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Journal Abstract Search
181 related items for PubMed ID: 29489063
1. Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice. Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F. Acta Physiol (Oxf); 2018 Sep; 224(1):e13060. PubMed ID: 29489063 [Abstract] [Full Text] [Related]
2. Proteolytic activation of the epithelial sodium channel (ENaC) by factor VII activating protease (FSAP) and its relevance for sodium retention in nephrotic mice. Artunc F, Bohnert BN, Schneider JC, Staudner T, Sure F, Ilyaskin AV, Wörn M, Essigke D, Janessa A, Nielsen NV, Birkenfeld AL, Etscheid M, Haerteis S, Korbmacher C, Kanse SM. Pflugers Arch; 2022 Feb; 474(2):217-229. PubMed ID: 34870751 [Abstract] [Full Text] [Related]
3. Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome. Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F. Kidney Int; 2018 Jan; 93(1):159-172. PubMed ID: 29042083 [Abstract] [Full Text] [Related]
4. Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome. Bohnert BN, Daiminger S, Wörn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F. Acta Physiol (Oxf); 2019 Dec; 227(4):e13286. PubMed ID: 31006168 [Abstract] [Full Text] [Related]
5. Sodium retention in nephrotic syndrome is independent of the activation of the membrane-anchored serine protease prostasin (CAP1/PRSS8) and its enzymatic activity. Essigke D, Bohnert BN, Janessa A, Wörn M, Omage K, Kalbacher H, Birkenfeld AL, Bugge TH, Szabo R, Artunc F. Pflugers Arch; 2022 Jun; 474(6):613-624. PubMed ID: 35312839 [Abstract] [Full Text] [Related]
6. Experimental nephrotic syndrome leads to proteolytic activation of the epithelial Na+ channel in the mouse kidney. Bohnert BN, Essigke D, Janessa A, Schneider JC, Wörn M, Kalo MZ, Xiao M, Kong L, Omage K, Hennenlotter J, Amend B, Birkenfeld AL, Artunc F. Am J Physiol Renal Physiol; 2021 Oct 01; 321(4):F480-F493. PubMed ID: 34423678 [Abstract] [Full Text] [Related]
7. Tissue kallikrein activation of the epithelial Na channel. Patel AB, Chao J, Palmer LG. Am J Physiol Renal Physiol; 2012 Aug 15; 303(4):F540-50. PubMed ID: 22622459 [Abstract] [Full Text] [Related]
8. Urinary serine proteases and activation of ENaC in kidney--implications for physiological renal salt handling and hypertensive disorders with albuminuria. Svenningsen P, Andersen H, Nielsen LH, Jensen BL. Pflugers Arch; 2015 Mar 15; 467(3):531-42. PubMed ID: 25482671 [Abstract] [Full Text] [Related]
9. Proteasuria-The impact of active urinary proteases on sodium retention in nephrotic syndrome. Artunc F, Wörn M, Schork A, Bohnert BN. Acta Physiol (Oxf); 2019 Apr 15; 225(4):e13249. PubMed ID: 30597733 [Abstract] [Full Text] [Related]
10. Proteolytic activation of the human epithelial sodium channel by trypsin IV and trypsin I involves distinct cleavage sites. Haerteis S, Krappitz A, Krappitz M, Murphy JE, Bertog M, Krueger B, Nacken R, Chung H, Hollenberg MD, Knecht W, Bunnett NW, Korbmacher C. J Biol Chem; 2014 Jul 04; 289(27):19067-78. PubMed ID: 24841206 [Abstract] [Full Text] [Related]
11. Association of Plasminuria with Overhydration in Patients with CKD. Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Haerteis S, Korbmacher C, Heyne N, Peter A, Häring HU, Artunc F. Clin J Am Soc Nephrol; 2016 May 06; 11(5):761-769. PubMed ID: 26933188 [Abstract] [Full Text] [Related]
12. Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S. Haerteis S, Krappitz M, Bertog M, Krappitz A, Baraznenok V, Henderson I, Lindström E, Murphy JE, Bunnett NW, Korbmacher C. Pflugers Arch; 2012 Oct 06; 464(4):353-65. PubMed ID: 22864553 [Abstract] [Full Text] [Related]
14. Plasmin and chymotrypsin have distinct preferences for channel activating cleavage sites in the γ subunit of the human epithelial sodium channel. Haerteis S, Krappitz M, Diakov A, Krappitz A, Rauh R, Korbmacher C. J Gen Physiol; 2012 Oct 06; 140(4):375-89. PubMed ID: 22966015 [Abstract] [Full Text] [Related]
15. Cathepsin B increases ENaC activity leading to hypertension early in nephrotic syndrome. Larionov A, Dahlke E, Kunke M, Zanon Rodriguez L, Schiessl IM, Magnin JL, Kern U, Alli AA, Mollet G, Schilling O, Castrop H, Theilig F. J Cell Mol Med; 2019 Oct 06; 23(10):6543-6553. PubMed ID: 31368174 [Abstract] [Full Text] [Related]
17. Plasmin in nephrotic urine activates the epithelial sodium channel. Svenningsen P, Bistrup C, Friis UG, Bertog M, Haerteis S, Krueger B, Stubbe J, Jensen ON, Thiesson HC, Uhrenholt TR, Jespersen B, Jensen BL, Korbmacher C, Skøtt O. J Am Soc Nephrol; 2009 Feb 06; 20(2):299-310. PubMed ID: 19073825 [Abstract] [Full Text] [Related]
18. Plasminogen deficiency does not prevent sodium retention in a genetic mouse model of experimental nephrotic syndrome. Xiao M, Bohnert BN, Aypek H, Kretz O, Grahammer F, Aukschun U, Wörn M, Janessa A, Essigke D, Daniel C, Amann K, Huber TB, Plow EF, Birkenfeld AL, Artunc F. Acta Physiol (Oxf); 2021 Jan 06; 231(1):e13512. PubMed ID: 32455507 [Abstract] [Full Text] [Related]
19. Proteolytic Activity against the Distal Polybasic Tract of the Gamma Subunit of the Epithelial Sodium Channel ENaC in Nephrotic Urine. Wörn M, Kalbacher H, Artunc F. Curr Med Chem; 2022 Jan 06; 29(42):6433-6445. PubMed ID: 35676854 [Abstract] [Full Text] [Related]