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PUBMED FOR HANDHELDS

Journal Abstract Search


127 related items for PubMed ID: 2888690

  • 1.
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  • 2. Rate of chase-promoted hydrolysis of ATP in the high affinity catalytic site of beef heart mitochondrial ATPase.
    Penefsky HS.
    J Biol Chem; 1988 May 05; 263(13):6020-2. PubMed ID: 2896192
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  • 3. Acceleration of unisite catalysis of mitochondrial F1-adenosinetriphosphatase by ATP, ADP and pyrophosphate--hydrolysis and release of the previously bound [gamma-32P]ATP.
    García JJ, Gómez-Puyou A, Maldonado E, Tuena De Gómez-Puyou M.
    Eur J Biochem; 1997 Oct 15; 249(2):622-9. PubMed ID: 9370375
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  • 5. Interaction of mitochondrial F1-ATPase with trinitrophenyl derivatives of ATP. Photoaffinity labeling of binding sites with 2-azido-2',3'-O-(4,6-trinitrophenyl)adenosine 5'-triphosphate.
    Murataliev MB.
    Eur J Biochem; 1995 Sep 01; 232(2):578-85. PubMed ID: 7556210
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  • 8. Aurovertin fluorescence changes of the mitochondrial F1-ATPase during multi- and uni-site ATP hydrolysis.
    Vázquez-Laslop N, Ramírez J, Dreyfus G.
    J Biol Chem; 1989 Oct 15; 264(29):17064-8. PubMed ID: 2529256
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  • 10. The complex of mitochondrial F1-ATPase with the natural inhibitor protein is unable to catalyze single-site ATP hydrolysis.
    Kalashnikova TYu, Milgrom YaM, Postanogova NV.
    FEBS Lett; 1988 Mar 28; 230(1-2):163-6. PubMed ID: 2895018
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  • 11. Cooperatively between catalytic sites in the mechanism of action of beef heart mitochondrial adenosine triphosphatase.
    Grubmeyer C, Penefsky HS.
    J Biol Chem; 1981 Apr 25; 256(8):3728-34. PubMed ID: 6452455
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  • 12. Structural alterations and inhibition of unisite and multisite ATP hydrolysis in soluble mitochondrial F1 by guanidinium chloride.
    Tuena de Gómez-Puyou M, Domínguez-Ramírez L, Reyes-Vivas H, Gómez-Puyou A.
    Biochemistry; 2001 Mar 20; 40(11):3396-402. PubMed ID: 11258961
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  • 15. When beef-heart mitochondrial F1-ATPase is inhibited by inhibitor protein a nucleotide is trapped in one of the catalytic sites.
    Milgrom YM.
    Eur J Biochem; 1991 Sep 15; 200(3):789-95. PubMed ID: 1833193
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  • 16. The native mitochondrial F1-inhibitor protein complex carries out uni- and multisite ATP hydrolysis.
    Vázquez-Laslop N, Dreyfus G.
    J Biol Chem; 1990 Nov 05; 265(31):19002-6. PubMed ID: 2146268
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  • 17. Synthesis and release of ATP by soluble mitochondrial F1 in complex with its inhibitor protein during dimethylsulfoxide-water transitions.
    Tuena de Gómez-Puyou M, Sandoval F, García JJ, Gómez-Puyou A.
    Eur J Biochem; 1998 Jul 01; 255(1):303-8. PubMed ID: 9692932
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  • 18. Identification of the nucleotide-binding site for ATP synthesis and hydrolysis in mitochondrial soluble F1-ATPase.
    Sakamoto J.
    J Biochem; 1984 Aug 01; 96(2):475-81. PubMed ID: 6238951
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  • 19. Kinetic mechanism of Fo x F1 mitochondrial ATPase: Mg2+ requirement for Mg x ATP hydrolysis.
    Syroeshkin AV, Galkin MA, Sedlov AV, Vinogradov AD.
    Biochemistry (Mosc); 1999 Oct 01; 64(10):1128-37. PubMed ID: 10561559
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  • 20. Catalytic cooperativity of beef heart mitochondrial F1-ATPase revealed by using 2',3'-O-(2,4,6-trinitrophenyl)-ATP as a substrate; an indication of mutually activating catalytic sites.
    Muneyuki E, Hisabori T, Allison WS, Jault JM, Sasayama T, Yoshida M.
    Biochim Biophys Acta; 1994 Nov 01; 1188(1-2):108-16. PubMed ID: 7947899
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