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Journal Abstract Search

138 related items for PubMed ID: 2876960

  • 1. Inability of pyrogenic, purified Bordetella pertussis lipid A to induce interleukin-1 release by human monocytes.
    Caroff M, Cavaillon JM, Fitting C, Haeffner-Cavaillon N.
    Infect Immun; 1986 Nov; 54(2):465-71. PubMed ID: 2876960
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  • 2. The role of serum in interleukin 1 production by human monocytes activated by endotoxins and their polysaccharide moieties.
    Cavaillon JM, Haeffner-Cavaillon N.
    Immunol Lett; 1985 Nov; 10(1):35-41. PubMed ID: 2861158
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  • 3. Inhibition by gangliosides of the specific binding of lipopolysaccharide (LPS) to human monocytes prevents LPS-induced interleukin-1 production.
    Cavaillon JM, Fitting C, Hauttecoeur B, Haeffner-Cavaillon N.
    Cell Immunol; 1987 May; 106(2):293-303. PubMed ID: 2436818
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  • 6. Dissociation of cell-associated interleukin-1 (IL-1) and IL-1 release induced by lipopolysaccharide and lipid A.
    Cavaillon JM, Fitting C, Caroff M, Haeffner-Cavaillon N.
    Infect Immun; 1989 Mar; 57(3):791-7. PubMed ID: 2537258
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  • 10. LPS from Neisseria meningitidis is crucial for inducing monocyte- and microparticle-associated tissue factor activity but not for tissue factor expression.
    Øvstebø R, Aass HC, Haug KB, Trøseid AM, Gopinathan U, Kierulf P, Berg JP, Brandtzaeg P, Henriksson CE.
    Innate Immun; 2012 Aug; 18(4):580-91. PubMed ID: 22180561
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  • 12. Variability in LPS composition, antigenicity and reactogenicity of phase variants of Bordetella pertussis.
    Ray A, Redhead K, Selkirk S, Poole S.
    FEMS Microbiol Lett; 1991 Apr 15; 63(2-3):211-7. PubMed ID: 2060761
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  • 14. Lipopolysaccharides from Bordetella pertussis and Bordetella parapertussis differently modulate human dendritic cell functions resulting in divergent prevalence of Th17-polarized responses.
    Fedele G, Nasso M, Spensieri F, Palazzo R, Frasca L, Watanabe M, Ausiello CM.
    J Immunol; 2008 Jul 01; 181(1):208-16. PubMed ID: 18566386
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  • 15. Substitution of the Bordetella pertussis lipid A phosphate groups with glucosamine is required for robust NF-kappaB activation and release of proinflammatory cytokines in cells expressing human but not murine Toll-like receptor 4-MD-2-CD14.
    Marr N, Hajjar AM, Shah NR, Novikov A, Yam CS, Caroff M, Fernandez RC.
    Infect Immun; 2010 May 01; 78(5):2060-9. PubMed ID: 20176798
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  • 16. Suppressive effect of lipid A partial structures on lipopolysaccharide or lipid A-induced release of interleukin 1 by human monocytes.
    Wang MH, Feist W, Herzbeck H, Brade H, Kusumoto S, Rietschel ET, Flad HD, Ulmer AJ.
    FEMS Microbiol Immunol; 1990 Oct 01; 2(3):179-85. PubMed ID: 2257173
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  • 17. Gram-negative bacteria induce proinflammatory cytokine production by monocytes in the absence of lipopolysaccharide (LPS).
    Uronen H, Williams AJ, Dixon G, Andersen SR, Van Der Ley P, Van Deuren M, Callard RE, Klein N.
    Clin Exp Immunol; 2000 Dec 01; 122(3):312-5. PubMed ID: 11122234
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  • 18. Characterization of the pyrogenicity of two different lipopolysaccharides and their lipid A-bovine serum albumin complexes.
    Ogawa Y, Murai T, Kanoh S.
    J Pharmacobiodyn; 1986 Sep 01; 9(9):722-8. PubMed ID: 3540273
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  • 19. Induction by lipopolysaccharide of intracellular and extracellular interleukin 1 production: analysis with synthetic models.
    Lasfargues A, Ledur A, Charon D, Szabo L, Chaby R.
    J Immunol; 1987 Jul 15; 139(2):429-36. PubMed ID: 3496381
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  • 20. Modulation of endotoxin-induced monokine release in human monocytes by lipid A partial structures that inhibit binding of 125I-lipopolysaccharide.
    Ulmer AJ, Feist W, Heine H, Kirikae T, Kirikae F, Kusumoto S, Kusama T, Brade H, Schade U, Rietschel ET.
    Infect Immun; 1992 Dec 15; 60(12):5145-52. PubMed ID: 1280625
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