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359 related items for PubMed ID: 29268264
1. Progressive Reduction in Myocyte Autophagy After Myocardial Infarction in Rabbits: Association with Oxidative Stress and Left Ventricular Remodeling. Chi RF, Wang JP, Wang K, Zhang XL, Zhang YA, Kang YM, Han XB, Li B, Qin FZ, Fan BA. Cell Physiol Biochem; 2017; 44(6):2439-2454. PubMed ID: 29268264 [Abstract] [Full Text] [Related]
2. Activation of NADPH oxidase mediates increased endoplasmic reticulum stress and left ventricular remodeling after myocardial infarction in rabbits. Li B, Tian J, Sun Y, Xu TR, Chi RF, Zhang XL, Hu XL, Zhang YA, Qin FZ, Zhang WF. Biochim Biophys Acta; 2015 May; 1852(5):805-15. PubMed ID: 25615792 [Abstract] [Full Text] [Related]
3. Progressive left ventricular remodeling, myocyte apoptosis, and protein signaling cascades after myocardial infarction in rabbits. Qin F, Liang MC, Liang CS. Biochim Biophys Acta; 2005 Jun 10; 1740(3):499-513. PubMed ID: 15949720 [Abstract] [Full Text] [Related]
4. Sphingosine-1-phosphate induces myocyte autophagy after myocardial infarction through mTOR inhibition. Yang LG, Wang AL, Li L, Yang H, Jie X, Zhu ZF, Zhang XJ, Zhao HP, Chi RF, Li B, Qin FZ, Wang JP, Wang K. Eur J Pharmacol; 2021 Sep 15; 907():174260. PubMed ID: 34144026 [Abstract] [Full Text] [Related]
5. Antioxidant N-acetylcysteine inhibits maladaptive myocyte autophagy in pressure overload induced cardiac remodeling in rats. Li B, Sun Y, Wang JP, Chi RF, Wang K, Yang ZJ, Qin FZ, Fan B. Eur J Pharmacol; 2018 Nov 15; 839():47-56. PubMed ID: 30194941 [Abstract] [Full Text] [Related]
6. Oxidative stress impairs myocyte autophagy, resulting in myocyte hypertrophy. Wang JP, Chi RF, Wang K, Ma T, Guo XF, Zhang XL, Li B, Qin FZ, Han XB, Fan BA. Exp Physiol; 2018 Apr 01; 103(4):461-472. PubMed ID: 29327381 [Abstract] [Full Text] [Related]
7. Dihydroxyflavonol reduces post-infarction left ventricular remodeling by preventing myocyte apoptosis in the non-infarcted zone in goats. Wang S, Fei K, Xu YW, Wang LX, Chen YQ. Chin Med J (Engl); 2009 Jan 05; 122(1):61-7. PubMed ID: 19187619 [Abstract] [Full Text] [Related]
8. Early administration of Enalapril prevents diastolic dysfunction and ventricular remodeling in rabbits with myocardial infarction. González GE, Wilensky L, Cassaglia P, Morales C, Gelpi RJ. Cardiovasc Pathol; 2016 Jan 05; 25(3):208-213. PubMed ID: 26897484 [Abstract] [Full Text] [Related]
9. Apocynin prevents reduced myocardial nerve growth factor, contributing to amelioration of myocardial apoptosis and failure. Jie X, Yang H, Wang K, Zhu ZF, Wang JP, Yang LG, Yang ZJ, Zhang XJ, Wang AL, Li L, Chi RF, Qin FZ, Li B, Fan B. Clin Exp Pharmacol Physiol; 2021 May 05; 48(5):704-716. PubMed ID: 33650189 [Abstract] [Full Text] [Related]
10. Inhibition of NADPH oxidase reduces myocardial oxidative stress and apoptosis and improves cardiac function in heart failure after myocardial infarction. Qin F, Simeone M, Patel R. Free Radic Biol Med; 2007 Jul 15; 43(2):271-81. PubMed ID: 17603936 [Abstract] [Full Text] [Related]
11. Distinct changes of myocyte autophagy during myocardial hypertrophy and heart failure: association with oxidative stress. Li B, Chi RF, Qin FZ, Guo XF. Exp Physiol; 2016 Aug 01; 101(8):1050-63. PubMed ID: 27219474 [Abstract] [Full Text] [Related]
12. Endothelial nitric oxide synthase limits left ventricular remodeling after myocardial infarction in mice. Scherrer-Crosbie M, Ullrich R, Bloch KD, Nakajima H, Nasseri B, Aretz HT, Lindsey ML, Vançon AC, Huang PL, Lee RT, Zapol WM, Picard MH. Circulation; 2001 Sep 11; 104(11):1286-91. PubMed ID: 11551881 [Abstract] [Full Text] [Related]
13. Enhanced oxidative stress mediates pathological autophagy and necroptosis in cardiac myocytes in pressure overload induced heart failure in rats. Chi RF, Li L, Wang AL, Yang H, Xi J, Zhu ZF, Wang K, Li B, Yang LG, Qin FZ, Zhang C. Clin Exp Pharmacol Physiol; 2022 Jan 11; 49(1):60-69. PubMed ID: 34453856 [Abstract] [Full Text] [Related]
14. Vitamins C and E attenuate apoptosis, beta-adrenergic receptor desensitization, and sarcoplasmic reticular Ca2+ ATPase downregulation after myocardial infarction. Qin F, Yan C, Patel R, Liu W, Dong E. Free Radic Biol Med; 2006 May 15; 40(10):1827-42. PubMed ID: 16678021 [Abstract] [Full Text] [Related]
15. p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat. See F, Thomas W, Way K, Tzanidis A, Kompa A, Lewis D, Itescu S, Krum H. J Am Coll Cardiol; 2004 Oct 19; 44(8):1679-89. PubMed ID: 15489104 [Abstract] [Full Text] [Related]