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Journal Abstract Search

455 related items for PubMed ID: 29187895

  • 1. Follicular Stimulating Hormone Accelerates Atherogenesis by Increasing Endothelial VCAM-1 Expression.
    Li X, Chen W, Li P, Wei J, Cheng Y, Liu P, Yan Q, Xu X, Cui Y, Gu Z, Simoncini T, Fu X.
    Theranostics; 2017; 7(19):4671-4688. PubMed ID: 29187895
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  • 2. Follicle-Stimulating Hormone Accelerates Atherosclerosis by Activating PI3K/Akt/NF-κB Pathway in Mice with Androgen Deprivation.
    Piao J, Yin Y, Zhao Y, Han Y, Zhan H, Luo D, Guo J.
    J Vasc Res; 2022; 59(6):358-368. PubMed ID: 36412620
    [Abstract] [Full Text] [Related]

  • 3. Visfatin Promotes Monocyte Adhesion by Upregulating ICAM-1 and VCAM-1 Expression in Endothelial Cells via Activation of p38-PI3K-Akt Signaling and Subsequent ROS Production and IKK/NF-κB Activation.
    Lin YT, Chen LK, Jian DY, Hsu TC, Huang WC, Kuan TT, Wu SY, Kwok CF, Ho LT, Juan CC.
    Cell Physiol Biochem; 2019; 52(6):1398-1411. PubMed ID: 31075190
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  • 4. [Effects of PI3K/Akt/NF-κB signal pathway on FSH facilitation on cell proliferation and invasion by human epithelial ovarian cancer].
    Xu CL, Lu XL, Yan XN, Wang HL, Chen SQ.
    Zhonghua Fu Chan Ke Za Zhi; 2012 Feb; 47(2):134-8. PubMed ID: 22455747
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  • 5. Hypoxia-induced mitogenic factor promotes vascular adhesion molecule-1 expression via the PI-3K/Akt-NF-kappaB signaling pathway.
    Tong Q, Zheng L, Lin L, Li B, Wang D, Li D.
    Am J Respir Cell Mol Biol; 2006 Oct; 35(4):444-56. PubMed ID: 16709959
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  • 6. FSH Is Responsible for Androgen Deprivation Therapy-Associated Atherosclerosis in Mice by Exaggerating Endothelial Inflammation and Monocyte Adhesion.
    Wang Q, Han J, Liang Z, Geng X, Du Y, Zhou J, Yao W, Xu T.
    Arterioscler Thromb Vasc Biol; 2024 Mar; 44(3):698-719. PubMed ID: 38205641
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  • 12. Soy protein inhibits inflammation-induced VCAM-1 and inflammatory cytokine induction by inhibiting the NF-κB and AKT signaling pathway in apolipoprotein E-deficient mice.
    Burris RL, Ng HP, Nagarajan S.
    Eur J Nutr; 2014 Feb; 53(1):135-48. PubMed ID: 23468309
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  • 13. Salusin-β accelerates inflammatory responses in vascular endothelial cells via NF-κB signaling in LDL receptor-deficient mice in vivo and HUVECs in vitro.
    Koya T, Miyazaki T, Watanabe T, Shichiri M, Atsumi T, Kim-Kaneyama JR, Miyazaki A.
    Am J Physiol Heart Circ Physiol; 2012 Jul; 303(1):H96-105. PubMed ID: 22561298
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  • 14. Role of FSH and FSH receptor on HUVECs migration.
    Tan D, Zhao Y, Ma D, Tong F.
    Gene Ther; 2021 Apr; 28(3-4):155-161. PubMed ID: 32994568
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  • 15. Dihydrotestosterone promotes vascular cell adhesion molecule-1 expression in male human endothelial cells via a nuclear factor-kappaB-dependent pathway.
    Death AK, McGrath KC, Sader MA, Nakhla S, Jessup W, Handelsman DJ, Celermajer DS.
    Endocrinology; 2004 Apr; 145(4):1889-97. PubMed ID: 14684616
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  • 17. Naringin inhibits TNF-α induced oxidative stress and inflammatory response in HUVECs via Nox4/NF-κ B and PI3K/Akt pathways.
    Li W, Wang C, Peng J, Liang J, Jin Y, Liu Q, Meng Q, Liu K, Sun H.
    Curr Pharm Biotechnol; 2014 Apr; 15(12):1173-82. PubMed ID: 25391245
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  • 18. RGC-32 (Response Gene to Complement 32) Deficiency Protects Endothelial Cells From Inflammation and Attenuates Atherosclerosis.
    Cui XB, Luan JN, Dong K, Chen S, Wang Y, Watford WT, Chen SY.
    Arterioscler Thromb Vasc Biol; 2018 Apr; 38(4):e36-e47. PubMed ID: 29449334
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