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479 related items for PubMed ID: 28725637

  • 1. Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.
    Roberts JS, Atanasova KR, Lee J, Diamond G, Deguzman J, Hee Choi C, Yilmaz Ö.
    Front Cell Infect Microbiol; 2017; 7():291. PubMed ID: 28725637
    [Abstract] [Full Text] [Related]

  • 2. Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.
    Choi CH, Spooner R, DeGuzman J, Koutouzis T, Ojcius DM, Yilmaz Ö.
    Cell Microbiol; 2013 Jun; 15(6):961-76. PubMed ID: 23241000
    [Abstract] [Full Text] [Related]

  • 3. A novel kinase function of a nucleoside-diphosphate-kinase homologue in Porphyromonas gingivalis is critical in subversion of host cell apoptosis by targeting heat-shock protein 27.
    Lee J, Roberts JS, Atanasova KR, Chowdhury N, Yilmaz Ö.
    Cell Microbiol; 2018 May; 20(5):e12825. PubMed ID: 29359393
    [Abstract] [Full Text] [Related]

  • 4. Host surface ectonucleotidase-CD73 and the opportunistic pathogen, Porphyromonas gingivalis, cross-modulation underlies a new homeostatic mechanism for chronic bacterial survival in human epithelial cells.
    Lee JS, Chowdhury N, Roberts JS, Yilmaz Ö.
    Virulence; 2020 Dec; 11(1):414-429. PubMed ID: 32419582
    [Abstract] [Full Text] [Related]

  • 5. Nucleoside-Diphosphate-Kinase of P. gingivalis is Secreted from Epithelial Cells In the Absence of a Leader Sequence Through a Pannexin-1 Interactome.
    Atanasova K, Lee J, Roberts J, Lee K, Ojcius DM, Yilmaz Ö.
    Sci Rep; 2016 Nov 24; 6():37643. PubMed ID: 27883084
    [Abstract] [Full Text] [Related]

  • 6. NOX1/2 activation in human gingival fibroblasts by Fusobacterium nucleatum facilitates attachment of Porphyromonas gingivalis.
    Ahn SH, Song JE, Kim S, Cho SH, Lim YK, Kook JK, Kook MS, Lee TH.
    Arch Microbiol; 2016 Aug 24; 198(6):573-83. PubMed ID: 27071620
    [Abstract] [Full Text] [Related]

  • 7. Porphyromonas gingivalis traffics into endoplasmic reticulum-rich-autophagosomes for successful survival in human gingival epithelial cells.
    Lee K, Roberts JS, Choi CH, Atanasova KR, Yilmaz Ö.
    Virulence; 2018 Dec 31; 9(1):845-859. PubMed ID: 29616874
    [Abstract] [Full Text] [Related]

  • 8. Porphyromonas gingivalis infection sequesters pro-apoptotic Bad through Akt in primary gingival epithelial cells.
    Yao L, Jermanus C, Barbetta B, Choi C, Verbeke P, Ojcius DM, Yilmaz O.
    Mol Oral Microbiol; 2010 Apr 31; 25(2):89-101. PubMed ID: 20331797
    [Abstract] [Full Text] [Related]

  • 9. P2X7 receptor-mediated leukocyte recruitment and Porphyromonas gingivalis clearance requires IL-1β production and autocrine IL-1 receptor activation.
    Almeida-da-Silva CLC, Ramos-Junior ES, Morandini AC, Rocha GDC, Marinho Y, Tamura AS, de Andrade KQ, Bellio M, Savio LEB, Scharfstein J, Ojcius DM, Coutinho-Silva R.
    Immunobiology; 2019 Jan 31; 224(1):50-59. PubMed ID: 30429052
    [Abstract] [Full Text] [Related]

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  • 11. ATP-dependent activation of an inflammasome in primary gingival epithelial cells infected by Porphyromonas gingivalis.
    Yilmaz O, Sater AA, Yao L, Koutouzis T, Pettengill M, Ojcius DM.
    Cell Microbiol; 2010 Feb 31; 12(2):188-98. PubMed ID: 19811501
    [Abstract] [Full Text] [Related]

  • 12. Porphyromonas gingivalis attenuates ATP-mediated inflammasome activation and HMGB1 release through expression of a nucleoside-diphosphate kinase.
    Johnson L, Atanasova KR, Bui PQ, Lee J, Hung SC, Yilmaz Ö, Ojcius DM.
    Microbes Infect; 2015 May 31; 17(5):369-77. PubMed ID: 25828169
    [Abstract] [Full Text] [Related]

  • 13. Intercellular spreading of Porphyromonas gingivalis infection in primary gingival epithelial cells.
    Yilmaz O, Verbeke P, Lamont RJ, Ojcius DM.
    Infect Immun; 2006 Jan 31; 74(1):703-10. PubMed ID: 16369027
    [Abstract] [Full Text] [Related]

  • 14. Expression of human caspase-4 in the gingival epithelium affected with periodontitis: Its involvement in Porphyromonas gingivalis-challenged gingival epithelial cells.
    Kantrong N, Buranaphatthana W, Hormdee D, Suwannarong W, Chaichit R, Pattanaporn K, Klanrit P, Krisanaprakornkit S.
    Arch Oral Biol; 2022 Aug 31; 140():105466. PubMed ID: 35640321
    [Abstract] [Full Text] [Related]

  • 15. The role of reactive-oxygen-species in microbial persistence and inflammation.
    Spooner R, Yilmaz O.
    Int J Mol Sci; 2011 Jan 13; 12(1):334-52. PubMed ID: 21339989
    [Abstract] [Full Text] [Related]

  • 16. Porphyromonas gingivalis fimbriae dampen P2X7-dependent interleukin-1β secretion.
    Morandini AC, Ramos-Junior ES, Potempa J, Nguyen KA, Oliveira AC, Bellio M, Ojcius DM, Scharfstein J, Coutinho-Silva R.
    J Innate Immun; 2014 Jan 13; 6(6):831-45. PubMed ID: 24925032
    [Abstract] [Full Text] [Related]

  • 17. Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalis.
    Yilmaz O, Jungas T, Verbeke P, Ojcius DM.
    Infect Immun; 2004 Jul 13; 72(7):3743-51. PubMed ID: 15213114
    [Abstract] [Full Text] [Related]

  • 18. Noncanonical activation of β-catenin by Porphyromonas gingivalis.
    Zhou Y, Sztukowska M, Wang Q, Inaba H, Potempa J, Scott DA, Wang H, Lamont RJ.
    Infect Immun; 2015 Aug 13; 83(8):3195-203. PubMed ID: 26034209
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