt B):2547-75. PubMed ID: 25448878
    [Abstract] [Full Text] [Related]

  • 4. Ca(2+)-mediated regulation of VDAC1 expression levels is associated with cell death induction.
    Weisthal S, Keinan N, Ben-Hail D, Arif T, Shoshan-Barmatz V.
    Biochim Biophys Acta; 2014 Oct; 1843(10):2270-81. PubMed ID: 24704533
    [Abstract] [Full Text] [Related]

  • 5. The role of calcium in VDAC1 oligomerization and mitochondria-mediated apoptosis.
    Keinan N, Pahima H, Ben-Hail D, Shoshan-Barmatz V.
    Biochim Biophys Acta; 2013 Jul; 1833(7):1745-54. PubMed ID: 23542128
    [Abstract] [Full Text] [Related]

  • 6. Oligomerization of the mitochondrial protein VDAC1: from structure to function and cancer therapy.
    Shoshan-Barmatz V, Mizrachi D, Keinan N.
    Prog Mol Biol Transl Sci; 2013 Jul; 117():303-34. PubMed ID: 23663973
    [Abstract] [Full Text] [Related]

  • 7. VDAC1 functions in Ca2+ homeostasis and cell life and death in health and disease.
    Shoshan-Barmatz V, Krelin Y, Shteinfer-Kuzmine A.
    Cell Calcium; 2018 Jan; 69():81-100. PubMed ID: 28712506
    [Abstract] [Full Text] [Related]

  • 8. Novel Compounds Targeting the Mitochondrial Protein VDAC1 Inhibit Apoptosis and Protect against Mitochondrial Dysfunction.
    Ben-Hail D, Begas-Shvartz R, Shalev M, Shteinfer-Kuzmine A, Gruzman A, Reina S, De Pinto V, Shoshan-Barmatz V.
    J Biol Chem; 2016 Nov 25; 291(48):24986-25003. PubMed ID: 27738100
    [Abstract] [Full Text] [Related]

  • 9. Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo.
    Shoshan-Barmatz V, Keinan N, Abu-Hamad S, Tyomkin D, Aram L.
    Biochim Biophys Acta; 2010 Nov 25; 1797(6-7):1281-91. PubMed ID: 20214874
    [Abstract] [Full Text] [Related]

  • 10. The Voltage-dependent Anion Channel 1 Mediates Amyloid β Toxicity and Represents a Potential Target for Alzheimer Disease Therapy.
    Smilansky A, Dangoor L, Nakdimon I, Ben-Hail D, Mizrachi D, Shoshan-Barmatz V.
    J Biol Chem; 2015 Dec 25; 290(52):30670-83. PubMed ID: 26542804
    [Abstract] [Full Text] [Related]

  • 11. Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis.
    Mader A, Abu-Hamad S, Arbel N, Gutiérrez-Aguilar M, Shoshan-Barmatz V.
    Biochem J; 2010 Jul 01; 429(1):147-55. PubMed ID: 20420578
    [Abstract] [Full Text] [Related]

  • 12. Mitochondrial VDAC, the Na+/Ca2+ Exchanger, and the Ca2+ Uniporter in Ca2+ Dynamics and Signaling.
    Shoshan-Barmatz V, De S.
    Adv Exp Med Biol; 2017 Jul 01; 981():323-347. PubMed ID: 29594867
    [Abstract] [Full Text] [Related]

  • 13. The Mitochondrial Voltage-Dependent Anion Channel 1, Ca2+ Transport, Apoptosis, and Their Regulation.
    Shoshan-Barmatz V, De S, Meir A.
    Front Oncol; 2017 Jul 01; 7():60. PubMed ID: 28443244
    [Abstract] [Full Text] [Related]

  • 14. VDAC1 at the Intersection of Cell Metabolism, Apoptosis, and Diseases.
    Shoshan-Barmatz V, Shteinfer-Kuzmine A, Verma A.
    Biomolecules; 2020 Oct 26; 10(11):. PubMed ID: 33114780
    [Abstract] [Full Text] [Related]

  • 15. Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics.
    Shoshan-Barmatz V, Krelin Y, Shteinfer-Kuzmine A, Arif T.
    Front Oncol; 2017 Oct 26; 7():154. PubMed ID: 28824871
    [Abstract] [Full Text] [Related]

  • 16. VDAC1-based peptides: novel pro-apoptotic agents and potential therapeutics for B-cell chronic lymphocytic leukemia.
    Prezma T, Shteinfer A, Admoni L, Raviv Z, Sela I, Levi I, Shoshan-Barmatz V.
    Cell Death Dis; 2013 Sep 19; 4(9):e809. PubMed ID: 24052077
    [Abstract] [Full Text] [Related]

  • 17. Quinocetone induces mitochondrial apoptosis in HepG2 cells through ROS-dependent promotion of VDAC1 oligomerization and suppression of Wnt1/β-catenin signaling pathway.
    Yang X, Tang S, Dai C, Li D, Zhang S, Deng S, Zhou Y, Xiao X.
    Food Chem Toxicol; 2017 Jul 19; 105():161-176. PubMed ID: 28343033
    [Abstract] [Full Text] [Related]

  • 18. The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins.
    Abu-Hamad S, Arbel N, Calo D, Arzoine L, Israelson A, Keinan N, Ben-Romano R, Friedman O, Shoshan-Barmatz V.
    J Cell Sci; 2009 Jun 01; 122(Pt 11):1906-16. PubMed ID: 19461077
    [Abstract] [Full Text] [Related]

  • 19. VDAC1, mitochondrial dysfunction, and Alzheimer's disease.
    Shoshan-Barmatz V, Nahon-Crystal E, Shteinfer-Kuzmine A, Gupta R.
    Pharmacol Res; 2018 May 01; 131():87-101. PubMed ID: 29551631
    [Abstract] [Full Text] [Related]

  • 20. VDAC1 cysteine residues: topology and function in channel activity and apoptosis.
    Aram L, Geula S, Arbel N, Shoshan-Barmatz V.
    Biochem J; 2010 Apr 14; 427(3):445-54. PubMed ID: 20192921
    [Abstract] [Full Text] [Related]

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