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Journal Abstract Search


237 related items for PubMed ID: 29084939

  • 21. Protein kinase C alpha trigger Ras and Raf-independent MEK/ERK activation for TPA-induced growth inhibition of human hepatoma cell HepG2.
    Wen-Sheng W.
    Cancer Lett; 2006 Jul 28; 239(1):27-35. PubMed ID: 16169661
    [Abstract] [Full Text] [Related]

  • 22. Classical RAS proteins are not essential for paradoxical ERK activation induced by RAF inhibitors.
    Lai LP, Fer N, Burgan W, Wall VE, Xu B, Soppet D, Esposito D, Nissley DV, McCormick F.
    Proc Natl Acad Sci U S A; 2022 Feb 01; 119(5):. PubMed ID: 35091470
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  • 23. Involvement of Ras/Raf-1/ERK actions in the magnolol-induced upregulation of p21 and cell-cycle arrest in colon cancer cells.
    Hsu YF, Lee TS, Lin SY, Hsu SP, Juan SH, Hsu YH, Zhong WB, Lee WS.
    Mol Carcinog; 2007 Apr 01; 46(4):275-83. PubMed ID: 17295239
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  • 24. Inhibitors of Raf kinase activity block growth of thyroid cancer cells with RET/PTC or BRAF mutations in vitro and in vivo.
    Ouyang B, Knauf JA, Smith EP, Zhang L, Ramsey T, Yusuff N, Batt D, Fagin JA.
    Clin Cancer Res; 2006 Mar 15; 12(6):1785-93. PubMed ID: 16551863
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  • 33. Targeting the RAF-MEK-ERK pathway in cancer therapy.
    Montagut C, Settleman J.
    Cancer Lett; 2009 Oct 08; 283(2):125-34. PubMed ID: 19217204
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  • 34. SPRED1 Interferes with K-ras but Not H-ras Membrane Anchorage and Signaling.
    Siljamäki E, Abankwa D.
    Mol Cell Biol; 2016 Oct 15; 36(20):2612-25. PubMed ID: 27503857
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  • 35. Inhibition of RAF Isoforms and Active Dimers by LY3009120 Leads to Anti-tumor Activities in RAS or BRAF Mutant Cancers.
    Peng SB, Henry JR, Kaufman MD, Lu WP, Smith BD, Vogeti S, Rutkoski TJ, Wise S, Chun L, Zhang Y, Van Horn RD, Yin T, Zhang X, Yadav V, Chen SH, Gong X, Ma X, Webster Y, Buchanan S, Mochalkin I, Huber L, Kays L, Donoho GP, Walgren J, McCann D, Patel P, Conti I, Plowman GD, Starling JJ, Flynn DL.
    Cancer Cell; 2015 Sep 14; 28(3):384-98. PubMed ID: 26343583
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  • 36. Interferon-alpha2b reduces phosphorylation and activity of MEK and ERK through a Ras/Raf-independent mechanism.
    Romerio F, Riva A, Zella D.
    Br J Cancer; 2000 Aug 14; 83(4):532-8. PubMed ID: 10945503
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  • 37. Radicicol suppresses transformation and restores tropomyosin-2 expression in both ras- and MEK-transformed cells without inhibiting the Raf/MEK/ERK signaling cascade.
    Kim PN, Jonasch E, Mosterman BC, Mier JW, Janssen RA.
    Cell Growth Differ; 2001 Nov 14; 12(11):543-50. PubMed ID: 11714635
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  • 38. RAF inhibitors that evade paradoxical MAPK pathway activation.
    Zhang C, Spevak W, Zhang Y, Burton EA, Ma Y, Habets G, Zhang J, Lin J, Ewing T, Matusow B, Tsang G, Marimuthu A, Cho H, Wu G, Wang W, Fong D, Nguyen H, Shi S, Womack P, Nespi M, Shellooe R, Carias H, Powell B, Light E, Sanftner L, Walters J, Tsai J, West BL, Visor G, Rezaei H, Lin PS, Nolop K, Ibrahim PN, Hirth P, Bollag G.
    Nature; 2015 Oct 22; 526(7574):583-6. PubMed ID: 26466569
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  • 39. An Integrated Model of RAF Inhibitor Action Predicts Inhibitor Activity against Oncogenic BRAF Signaling.
    Karoulia Z, Wu Y, Ahmed TA, Xin Q, Bollard J, Krepler C, Wu X, Zhang C, Bollag G, Herlyn M, Fagin JA, Lujambio A, Gavathiotis E, Poulikakos PI.
    Cancer Cell; 2016 Sep 12; 30(3):485-498. PubMed ID: 27523909
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  • 40. Neuronal nitric oxide synthase-induced S-nitrosylation of H-Ras inhibits calcium ionophore-mediated extracellular-signal-regulated kinase activity.
    Raines KW, Cao GL, Lee EK, Rosen GM, Shapiro P.
    Biochem J; 2006 Jul 15; 397(2):329-36. PubMed ID: 16569214
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