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Journal Abstract Search

238 related items for PubMed ID: 29051216

  • 41. Prolyl hydroxylase 3 overexpression accelerates the progression of atherosclerosis in ApoE-/- mice.
    Liu H, Xia Y, Li B, Pan J, Lv M, Wang X, An F.
    Biochem Biophys Res Commun; 2016 Apr 22; 473(1):99-106. PubMed ID: 26995088
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  • 42. PHD1 interacts with ATF4 and negatively regulates its transcriptional activity without prolyl hydroxylation.
    Hiwatashi Y, Kanno K, Takasaki C, Goryo K, Sato T, Torii S, Sogawa K, Yasumoto K.
    Exp Cell Res; 2011 Dec 10; 317(20):2789-99. PubMed ID: 21951999
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  • 43. Diosgenin alleviates arsenic trioxide induced cardiac fibrosis by inhibiting endothelial mesenchymal transition.
    Cui H, Xu W, Liu L, Hong Y, Lou H, Tang P, Lin Y, Xu H, Xie M, Du M, Tang X, Wang Z, Wang Q, Zhang Y.
    Phytomedicine; 2024 Sep 10; 132():155891. PubMed ID: 39059093
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  • 44. Non-muscular myosin light chain kinase triggers intermittent hypoxia-induced interleukin-6 release, endothelial dysfunction and permeability.
    Recoquillon S, Gómez-Guzmán M, Rodier M, Koffi C, Nitiéma M, Gagnadoux F, Martínez MC, Andriantsitohaina R.
    Sci Rep; 2017 Oct 20; 7(1):13664. PubMed ID: 29057883
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  • 50. Suppression of ADAM8 attenuates angiotensin II-induced cardiac fibrosis and endothelial-mesenchymal transition via inhibiting TGF-β1/Smad2/Smad3 pathways.
    Yao L, Shao W, Chen Y, Wang S, Huang D.
    Exp Anim; 2022 Feb 09; 71(1):90-99. PubMed ID: 34615811
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  • 51. Endothelial-to-mesenchymal transition contributes to fibro-proliferative vascular disease and is modulated by fluid shear stress.
    Moonen JR, Lee ES, Schmidt M, Maleszewska M, Koerts JA, Brouwer LA, van Kooten TG, van Luyn MJ, Zeebregts CJ, Krenning G, Harmsen MC.
    Cardiovasc Res; 2015 Dec 01; 108(3):377-86. PubMed ID: 26084310
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  • 52. Inhibition of miR-21 alleviated cardiac perivascular fibrosis via repressing EndMT in T1DM.
    Li Q, Yao Y, Shi S, Zhou M, Zhou Y, Wang M, Chiu JJ, Huang Z, Zhang W, Liu M, Wang Q, Tu X.
    J Cell Mol Med; 2020 Jan 01; 24(1):910-920. PubMed ID: 31680453
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  • 55. Loss of CLOCK under high glucose upregulates ROCK1-mediated endothelial to mesenchymal transition and aggravates plaque vulnerability.
    Tang H, Zhu M, Zhao G, Fu W, Shi Z, Ding Y, Tang X, Guo D.
    Atherosclerosis; 2018 Aug 01; 275():58-67. PubMed ID: 29860109
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  • 56. SIRT1 activation attenuates cardiac fibrosis by endothelial-to-mesenchymal transition.
    Liu ZH, Zhang Y, Wang X, Fan XF, Zhang Y, Li X, Gong YS, Han LP.
    Biomed Pharmacother; 2019 Oct 01; 118():109227. PubMed ID: 31351433
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  • 57. Loss of PHD3 allows tumours to overcome hypoxic growth inhibition and sustain proliferation through EGFR.
    Henze AT, Garvalov BK, Seidel S, Cuesta AM, Ritter M, Filatova A, Foss F, Dopeso H, Essmann CL, Maxwell PH, Reifenberger G, Carmeliet P, Acker-Palmer A, Acker T.
    Nat Commun; 2014 Nov 25; 5():5582. PubMed ID: 25420773
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  • 58. MALAT1 Modulates TGF-β1-Induced Endothelial-to-Mesenchymal Transition through Downregulation of miR-145.
    Xiang Y, Zhang Y, Tang Y, Li Q.
    Cell Physiol Biochem; 2017 Nov 25; 42(1):357-372. PubMed ID: 28535533
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  • 59. Endothelial Cell-Specific Prolyl Hydroxylase-2 Deficiency Augments Angiotensin II-Induced Arterial Stiffness and Cardiac Pericyte Recruitment in Mice.
    Liu B, Zeng H, Su H, Williams QA, Besanson J, Chen Y, Chen JX.
    J Am Heart Assoc; 2024 Aug 20; 13(16):e035769. PubMed ID: 39056332
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