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Journal Abstract Search

166 related items for PubMed ID: 2923609

  • 1. High-energy phosphates, myocardial contractile function and material properties after short periods of oxygen deficiency.
    Hoffmeister HM, Storf R, Thiedemann KU, Seipel L.
    Basic Res Cardiol; 1989; 84(1):77-90. PubMed ID: 2923609
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  • 2. Tolerance of myocardium of aged animals to repeated oxygen deficiency.
    Hoffmeister HM, Seipel L.
    Basic Res Cardiol; 1992; 87(2):161-72. PubMed ID: 1590738
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  • 3. Effects of graded intensity of oxygen deficiency on function and energy metabolism in post-ischaemic myocardium.
    Hoffmeister HM, Storf R, Seipel L.
    Cardiovasc Res; 1988 Dec; 22(12):881-8. PubMed ID: 3256428
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  • 4. Enhanced sensitivity to hypoxia-induced diastolic dysfunction in pressure-overload left ventricular hypertrophy in the rat: role of high-energy phosphate depletion.
    Wexler LF, Lorell BH, Momomura S, Weinberg EO, Ingwall JS, Apstein CS.
    Circ Res; 1988 Apr; 62(4):766-75. PubMed ID: 2964946
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  • 5. Phosphocreatine and adenine nucleotides in postasphyxial hearts with normal basal function and normal oxygen demand.
    Hoffmeister HM, Stein G, Storf R, Seipel L.
    Basic Res Cardiol; 1987 Apr; 82 Suppl 2():311-6. PubMed ID: 3663024
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  • 6. Energy generation in hypertrophied postischemic myocardium. Feasibility of prolonged inotropic stimulation with dopamine in hypertrophied reperfused left ventricles.
    Hoffmeister HM, Kappelmann A, Beyer ME, Seipel L.
    Basic Res Cardiol; 1998 Jun; 93(3):201-8. PubMed ID: 9689446
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  • 7. Myocardial high-energy phosphates and function under different postischemic conditions. A study in a paracorporeal rat heart model.
    Hultman J, Ronquist G.
    Eur Surg Res; 1984 Jun; 16(4):201-13. PubMed ID: 6745308
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  • 11. Substrate-induced alterations of high energy phosphate metabolism and contractile function in the perfused heart.
    Zweier JL, Jacobus WE.
    J Biol Chem; 1987 Jun 15; 262(17):8015-21. PubMed ID: 3597359
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  • 14. Preserved high energy phosphate metabolic reserve in globally "stunned" hearts despite reduction of basal ATP content and contractility.
    Ambrosio G, Jacobus WE, Bergman CA, Weisman HF, Becker LC.
    J Mol Cell Cardiol; 1987 Oct 15; 19(10):953-64. PubMed ID: 3437454
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  • 15. Protection against injury during ischemia and reperfusion by acadesine derivatives GP-1-468 and GP-1-668. Studies in the transplanted rat heart.
    Galiñanes M, Zhai X, Bullough D, Mullane KM, Hearse DJ.
    J Thorac Cardiovasc Surg; 1995 Sep 15; 110(3):752-61. PubMed ID: 7564443
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  • 16. Correlation of contractile dysfunction with oxidative energy production and tissue high energy phosphate stores during partial coronary flow disruption in rabbit heart.
    Marshall RC.
    J Clin Invest; 1988 Jul 15; 82(1):86-95. PubMed ID: 3392219
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  • 17. Contractile failure and high-energy phosphate turnover during hypoxia: 31P-NMR surface coil studies in living rat.
    Bittl JA, Balschi JA, Ingwall JS.
    Circ Res; 1987 Jun 15; 60(6):871-8. PubMed ID: 2954720
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  • 18. Metabolic, functional, and histologic characterization of the heterotopically transplanted rat heart when used as a model for the study of long-term recovery from global ischemia.
    Galiñanes M, Hearse DJ.
    J Heart Lung Transplant; 1991 Jun 15; 10(1 Pt 1):79-91. PubMed ID: 2007173
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  • 19. Adenine nucleotide metabolites are beneficial for recovery of cardiac contractile force after hypoxia.
    Takeo S, Tanonaka K, Miyake K, Imago M.
    J Mol Cell Cardiol; 1988 Mar 15; 20(3):187-99. PubMed ID: 3398053
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  • 20. Differences in nucleotide compartmentation and energy state in isolated and in situ rat heart: assessment by 31P-NMR spectroscopy.
    Williams JP, Headrick JP.
    Biochim Biophys Acta; 1996 Aug 07; 1276(1):71-9. PubMed ID: 8764892
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