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PUBMED FOR HANDHELDS

Journal Abstract Search


708 related items for PubMed ID: 29525626

  • 21. The oxidative potential of PM2.5 exposures from indoor and outdoor sources in rural China.
    Secrest MH, Schauer JJ, Carter EM, Lai AM, Wang Y, Shan M, Yang X, Zhang Y, Baumgartner J.
    Sci Total Environ; 2016 Nov 15; 571():1477-89. PubMed ID: 27443462
    [Abstract] [Full Text] [Related]

  • 22. In-vitro human lung cell injuries induced by urban PM2.5 during a severe air pollution episode: Variations associated with particle components.
    Pang Y, Huang W, Luo XS, Chen Q, Zhao Z, Tang M, Hong Y, Chen J, Li H.
    Ecotoxicol Environ Saf; 2020 Dec 15; 206():111406. PubMed ID: 33007542
    [Abstract] [Full Text] [Related]

  • 23. Ambient PM2.5-bound polycyclic aromatic hydrocarbons (PAHs) in rural Beijing: Unabated with enhanced temporary emission control during the 2014 APEC summit and largely aggravated after the start of wintertime heating.
    Yu Q, Yang W, Zhu M, Gao B, Li S, Li G, Fang H, Zhou H, Zhang H, Wu Z, Song W, Tan J, Zhang Y, Bi X, Chen L, Wang X.
    Environ Pollut; 2018 Jul 15; 238():532-542. PubMed ID: 29605613
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  • 24. PM2.5-induced alteration of DNA methylation and RNA-transcription are associated with inflammatory response and lung injury.
    Shi Y, Zhao T, Yang X, Sun B, Li Y, Duan J, Sun Z.
    Sci Total Environ; 2019 Feb 10; 650(Pt 1):908-921. PubMed ID: 30308865
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  • 25. The cellular effects of PM2.5 collected in Chinese Taiyuan and Guangzhou and their associations with polycyclic aromatic hydrocarbons (PAHs), nitro-PAHs and hydroxy-PAHs.
    Song Y, Zhang Y, Li R, Chen W, Chung CKA, Cai Z.
    Ecotoxicol Environ Saf; 2020 Mar 15; 191():110225. PubMed ID: 32001423
    [Abstract] [Full Text] [Related]

  • 26. PM2.5 exposure upregulates pro-inflammatory protein expression in human microglial cells via oxidant stress and TLR4/NF-κB pathway.
    Zhang L, Xu F, Yang Y, Yang L, Wu Q, Sun H, An Z, Li J, Wu H, Song J, Wu W.
    Ecotoxicol Environ Saf; 2024 Jun 01; 277():116386. PubMed ID: 38657455
    [Abstract] [Full Text] [Related]

  • 27. PM2.5 stimulated the release of cytokines from BEAS-2B cells through activation of IKK/NF-κB pathway.
    Wang J, Zhang WJ, Xiong W, Lu WH, Zheng HY, Zhou X, Yuan J.
    Hum Exp Toxicol; 2019 Mar 01; 38(3):311-320. PubMed ID: 30354488
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  • 28. Aloe vera downregulates LPS-induced inflammatory cytokine production and expression of NLRP3 inflammasome in human macrophages.
    Budai MM, Varga A, Milesz S, Tőzsér J, Benkő S.
    Mol Immunol; 2013 Dec 01; 56(4):471-9. PubMed ID: 23911403
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  • 29. Cumulative health risk assessment of halogenated and parent polycyclic aromatic hydrocarbons associated with particulate matters in urban air.
    Sun JL, Jing X, Chang WJ, Chen ZX, Zeng H.
    Ecotoxicol Environ Saf; 2015 Mar 01; 113():31-7. PubMed ID: 25483369
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  • 30. PM2.5 collected in China causes inflammatory and oxidative stress responses in macrophages through the multiple pathways.
    Bekki K, Ito T, Yoshida Y, He C, Arashidani K, He M, Sun G, Zeng Y, Sone H, Kunugita N, Ichinose T.
    Environ Toxicol Pharmacol; 2016 Jul 01; 45():362-9. PubMed ID: 27393915
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  • 31. Selenium-enriched soybean peptides pretreatment attenuates lung injury in mice induced by fine particulate matters (PM2.5) through inhibition of TLR4/NF-κB/IκBα signaling pathway and inflammasome generation.
    Zhang J, Chen X, Li H, Liu W, Liu X, Song Y, Cong X.
    Food Funct; 2022 Sep 22; 13(18):9459-9469. PubMed ID: 35979800
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  • 32. Particulate matters induce acute exacerbation of allergic airway inflammation via the TLR2/NF-κB/NLRP3 signaling pathway.
    Dai MY, Chen FF, Wang Y, Wang MZ, Lv YX, Liu RY.
    Toxicol Lett; 2020 Mar 15; 321():146-154. PubMed ID: 31836503
    [Abstract] [Full Text] [Related]

  • 33. PM2.5-induced oxidative stress increases intercellular adhesion molecule-1 expression in lung epithelial cells through the IL-6/AKT/STAT3/NF-κB-dependent pathway.
    Liu CW, Lee TL, Chen YC, Liang CJ, Wang SH, Lue JH, Tsai JS, Lee SW, Chen SH, Yang YF, Chuang TY, Chen YL.
    Part Fibre Toxicol; 2018 Jan 12; 15(1):4. PubMed ID: 29329563
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  • 34. PM2.5-induced pulmonary inflammation via activating of the NLRP3/caspase-1 signaling pathway.
    Jia H, Liu Y, Guo D, He W, Zhao L, Xia S.
    Environ Toxicol; 2021 Mar 12; 36(3):298-307. PubMed ID: 32996690
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  • 35. Hydrophobic Organic Components of Ambient Fine Particulate Matter (PM2.5) Associated with Inflammatory Cellular Response.
    Jiang X, Xu F, Qiu X, Shi X, Pardo M, Shang Y, Wang J, Rudich Y, Zhu T.
    Environ Sci Technol; 2019 Sep 03; 53(17):10479-10486. PubMed ID: 31397158
    [Abstract] [Full Text] [Related]

  • 36. Contributions of City-Specific Fine Particulate Matter (PM2.5) to Differential In Vitro Oxidative Stress and Toxicity Implications between Beijing and Guangzhou of China.
    Jin L, Xie J, Wong CKC, Chan SKY, Abbaszade G, Schnelle-Kreis J, Zimmermann R, Li J, Zhang G, Fu P, Li X.
    Environ Sci Technol; 2019 Mar 05; 53(5):2881-2891. PubMed ID: 30730710
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  • 37. Molecular mechanisms underlying NLRP3 inflammasome activation and IL-1β production in air pollution fine particulate matter (PM2.5)-primed macrophages.
    Caceres L, Abogunloko T, Malchow S, Ehret F, Merz J, Li X, Sol Mitre L, Magnani N, Tasat D, Mwinyella T, Spiga L, Suchanek D, Fischer L, Gorka O, Colin Gissler M, Hilgendorf I, Stachon P, Rog-Zielinska E, Groß O, Westermann D, Evelson P, Wolf D, Marchini T.
    Environ Pollut; 2024 Jan 15; 341():122997. PubMed ID: 38000727
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  • 38. NLRP3 inflammasome activation determines the fibrogenic potential of PM2.5 air pollution particles in the lung.
    Cao W, Wang X, Li J, Yan M, Chang CH, Kim J, Jiang J, Liao YP, Tseng S, Kusumoputro S, Lau C, Huang M, Han P, Lu P, Xia T.
    J Environ Sci (China); 2022 Jan 15; 111():429-441. PubMed ID: 34949371
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  • 39. Epidermal growth factor receptor (EGFR)-MAPK-nuclear factor(NF)-κB-IL8: A possible mechanism of particulate matter(PM) 2.5-induced lung toxicity.
    Jeong SC, Cho Y, Song MK, Lee E, Ryu JC.
    Environ Toxicol; 2017 May 15; 32(5):1628-1636. PubMed ID: 28101945
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  • 40. Early-life exposure to three size-fractionated ultrafine and fine atmospheric particulates in Beijing exacerbates asthma development in mature mice.
    Mei M, Song H, Chen L, Hu B, Bai R, Xu D, Liu Y, Zhao Y, Chen C.
    Part Fibre Toxicol; 2018 Mar 14; 15(1):13. PubMed ID: 29540228
    [Abstract] [Full Text] [Related]


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