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Journal Abstract Search


425 related items for PubMed ID: 29378298

  • 1. miR-34a deficiency in APP/PS1 mice promotes cognitive function by increasing synaptic plasticity via AMPA and NMDA receptors.
    Xu Y, Chen P, Wang X, Yao J, Zhuang S.
    Neurosci Lett; 2018 Mar 23; 670():94-104. PubMed ID: 29378298
    [Abstract] [Full Text] [Related]

  • 2. Over-expression of miR-34a induces rapid cognitive impairment and Alzheimer's disease-like pathology.
    Sarkar S, Engler-Chiurazzi EB, Cavendish JZ, Povroznik JM, Russell AE, Quintana DD, Mathers PH, Simpkins JW.
    Brain Res; 2019 Oct 15; 1721():146327. PubMed ID: 31295467
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  • 3. Disrupted Maturation of Prefrontal Layer 5 Neuronal Circuits in an Alzheimer's Mouse Model of Amyloid Deposition.
    Chen C, Wei J, Ma X, Xia B, Shakir N, Zhang JK, Zhang L, Cui Y, Ferguson D, Qiu S, Bai F.
    Neurosci Bull; 2023 Jun 15; 39(6):881-892. PubMed ID: 36152121
    [Abstract] [Full Text] [Related]

  • 4. Folic acid deficiency enhances abeta accumulation in APP/PS1 mice brain and decreases amyloid-associated miRNAs expression.
    Liu H, Tian T, Qin S, Li W, Zhang X, Wang X, Gao Y, Huang G.
    J Nutr Biochem; 2015 Dec 15; 26(12):1502-8. PubMed ID: 26345540
    [Abstract] [Full Text] [Related]

  • 5. MicroRNA-574 is involved in cognitive impairment in 5-month-old APP/PS1 mice through regulation of neuritin.
    Li F, Wei G, Bai Y, Li Y, Huang F, Lin J, Hou Q, Deng R, Zhou JH, Zhang SX, Chen DF.
    Brain Res; 2015 Nov 19; 1627():177-88. PubMed ID: 26423933
    [Abstract] [Full Text] [Related]

  • 6. miR-34a knockout attenuates cognitive deficits in APP/PS1 mice through inhibition of the amyloidogenic processing of APP.
    Jian C, Lu M, Zhang Z, Liu L, Li X, Huang F, Xu N, Qin L, Zhang Q, Zou D.
    Life Sci; 2017 Aug 01; 182():104-111. PubMed ID: 28533191
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  • 7. Treadmill exercise enhances synaptic plasticity, but does not alter β-amyloid deposition in hippocampi of aged APP/PS1 transgenic mice.
    Zhao G, Liu HL, Zhang H, Tong XJ.
    Neuroscience; 2015 Jul 09; 298():357-66. PubMed ID: 25917310
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  • 15. GluN2B-containing NMDA receptors contribute to the beneficial effects of hydrogen sulfide on cognitive and synaptic plasticity deficits in APP/PS1 transgenic mice.
    Yang YJ, Zhao Y, Yu B, Xu GG, Wang W, Zhan JQ, Tang ZY, Wang T, Wei B.
    Neuroscience; 2016 Oct 29; 335():170-83. PubMed ID: 27581687
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  • 18. Identification of Circulating miR-125b as a Potential Biomarker of Alzheimer's Disease in APP/PS1 Transgenic Mouse.
    Hong H, Li Y, Su B.
    J Alzheimers Dis; 2017 Oct 29; 59(4):1449-1458. PubMed ID: 28731435
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  • 19. In vivo hippocampal microdialysis reveals impairment of NMDA receptor-cGMP signaling in APP(SW) and APP(SW)/PS1(L166P) Alzheimer's transgenic mice.
    Duszczyk M, Kuszczyk M, Guridi M, Lazarewicz JW, Sadowski MJ.
    Neurochem Int; 2012 Dec 29; 61(7):976-80. PubMed ID: 22841892
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  • 20. CC chemokine receptor 2 deficiency aggravates cognitive impairments and amyloid pathology in a transgenic mouse model of Alzheimer's disease.
    Naert G, Rivest S.
    J Neurosci; 2011 Apr 20; 31(16):6208-20. PubMed ID: 21508244
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