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Journal Abstract Search

138 related items for PubMed ID: 2906199

  • 1. Calcium accumulation by glutamate receptor activation is involved in hippocampal cell damage after ischemia.
    Benveniste H, Jørgensen MB, Diemer NH, Hansen AJ.
    Acta Neurol Scand; 1988 Dec; 78(6):529-36. PubMed ID: 2906199
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  • 2. Participation of NMDA-receptors in ischemic changes of calcium homeostasis in rabbit brain.
    Salinska E, Pluta R, Lazarewicz JW.
    Biomed Biochim Acta; 1989 Dec; 48(2-3):S170-3. PubMed ID: 2567159
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  • 3. Effects of D-(-)-aminophosphonovalerate on behavioral and histological changes induced by systemic kainic acid.
    Lason W, Simpson JN, McGinty JF.
    Neurosci Lett; 1988 Apr 22; 87(1-2):23-8. PubMed ID: 2898113
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  • 4. Blockade of N-methyl-D-aspartate-sensitive excitatory amino acid receptors with 2-amino-5-phosphonovalerate reduces ischemia-evoked calcium redistribution in rabbit hippocampus.
    Salińska E, Pluta R, Puka M, Lazarewicz JW.
    Exp Neurol; 1991 Apr 22; 112(1):89-94. PubMed ID: 1672853
    [Abstract] [Full Text] [Related]

  • 5. Ischemic CA-1 pyramidal cell loss is prevented by preischemic colchicine destruction of dentate gyrus granule cells.
    Johansen FF, Jørgensen MB, Diemer NH.
    Brain Res; 1986 Jul 09; 377(2):344-7. PubMed ID: 2873869
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  • 6. The expression of N-methyl-D-aspartate-receptor-mediated component during epileptiform synaptic activity in hippocampus.
    Ashwood TJ, Wheal HV.
    Br J Pharmacol; 1987 Aug 09; 91(4):815-22. PubMed ID: 2889490
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  • 7. Intracellular calcium levels and calcium fluxes in the CA1 region of the rat hippocampal slice during in vitro ischemia: relationship to electrophysiological cell damage.
    Lobner D, Lipton P.
    J Neurosci; 1993 Nov 09; 13(11):4861-71. PubMed ID: 8229202
    [Abstract] [Full Text] [Related]

  • 8. Quantitative correlation between tetanus-induced decreases in extracellular calcium and LTP.
    Melchers BP, Pennartz CM, Wadman WJ, Lopes da Silva FH.
    Brain Res; 1988 Jun 28; 454(1-2):1-10. PubMed ID: 2900660
    [Abstract] [Full Text] [Related]

  • 9. Degeneration of hippocampal CA1 neurons following transient ischemia due to raised intracranial pressure: evidence for a temperature-dependent excitotoxic process.
    Duhaime AC, Ross DT.
    Brain Res; 1990 Mar 26; 512(1):169-74. PubMed ID: 1970944
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  • 13. The role of early Ca2+ influx in the pathogenesis of delayed neuronal death after brief forebrain ischemia in gerbils.
    Nakamura K, Hatakeyama T, Furuta S, Sakaki S.
    Brain Res; 1993 Jun 11; 613(2):181-92. PubMed ID: 8186966
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  • 15. Experimental ischemia induces a persistent depolarization blocked by decreased calcium and NMDA antagonists.
    Rader RK, Lanthorn TH.
    Neurosci Lett; 1989 Apr 24; 99(1-2):125-30. PubMed ID: 2546106
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  • 16. NMDA receptor activation accelerates ischemic energy depletion in the hippocampal slice and the demonstration of a threshold for ischemic damage to protein synthesis.
    Raley KM, Lipton P.
    Neurosci Lett; 1990 Mar 02; 110(1-2):118-23. PubMed ID: 2158012
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  • 17. Nitric oxide synthase inhibition and extracellular glutamate concentration after cerebral ischemia/reperfusion.
    Zhang J, Benveniste H, Klitzman B, Piantadosi CA.
    Stroke; 1995 Feb 02; 26(2):298-304. PubMed ID: 7530389
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  • 18. NMDA antagonists: lack of protective effect against hypoxic damage in CA1 region of hippocampal slices.
    Aitken PG, Balestrino M, Somjen GG.
    Neurosci Lett; 1988 Jun 29; 89(2):187-92. PubMed ID: 2899309
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  • 19. Extracellular accumulation of glutamate in the hippocampus induced by ischemia is not calcium dependent--in vitro and in vivo evidence.
    Ikeda M, Nakazawa T, Abe K, Kaneko T, Yamatsu K.
    Neurosci Lett; 1989 Jan 16; 96(2):202-6. PubMed ID: 2564653
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