These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
244 related items for PubMed ID: 28823647
1. Sprifermin (rhFGF18) enables proliferation of chondrocytes producing a hyaline cartilage matrix. Gigout A, Guehring H, Froemel D, Meurer A, Ladel C, Reker D, Bay-Jensen AC, Karsdal MA, Lindemann S. Osteoarthritis Cartilage; 2017 Nov; 25(11):1858-1867. PubMed ID: 28823647 [Abstract] [Full Text] [Related]
2. Sprifermin (rhFGF18) modulates extracellular matrix turnover in cartilage explants ex vivo. Reker D, Kjelgaard-Petersen CF, Siebuhr AS, Michaelis M, Gigout A, Karsdal MA, Ladel C, Bay-Jensen AC. J Transl Med; 2017 Dec 12; 15(1):250. PubMed ID: 29233174 [Abstract] [Full Text] [Related]
3. Effects of Sprifermin, IGF1, IGF2, BMP7, or CNP on Bovine Chondrocytes in Monolayer and 3D Culture. Müller S, Lindemann S, Gigout A. J Orthop Res; 2020 Mar 12; 38(3):653-662. PubMed ID: 31608492 [Abstract] [Full Text] [Related]
4. Sprifermin (rhFGF18) versus vehicle induces a biphasic process of extracellular matrix remodeling in human knee OA articular cartilage ex vivo. Reker D, Siebuhr AS, Thudium CS, Gantzel T, Ladel C, Michaelis M, Aspberg A, Berchtold MW, Karsdal MA, Gigout A, Bay-Jensen AC. Sci Rep; 2020 Apr 07; 10(1):6011. PubMed ID: 32265494 [Abstract] [Full Text] [Related]
5. Sprifermin (recombinant human FGF18) is internalized through clathrin- and dynamin-independent pathways and degraded in primary chondrocytes. Sieber S, Gigout A. Exp Cell Res; 2020 Oct 15; 395(2):112236. PubMed ID: 32798495 [Abstract] [Full Text] [Related]
9. Extracellular matrix derived from chondrocytes promotes rapid expansion of human primary chondrocytes in vitro with reduced dedifferentiation. Mao Y, Block T, Singh-Varma A, Sheldrake A, Leeth R, Griffey S, Kohn J. Acta Biomater; 2019 Feb 15; 85():75-83. PubMed ID: 30528605 [Abstract] [Full Text] [Related]