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397 related items for PubMed ID: 28931523

  • 1. Contribution of myo-inositol oxygenase in AGE:RAGE-mediated renal tubulointerstitial injury in the context of diabetic nephropathy.
    Sharma I, Tupe RS, Wallner AK, Kanwar YS.
    Am J Physiol Renal Physiol; 2018 Jan 01; 314(1):F107-F121. PubMed ID: 28931523
    [Abstract] [Full Text] [Related]

  • 2. myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience: A NEW MECHANISM RELEVANT TO THE PATHOGENESIS OF DIABETIC NEPHROPATHY.
    Sun L, Dutta RK, Xie P, Kanwar YS.
    J Biol Chem; 2016 Mar 11; 291(11):5688-5707. PubMed ID: 26792859
    [Abstract] [Full Text] [Related]

  • 3. Pathobiology of renal-specific oxidoreductase/myo-inositol oxygenase in diabetic nephropathy: its implications in tubulointerstitial fibrosis.
    Xie P, Sun L, Oates PJ, Srivastava SK, Kanwar YS.
    Am J Physiol Renal Physiol; 2010 Jun 11; 298(6):F1393-404. PubMed ID: 20335317
    [Abstract] [Full Text] [Related]

  • 4. Osteomeles schwerinae extracts inhibits the binding to receptors of advanced glycation end products and TGF-β1 expression in mesangial cells under diabetic conditions.
    Kim YS, Jung DH, Lee IS, Pyun BJ, Kim JS.
    Phytomedicine; 2016 Apr 15; 23(4):388-97. PubMed ID: 27002409
    [Abstract] [Full Text] [Related]

  • 5. Transcriptional and Translational Modulation of myo-Inositol Oxygenase (Miox) by Fatty Acids: IMPLICATIONS IN RENAL TUBULAR INJURY INDUCED IN OBESITY AND DIABETES.
    Tominaga T, Dutta RK, Joladarashi D, Doi T, Reddy JK, Kanwar YS.
    J Biol Chem; 2016 Jan 15; 291(3):1348-67. PubMed ID: 26578517
    [Abstract] [Full Text] [Related]

  • 6. Myo-inositol Oxygenase (MIOX) Overexpression Drives the Progression of Renal Tubulointerstitial Injury in Diabetes.
    Sharma I, Deng F, Liao Y, Kanwar YS.
    Diabetes; 2020 Jun 15; 69(6):1248-1263. PubMed ID: 32169892
    [Abstract] [Full Text] [Related]

  • 7. Disruption of renal tubular mitochondrial quality control by Myo-inositol oxygenase in diabetic kidney disease.
    Zhan M, Usman IM, Sun L, Kanwar YS.
    J Am Soc Nephrol; 2015 Jun 15; 26(6):1304-21. PubMed ID: 25270067
    [Abstract] [Full Text] [Related]

  • 8. Sirt1 resists advanced glycation end products-induced expressions of fibronectin and TGF-β1 by activating the Nrf2/ARE pathway in glomerular mesangial cells.
    Huang K, Huang J, Xie X, Wang S, Chen C, Shen X, Liu P, Huang H.
    Free Radic Biol Med; 2013 Dec 15; 65():528-540. PubMed ID: 23891678
    [Abstract] [Full Text] [Related]

  • 9. Increased expression of myo-inositol oxygenase is involved in the tubulointerstitial injury of diabetic nephropathy.
    Lu Y, Liu C, Miao X, Xu K, Wu X, Liu C.
    Exp Clin Endocrinol Diabetes; 2009 Jun 15; 117(6):257-65. PubMed ID: 19053028
    [Abstract] [Full Text] [Related]

  • 10. Mechanistic role of Syzygium cumini (L.) Skeels in glycation induced diabetic nephropathy via RAGE-NF-κB pathway and extracellular proteins modifications: A molecular approach.
    Apte MM, Khattar E, Tupe RS.
    J Ethnopharmacol; 2024 Mar 25; 322():117573. PubMed ID: 38110133
    [Abstract] [Full Text] [Related]

  • 11. Myo-inositol oxygenase accentuates renal tubular injury initiated by endoplasmic reticulum stress.
    Tominaga T, Sharma I, Fujita Y, Doi T, Wallner AK, Kanwar YS.
    Am J Physiol Renal Physiol; 2019 Feb 01; 316(2):F301-F315. PubMed ID: 30539651
    [Abstract] [Full Text] [Related]

  • 12. High Glucose-Induced Hypomethylation Promotes Binding of Sp-1 to Myo-Inositol Oxygenase: Implication in the Pathobiology of Diabetic Tubulopathy.
    Sharma I, Dutta RK, Singh NK, Kanwar YS.
    Am J Pathol; 2017 Apr 01; 187(4):724-739. PubMed ID: 28208054
    [Abstract] [Full Text] [Related]

  • 13. Modulation of gentamicin-induced acute kidney injury by myo-inositol oxygenase via the ROS/ALOX-12/12-HETE/GPR31 signaling pathway.
    Sharma I, Liao Y, Zheng X, Kanwar YS.
    JCI Insight; 2022 Mar 22; 7(6):. PubMed ID: 35315361
    [Abstract] [Full Text] [Related]

  • 14. AGE-RAGE axis blockade in diabetic nephropathy: Current status and future directions.
    Sanajou D, Ghorbani Haghjo A, Argani H, Aslani S.
    Eur J Pharmacol; 2018 Aug 15; 833():158-164. PubMed ID: 29883668
    [Abstract] [Full Text] [Related]

  • 15. The Kidney Specific Protein myo-Inositol Oxygenase, a Potential Biomarker for Diabetic Nephropathy.
    Gao P, Xu B, Song P, Zhu X, Yuan S, Kanwar YS, Sun L.
    Kidney Blood Press Res; 2018 Aug 15; 43(6):1772-1785. PubMed ID: 30504713
    [Abstract] [Full Text] [Related]

  • 16. Beneficial Effects of Myo-Inositol Oxygenase Deficiency in Cisplatin-Induced AKI.
    Dutta RK, Kondeti VK, Sharma I, Chandel NS, Quaggin SE, Kanwar YS.
    J Am Soc Nephrol; 2017 May 15; 28(5):1421-1436. PubMed ID: 27895157
    [Abstract] [Full Text] [Related]

  • 17. The receptor for advanced glycation endproducts mediates podocyte heparanase expression through NF-κB signaling pathway.
    An X, Zhang L, Yao Q, Li L, Wang B, Zhang J, He M, Zhang J.
    Mol Cell Endocrinol; 2018 Jul 15; 470():14-25. PubMed ID: 28478303
    [Abstract] [Full Text] [Related]

  • 18. Pigment epithelium-derived factor (PEDF) inhibits proximal tubular cell injury in early diabetic nephropathy by suppressing advanced glycation end products (AGEs)-receptor (RAGE) axis.
    Maeda S, Matsui T, Takeuchi M, Yoshida Y, Yamakawa R, Fukami K, Yamagishi S.
    Pharmacol Res; 2011 Mar 15; 63(3):241-8. PubMed ID: 21115116
    [Abstract] [Full Text] [Related]

  • 19. Metformin inhibits advanced glycation end products (AGEs)-induced renal tubular cell injury by suppressing reactive oxygen species generation via reducing receptor for AGEs (RAGE) expression.
    Ishibashi Y, Matsui T, Takeuchi M, Yamagishi S.
    Horm Metab Res; 2012 Nov 15; 44(12):891-5. PubMed ID: 22864903
    [Abstract] [Full Text] [Related]

  • 20. Salvianolic Acid A Protects Against Diabetic Nephropathy through Ameliorating Glomerular Endothelial Dysfunction via Inhibiting AGE-RAGE Signaling.
    Hou B, Qiang G, Zhao Y, Yang X, Chen X, Yan Y, Wang X, Liu C, Zhang L, Du G.
    Cell Physiol Biochem; 2017 Nov 15; 44(6):2378-2394. PubMed ID: 29262395
    [Abstract] [Full Text] [Related]

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