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Journal Abstract Search

696 related items for PubMed ID: 29502344

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  • 4. Differential involvement of vesicular and glial glutamate transporters around spinal α-motoneurons in the pathogenesis of SOD1G93A mouse model of amyotrophic lateral sclerosis.
    Ohgomori T, Yamasaki R, Takeuchi H, Kadomatsu K, Kira JI, Jinno S.
    Neuroscience; 2017 Jul 25; 356():114-124. PubMed ID: 28526579
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  • 5. The role of mutated SOD1 gene in synaptic stripping and MHC class I expression following nerve axotomy in ALS murine model.
    Kassa RM, Bonafede R, Boschi F, Malatesta M, Mariotti R.
    Eur J Histochem; 2018 May 17; 62(2):2904. PubMed ID: 29943955
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  • 9. Dysregulation of persistent inward and outward currents in spinal motoneurons of symptomatic SOD1-G93A mice.
    Deutsch AJ, Elbasiouny SM.
    J Physiol; 2024 Aug 17; 602(15):3715-3736. PubMed ID: 38924530
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  • 10. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 17; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 11. Morphological changes and selective loss of motoneurons in the lumbar part of the spinal cord in a rat model of familial amyotrophic lateral sclerosis (fALS).
    Gadamski R, Chrapusta SJ, Wojda R, Grieb P.
    Folia Neuropathol; 2006 Dec 17; 44(3):154-61. PubMed ID: 17039410
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  • 12. Experimental Design and Data Analysis Issues Contribute to Inconsistent Results of C-Bouton Changes in Amyotrophic Lateral Sclerosis.
    Dukkipati SS, Chihi A, Wang Y, Elbasiouny SM.
    eNeuro; 2017 Dec 17; 4(1):. PubMed ID: 28101533
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  • 13. Excitatory action of low frequency depolarizing GABA/glycine synaptic inputs is prevalent in prenatal spinal SOD1G93A motoneurons.
    Zhu H, Dalvi U, Cazenave W, Cattaert D, Branchereau P.
    J Physiol; 2024 Mar 17; 602(5):913-932. PubMed ID: 38345477
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  • 14. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 17; 64():48-59. PubMed ID: 24361555
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  • 17. Cytotoxic CD8+ T lymphocytes expressing ALS-causing SOD1 mutant selectively trigger death of spinal motoneurons.
    Coque E, Salsac C, Espinosa-Carrasco G, Varga B, Degauque N, Cadoux M, Crabé R, Virenque A, Soulard C, Fierle JK, Brodovitch A, Libralato M, Végh AG, Venteo S, Scamps F, Boucraut J, Laplaud D, Hernandez J, Gergely C, Vincent T, Raoul C.
    Proc Natl Acad Sci U S A; 2019 Feb 05; 116(6):2312-2317. PubMed ID: 30674678
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  • 18. Cellular changes in motoneurons in a transgenic mouse model for amyotrophic lateral sclerosis as revealed by monoclonal antibody Py.
    Joosten EA, Van Westerlaak MG, Biesheuvel C, Woodhams PL, Brook GA, Veldman H, Bär PR.
    Brain Res Dev Brain Res; 2001 Nov 26; 131(1-2):153-9. PubMed ID: 11718846
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  • 19. Compensatory changes in degenerating spinal motoneurons sustain functional sparing in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.
    Giusto E, Codrich M, de Leo G, Francardo V, Coradazzi M, Parenti R, Gulisano M, Vicario N, Gulino R, Leanza G.
    J Comp Neurol; 2020 Feb 01; 528(2):231-243. PubMed ID: 31364764
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  • 20. Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice.
    Avossa D, Grandolfo M, Mazzarol F, Zatta M, Ballerini L.
    Neuroscience; 2006 Feb 01; 138(4):1179-94. PubMed ID: 16442737
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