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309 related items for PubMed ID: 28825114
1. Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 ameliorate neuroinflammatory responses in chronic cerebral hypoperfusion model by blocking NF-κB pathways. Su SH, Wu YF, Lin Q, Hai J. Naunyn Schmiedebergs Arch Pharmacol; 2017 Dec; 390(12):1189-1200. PubMed ID: 28825114 [Abstract] [Full Text] [Related]
2. Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 suppress chronic cerebral hypoperfusion-induced neuronal apoptosis by inhibiting c-Jun N-terminal kinase signaling. Su SH, Wu YF, Lin Q, Yu F, Hai J. Neuroscience; 2015 Aug 20; 301():563-75. PubMed ID: 25795598 [Abstract] [Full Text] [Related]
3. Cannabinoid receptor agonist WIN55,212-2 and fatty acid amide hydrolase inhibitor URB597 may protect against cognitive impairment in rats of chronic cerebral hypoperfusion via PI3K/AKT signaling. Su SH, Wang YQ, Wu YF, Wang DP, Lin Q, Hai J. Behav Brain Res; 2016 Oct 15; 313():334-344. PubMed ID: 27424778 [Abstract] [Full Text] [Related]
4. URB597 protects against NLRP3 inflammasome activation by inhibiting autophagy dysfunction in a rat model of chronic cerebral hypoperfusion. Su SH, Wu YF, Lin Q, Wang DP, Hai J. J Neuroinflammation; 2019 Dec 09; 16(1):260. PubMed ID: 31815636 [Abstract] [Full Text] [Related]
5. The Cannabinoid Receptor Agonist WIN55,212-2 Ameliorates Hippocampal Neuronal Damage After Chronic Cerebral Hypoperfusion Possibly Through Inhibiting Oxidative Stress and ASK1-p38 Signaling. Wang DP, Lv QL, Lin Q, Kang K, Jin KY, Hai J. Neurotox Res; 2020 Apr 09; 37(4):847-856. PubMed ID: 31808139 [Abstract] [Full Text] [Related]
6. Increased Contextual Fear Conditioning in iNOS Knockout Mice: Additional Evidence for the Involvement of Nitric Oxide in Stress-Related Disorders and Contribution of the Endocannabinoid System. Lisboa SF, Gomes FV, Silva AL, Uliana DL, Camargo LH, Guimarães FS, Cunha FQ, Joca SR, Resstel LB. Int J Neuropsychopharmacol; 2015 Jan 24; 18(8):. PubMed ID: 25618404 [Abstract] [Full Text] [Related]
7. Effects of URB597 as an inhibitor of fatty acid amide hydrolase on WIN55, 212-2-induced learning and memory deficits in rats. Hasanein P, Teimuri Far M. Pharmacol Biochem Behav; 2015 Apr 24; 131():130-5. PubMed ID: 25689415 [Abstract] [Full Text] [Related]
8. Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro. Slivicki RA, Xu Z, Mali SS, Hohmann AG. Pharmacol Res; 2019 Apr 24; 142():267-282. PubMed ID: 30739035 [Abstract] [Full Text] [Related]
9. WIN55212-2 attenuates amyloid-beta-induced neuroinflammation in rats through activation of cannabinoid receptors and PPAR-γ pathway. Fakhfouri G, Ahmadiani A, Rahimian R, Grolla AA, Moradi F, Haeri A. Neuropharmacology; 2012 Sep 24; 63(4):653-66. PubMed ID: 22634229 [Abstract] [Full Text] [Related]
10. The potential protective effects of cannabinoid receptor agonist WIN55,212-2 on cognitive dysfunction is associated with the suppression of autophagy and inflammation in an experimental model of vascular dementia. Wang DP, Yin H, Kang K, Lin Q, Su SH, Hai J. Psychiatry Res; 2018 Sep 24; 267():281-288. PubMed ID: 29945070 [Abstract] [Full Text] [Related]
11. Chronic treatment with URB597 ameliorates post-stress symptoms in a rat model of PTSD. Fidelman S, Mizrachi Zer-Aviv T, Lange R, Hillard CJ, Akirav I. Eur Neuropsychopharmacol; 2018 May 24; 28(5):630-642. PubMed ID: 29519609 [Abstract] [Full Text] [Related]
12. WIN 55,212-2, agonist of cannabinoid receptors, prevents amyloid β1-42 effects on astrocytes in primary culture. Aguirre-Rueda D, Guerra-Ojeda S, Aldasoro M, Iradi A, Obrador E, Mauricio MD, Vila JM, Marchio P, Valles SL. PLoS One; 2015 May 24; 10(4):e0122843. PubMed ID: 25874692 [Abstract] [Full Text] [Related]
13. Inhibiting a spinal cord signaling pathway protects against ischemia injury in rats. Huo J, Ma R, Chai X, Liang HJ, Jiang P, Zhu XL, Chen X, Su BX. J Thorac Cardiovasc Surg; 2019 Feb 24; 157(2):494-503.e1. PubMed ID: 30195603 [Abstract] [Full Text] [Related]
15. Quercetin disrupts tyrosine-phosphorylated phosphatidylinositol 3-kinase and myeloid differentiation factor-88 association, and inhibits MAPK/AP-1 and IKK/NF-κB-induced inflammatory mediators production in RAW 264.7 cells. Endale M, Park SC, Kim S, Kim SH, Yang Y, Cho JY, Rhee MH. Immunobiology; 2013 Dec 24; 218(12):1452-67. PubMed ID: 23735482 [Abstract] [Full Text] [Related]
16. Pharmacological blockade of fatty acid amide hydrolase (FAAH) by URB597 improves memory and changes the phenotype of hippocampal microglia despite ethanol exposure. Rivera P, Fernández-Arjona MDM, Silva-Peña D, Blanco E, Vargas A, López-Ávalos MD, Grondona JM, Serrano A, Pavón FJ, Rodríguez de Fonseca F, Suárez J. Biochem Pharmacol; 2018 Nov 24; 157():244-257. PubMed ID: 30098312 [Abstract] [Full Text] [Related]
17. Elevation of endocannabinoid levels in the ventrolateral periaqueductal grey through inhibition of fatty acid amide hydrolase affects descending nociceptive pathways via both cannabinoid receptor type 1 and transient receptor potential vanilloid type-1 receptors. Maione S, Bisogno T, de Novellis V, Palazzo E, Cristino L, Valenti M, Petrosino S, Guglielmotti V, Rossi F, Di Marzo V. J Pharmacol Exp Ther; 2006 Mar 24; 316(3):969-82. PubMed ID: 16284279 [Abstract] [Full Text] [Related]
18. URB597 improves cognitive impairment induced by chronic cerebral hypoperfusion by inhibiting mTOR-dependent autophagy. Wang D, Lin Q, Su S, Liu K, Wu Y, Hai J. Neuroscience; 2017 Mar 06; 344():293-304. PubMed ID: 28042028 [Abstract] [Full Text] [Related]
19. Cannabinoids prevent depressive-like symptoms and alterations in BDNF expression in a rat model of PTSD. Burstein O, Shoshan N, Doron R, Akirav I. Prog Neuropsychopharmacol Biol Psychiatry; 2018 Jun 08; 84(Pt A):129-139. PubMed ID: 29458190 [Abstract] [Full Text] [Related]
20. Tetrachlorobenzoquinone exhibits neurotoxicity by inducing inflammatory responses through ROS-mediated IKK/IκB/NF-κB signaling. Fu J, Shi Q, Song X, Xia X, Su C, Liu Z, Song E, Song Y. Environ Toxicol Pharmacol; 2016 Jan 08; 41():241-50. PubMed ID: 26745386 [Abstract] [Full Text] [Related] Page: [Next] [New Search]