These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


PUBMED FOR HANDHELDS

Journal Abstract Search


169 related items for PubMed ID: 2901260

  • 21. Use of ADP analogs for functional and structural analysis of F1-ATPase.
    Schäfer G, Lücken U, Lübben M.
    Methods Enzymol; 1986; 126():682-712. PubMed ID: 2908477
    [No Abstract] [Full Text] [Related]

  • 22. Interaction of F1-ATPase, from ox heart mitochondria with its naturally occurring inhibitor protein. Studies using radio-iodinated inhibitor protein.
    Power J, Cross RL, Harris DA.
    Biochim Biophys Acta; 1983 Jul 29; 724(1):128-41. PubMed ID: 6223660
    [Abstract] [Full Text] [Related]

  • 23. The adenine nucleotide translocase modulates oligomycin-induced quenching of pyranine fluorescence in submitochondrial particles.
    Ziegler M, Penefsky HS.
    J Biol Chem; 1993 Dec 05; 268(34):25320-8. PubMed ID: 8244963
    [Abstract] [Full Text] [Related]

  • 24. Differential inhibition of F0F1-ATPase-catalysed reactions in bovine-heart submitochondrial particles by organotin compounds.
    Emanuel EL, Carver MA, Solani GC, Griffiths DE.
    Biochim Biophys Acta; 1984 Jul 27; 766(1):209-14. PubMed ID: 6204688
    [Abstract] [Full Text] [Related]

  • 25. Studies on polypeptide composition, hydrolytic activity and proton conduction of mitochondrial FoF1 H+ ATPase in regenerating rat liver.
    Buckle M, Guerrieri F, Pazienza A, Papa S.
    Eur J Biochem; 1986 Mar 03; 155(2):439-45. PubMed ID: 2869946
    [Abstract] [Full Text] [Related]

  • 26. Cation-dependent reassembly of F0F1-ATPase in submitochondrial particles: evidence for a binding site for F1 on F0 in the absence of F6 and oligomycin sensitivity-conferring protein.
    Sandri G, Wojtczak L, Ernster L.
    Arch Biochem Biophys; 1985 Jun 03; 239(2):595-602. PubMed ID: 2860874
    [Abstract] [Full Text] [Related]

  • 27.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 28.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 29.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 30. Resolution and reconstitution of F0F1-ATPase in beef heart submitochondrial particles.
    Ernster L, Hundal T, Sandri G.
    Methods Enzymol; 1986 Jun 03; 126():428-33. PubMed ID: 2908456
    [No Abstract] [Full Text] [Related]

  • 31. Isolation of alpha-subunits of factor F1 from submitochondrial particles and the reconstitution of active ATPase from isolated alpha-subunits and beta-subunits bound to the mitochondrial membrane.
    Kozlov IA, Milgrom YM, Tsybovski IS.
    Biochem J; 1980 Nov 15; 192(2):483-8. PubMed ID: 6453586
    [Abstract] [Full Text] [Related]

  • 32. Relevance of divalent cations to ATP-driven proton pumping in beef heart mitochondrial F0F1-ATPase.
    Papageorgiou S, Melandri AB, Solaini G.
    J Bioenerg Biomembr; 1998 Dec 15; 30(6):533-41. PubMed ID: 10206473
    [Abstract] [Full Text] [Related]

  • 33. Pressure effects on the interaction between natural inhibitor protein and mitochondrial F1-ATPase.
    Fornells LA, Guimarães-Motta H, Nehme JS, Martins OB, Silva JL.
    Arch Biochem Biophys; 1998 Jan 15; 349(2):304-12. PubMed ID: 9448719
    [Abstract] [Full Text] [Related]

  • 34. Mitochondrial H+-ATPase activation by an amine oxide detergent.
    Vázquez-Laslop N, Dreyfus G.
    J Biol Chem; 1986 Jun 15; 261(17):7807-10. PubMed ID: 2872219
    [Abstract] [Full Text] [Related]

  • 35. Kinetic analysis of proton translocation catalyzed by F0F1 ATPase.
    Muneyuki E, Hirata H.
    FEBS Lett; 1988 Jul 18; 234(2):455-8. PubMed ID: 2899034
    [Abstract] [Full Text] [Related]

  • 36.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 37. Effect of chemical modifiers of amino acid residues on proton conduction by the H+-ATPase of mitochondria.
    Guerrieri F, Papa S.
    J Bioenerg Biomembr; 1981 Dec 18; 13(5-6):393-409. PubMed ID: 6460757
    [Abstract] [Full Text] [Related]

  • 38. Oligomycin sensitivity conferring protein of mitochondrial ATP synthase: deletions in the N-terminal end cause defects in interactions with F1, while deletions in the C-terminal end cause defects in interactions with F0.
    Joshi S, Cao GJ, Nath C, Shah J.
    Biochemistry; 1996 Sep 17; 35(37):12094-103. PubMed ID: 8810915
    [Abstract] [Full Text] [Related]

  • 39. ATP synthase complex from bovine heart mitochondria. Passive H+ conduction through mitochondrial coupling factor 6-depleted F0 complexes.
    Joshi S, Pringle MJ.
    J Biol Chem; 1989 Sep 15; 264(26):15548-51. PubMed ID: 2527853
    [Abstract] [Full Text] [Related]

  • 40.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]


    Page: [Previous] [Next] [New Search]
    of 9.