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372 related items for PubMed ID: 28844763

  • 1. Hyperexcitability in synaptic and firing activities of spinal motoneurons in an adult mouse model of amyotrophic lateral sclerosis.
    Jiang MC, Adimula A, Birch D, Heckman CJ.
    Neuroscience; 2017 Oct 24; 362():33-46. PubMed ID: 28844763
    [Abstract] [Full Text] [Related]

  • 2. Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral sclerosis.
    Jiang M, Schuster JE, Fu R, Siddique T, Heckman CJ.
    J Neurosci; 2009 Dec 02; 29(48):15031-8. PubMed ID: 19955354
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  • 3. Excitatory action of low frequency depolarizing GABA/glycine synaptic inputs is prevalent in prenatal spinal SOD1G93A motoneurons.
    Zhu H, Dalvi U, Cazenave W, Cattaert D, Branchereau P.
    J Physiol; 2024 Mar 02; 602(5):913-932. PubMed ID: 38345477
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  • 4. Acute neurotoxicant exposure induces hyperexcitability in mouse lumbar spinal motor neurons.
    Sceniak MP, Spitsbergen JB, Sabo SL, Yuan Y, Atchison WD.
    J Neurophysiol; 2020 Apr 01; 123(4):1448-1459. PubMed ID: 32159428
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  • 5. Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice.
    Avossa D, Grandolfo M, Mazzarol F, Zatta M, Ballerini L.
    Neuroscience; 2006 Apr 01; 138(4):1179-94. PubMed ID: 16442737
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  • 6. Altered development in GABA co-release shapes glycinergic synaptic currents in cultured spinal slices of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Medelin M, Rancic V, Cellot G, Laishram J, Veeraraghavan P, Rossi C, Muzio L, Sivilotti L, Ballerini L.
    J Physiol; 2016 Jul 01; 594(13):3827-40. PubMed ID: 27098371
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  • 7. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
    Kuo JJ, Schonewille M, Siddique T, Schults AN, Fu R, Bär PR, Anelli R, Heckman CJ, Kroese AB.
    J Neurophysiol; 2004 Jan 01; 91(1):571-5. PubMed ID: 14523070
    [Abstract] [Full Text] [Related]

  • 8. Persistent sodium currents participate in fictive locomotion generation in neonatal mouse spinal cord.
    Zhong G, Masino MA, Harris-Warrick RM.
    J Neurosci; 2007 Apr 25; 27(17):4507-18. PubMed ID: 17460064
    [Abstract] [Full Text] [Related]

  • 9. An ex vivo preparation of mature mice spinal cord to study synaptic transmission on motoneurons.
    Moghaddasi M, Velumian AA, Zhang L, Fehlings MG.
    J Neurosci Methods; 2007 Jan 15; 159(1):1-7. PubMed ID: 16887193
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  • 10. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
    Amendola J, Gueritaud JP, Lamotte d'Incamps B, Bories C, Liabeuf S, Allene C, Pambo-Pambo A, Durand J.
    Arch Ital Biol; 2007 Nov 15; 145(3-4):311-23. PubMed ID: 18075124
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  • 12. Acute modulation of synaptic transmission to motoneurons by BDNF in the neonatal rat spinal cord.
    Arvanian VL, Mendell LM.
    Eur J Neurosci; 2001 Dec 15; 14(11):1800-8. PubMed ID: 11860475
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  • 17. Differential Effects of Invasive Anodal Trans-spinal Direct Current Stimulation on Monosynaptic Excitatory Postsynaptic Potentials, Ia Afferents Excitability, and Motoneuron Intrinsic Properties Between Superoxide Dismutase Type-1 Glycine to Alanine Substitution at Position 93 and Wildtype Mice.
    Jankowiak T, Cholewiński M, Bączyk M.
    Neuroscience; 2022 Aug 21; 498():125-143. PubMed ID: 35792195
    [Abstract] [Full Text] [Related]

  • 18. Increased persistent sodium current determines cortical hyperexcitability in a genetic model of amyotrophic lateral sclerosis.
    Pieri M, Carunchio I, Curcio L, Mercuri NB, Zona C.
    Exp Neurol; 2009 Feb 21; 215(2):368-79. PubMed ID: 19071115
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  • 19. Dysregulation of persistent inward and outward currents in spinal motoneurons of symptomatic SOD1-G93A mice.
    Deutsch AJ, Elbasiouny SM.
    J Physiol; 2024 Aug 21; 602(15):3715-3736. PubMed ID: 38924530
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