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PUBMED FOR HANDHELDS

Journal Abstract Search


211 related items for PubMed ID: 29304807

  • 1. Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.
    Gentile A, Musella A, De Vito F, Fresegna D, Bullitta S, Rizzo FR, Centonze D, Mandolesi G.
    J Neuroinflammation; 2018 Jan 05; 15(1):5. PubMed ID: 29304807
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  • 2. Expression of glutamate transporters GLT-1 and GLAST in different regions of rat brain during the course of experimental autoimmune encephalomyelitis.
    Mitosek-Szewczyk K, Sulkowski G, Stelmasiak Z, Struzyńska L.
    Neuroscience; 2008 Jul 31; 155(1):45-52. PubMed ID: 18572325
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  • 3. Laquinimod prevents inflammation-induced synaptic alterations occurring in experimental autoimmune encephalomyelitis.
    Ruffini F, Rossi S, Bergamaschi A, Brambilla E, Finardi A, Motta C, Studer V, Barbieri F, De Chiara V, Hayardeny L, Comi G, Centonze D, Martino G.
    Mult Scler; 2013 Jul 31; 19(8):1084-94. PubMed ID: 23232603
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  • 10. Modulation of autoimmune demyelination by laquinimod via induction of brain-derived neurotrophic factor.
    Thöne J, Ellrichmann G, Seubert S, Peruga I, Lee DH, Conrad R, Hayardeny L, Comi G, Wiese S, Linker RA, Gold R.
    Am J Pathol; 2012 Jan 31; 180(1):267-74. PubMed ID: 22152994
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  • 11. Laquinimod arrests experimental autoimmune encephalomyelitis by activating the aryl hydrocarbon receptor.
    Kaye J, Piryatinsky V, Birnberg T, Hingaly T, Raymond E, Kashi R, Amit-Romach E, Caballero IS, Towfic F, Ator MA, Rubinstein E, Laifenfeld D, Orbach A, Shinar D, Marantz Y, Grossman I, Knappertz V, Hayden MR, Laufer R.
    Proc Natl Acad Sci U S A; 2016 Oct 11; 113(41):E6145-E6152. PubMed ID: 27671624
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  • 13. IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
    Mandolesi G, Gentile A, Musella A, Centonze D.
    Cerebellum; 2015 Feb 11; 14(1):19-22. PubMed ID: 25326653
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  • 14. Glutamate, T cells and multiple sclerosis.
    Levite M.
    J Neural Transm (Vienna); 2017 Jul 11; 124(7):775-798. PubMed ID: 28236206
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  • 17. Glutamate induced neonatal excitotoxicity modifies the expression level of EAAT1 (GLAST) and EAAT2 (GLT-1) proteins in various brain regions of the adult rat.
    Castañeda-Cabral JL, López-Ortega JG, Fajardo-Fregoso BF, Beas-Zárate C, Ureña-Guerrero ME.
    Neurosci Lett; 2020 Sep 14; 735():135237. PubMed ID: 32645399
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  • 18. Blocking glutamate carboxypeptidase II inhibits glutamate excitotoxicity and regulates immune responses in experimental autoimmune encephalomyelitis.
    Ha D, Bing SJ, Ahn G, Kim J, Cho J, Kim A, Herath KH, Yu HS, Jo SA, Cho IH, Jee Y.
    FEBS J; 2016 Sep 14; 283(18):3438-56. PubMed ID: 27444540
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  • 19. Valproic acid attenuates manganese-induced reduction in expression of GLT-1 and GLAST with concomitant changes in murine dopaminergic neurotoxicity.
    Johnson J, Pajarillo E, Karki P, Kim J, Son DS, Aschner M, Lee E.
    Neurotoxicology; 2018 Jul 14; 67():112-120. PubMed ID: 29778792
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  • 20. Transient receptor potential vanilloid 1 channels modulate the synaptic effects of TNF-α and of IL-1β in experimental autoimmune encephalomyelitis.
    Musumeci G, Grasselli G, Rossi S, De Chiara V, Musella A, Motta C, Studer V, Bernardi G, Haji N, Sepman H, Fresegna D, Maccarrone M, Mandolesi G, Centonze D.
    Neurobiol Dis; 2011 Sep 14; 43(3):669-77. PubMed ID: 21672630
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