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Journal Abstract Search

238 related items for PubMed ID: 29572097

  • 1. Hippocampal microvasculature changes in association with oxidative stress in Alzheimer's disease.
    Sen A, Hongpaisan J.
    Free Radic Biol Med; 2018 May 20; 120():192-203. PubMed ID: 29572097
    [Abstract] [Full Text] [Related]

  • 2. Loss in PKC Epsilon Causes Downregulation of MnSOD and BDNF Expression in Neurons of Alzheimer's Disease Hippocampus.
    Sen A, Nelson TJ, Alkon DL, Hongpaisan J.
    J Alzheimers Dis; 2018 May 20; 63(3):1173-1189. PubMed ID: 29710707
    [Abstract] [Full Text] [Related]

  • 3. MicroRNA-98 reduces amyloid β-protein production and improves oxidative stress and mitochondrial dysfunction through the Notch signaling pathway via HEY2 in Alzheimer's disease mice.
    Chen FZ, Zhao Y, Chen HZ.
    Int J Mol Med; 2019 Jan 20; 43(1):91-102. PubMed ID: 30365070
    [Abstract] [Full Text] [Related]

  • 4. Mitochondria and vascular lesions as a central target for the development of Alzheimer's disease and Alzheimer disease-like pathology in transgenic mice.
    Aliev G, Seyidova D, Lamb BT, Obrenovich ME, Siedlak SL, Vinters HV, Friedland RP, LaManna JC, Smith MA, Perry G.
    Neurol Res; 2003 Sep 20; 25(6):665-74. PubMed ID: 14503022
    [Abstract] [Full Text] [Related]

  • 5. Relationship between ubiquilin-1 and BACE1 in human Alzheimer's disease and APdE9 transgenic mouse brain and cell-based models.
    Natunen T, Takalo M, Kemppainen S, Leskelä S, Marttinen M, Kurkinen KMA, Pursiheimo JP, Sarajärvi T, Viswanathan J, Gabbouj S, Solje E, Tahvanainen E, Pirttimäki T, Kurki M, Paananen J, Rauramaa T, Miettinen P, Mäkinen P, Leinonen V, Soininen H, Airenne K, Tanzi RE, Tanila H, Haapasalo A, Hiltunen M.
    Neurobiol Dis; 2016 Jan 20; 85():187-205. PubMed ID: 26563932
    [Abstract] [Full Text] [Related]

  • 6. PKCε deficits in Alzheimer's disease brains and skin fibroblasts.
    Khan TK, Sen A, Hongpaisan J, Lim CS, Nelson TJ, Alkon DL.
    J Alzheimers Dis; 2015 Jan 20; 43(2):491-509. PubMed ID: 25125477
    [Abstract] [Full Text] [Related]

  • 7. Reduction of Dendritic Inhibition in CA1 Pyramidal Neurons in Amyloidosis Models of Early Alzheimer's Disease.
    Ruiter M, Herstel LJ, Wierenga CJ.
    J Alzheimers Dis; 2020 Jan 20; 78(3):951-964. PubMed ID: 33074225
    [Abstract] [Full Text] [Related]

  • 8. α-secretase cleaved amyloid precursor protein (APP) accumulates in cholinergic dystrophic neurites in normal, aged hippocampus.
    Yoon SY, Choi JU, Cho MH, Yang KM, Ha H, Chung IJ, Cho GS, Kim DH.
    Neuropathol Appl Neurobiol; 2013 Dec 20; 39(7):800-16. PubMed ID: 23414335
    [Abstract] [Full Text] [Related]

  • 9. Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression.
    Manczak M, Anekonda TS, Henson E, Park BS, Quinn J, Reddy PH.
    Hum Mol Genet; 2006 May 01; 15(9):1437-49. PubMed ID: 16551656
    [Abstract] [Full Text] [Related]

  • 10. Association of Apolipoprotein E4-related Microvascular Disease in the Alzheimer's Disease Hippocampal CA1 Stratum Radiatum.
    Wang H, Zhang Z, Sittirattanayeunyong S, Hongpaisan J.
    Neuroscience; 2023 Aug 21; 526():204-222. PubMed ID: 37385335
    [Abstract] [Full Text] [Related]

  • 11. APP/Go protein Gβγ-complex signaling mediates Aβ degeneration and cognitive impairment in Alzheimer's disease models.
    Bignante EA, Ponce NE, Heredia F, Musso J, Krawczyk MC, Millán J, Pigino GF, Inestrosa NC, Boccia MM, Lorenzo A.
    Neurobiol Aging; 2018 Apr 21; 64():44-57. PubMed ID: 29331876
    [Abstract] [Full Text] [Related]

  • 12. Oxidative balance in Alzheimer's disease: relationship to APOE, Braak tangle stage, and the concentrations of soluble and insoluble amyloid-β.
    Tayler H, Fraser T, Miners JS, Kehoe PG, Love S.
    J Alzheimers Dis; 2010 Apr 21; 22(4):1363-73. PubMed ID: 20930272
    [Abstract] [Full Text] [Related]

  • 13. Low-level laser therapy for beta amyloid toxicity in rat hippocampus.
    Lu Y, Wang R, Dong Y, Tucker D, Zhao N, Ahmed ME, Zhu L, Liu TC, Cohen RM, Zhang Q.
    Neurobiol Aging; 2017 Jan 21; 49():165-182. PubMed ID: 27815990
    [Abstract] [Full Text] [Related]

  • 14. Alzheimer's disease.
    De-Paula VJ, Radanovic M, Diniz BS, Forlenza OV.
    Subcell Biochem; 2012 Jan 21; 65():329-52. PubMed ID: 23225010
    [Abstract] [Full Text] [Related]

  • 15. Exposure to As-, Cd-, and Pb-mixture induces Aβ, amyloidogenic APP processing and cognitive impairments via oxidative stress-dependent neuroinflammation in young rats.
    Ashok A, Rai NK, Tripathi S, Bandyopadhyay S.
    Toxicol Sci; 2015 Jan 21; 143(1):64-80. PubMed ID: 25288670
    [Abstract] [Full Text] [Related]

  • 16. Microdose Lithium NP03 Diminishes Pre-Plaque Oxidative Damage and Neuroinflammation in a Rat Model of Alzheimer's-like Amyloidosis.
    Wilson EN, Do Carmo S, Iulita MF, Hall H, Austin GL, Jia DT, Malcolm JC, Foret MK, Marks AR, Butterfield DA, Cuello AC.
    Curr Alzheimer Res; 2018 Jan 21; 15(13):1220-1230. PubMed ID: 30182855
    [Abstract] [Full Text] [Related]

  • 17. The effect of ageing on neurogenesis and oxidative stress in the APP(swe)/PS1(deltaE9) mouse model of Alzheimer's disease.
    Hamilton A, Holscher C.
    Brain Res; 2012 Apr 17; 1449():83-93. PubMed ID: 22418058
    [Abstract] [Full Text] [Related]

  • 18. Dexamethasone and Aβ₂₅-₃₅ accelerate learning and memory impairments due to elevate amyloid precursor protein expression and neuronal apoptosis in 12-month male rats.
    Li WZ, Li WP, Huang DK, Kan HW, Wang X, Wu WY, Yin YY, Yao YY.
    Behav Brain Res; 2012 Feb 01; 227(1):142-9. PubMed ID: 22061800
    [Abstract] [Full Text] [Related]

  • 19. Antisense directed at the Abeta region of APP decreases brain oxidative markers in aged senescence accelerated mice.
    Poon HF, Joshi G, Sultana R, Farr SA, Banks WA, Morley JE, Calabrese V, Butterfield DA.
    Brain Res; 2004 Aug 20; 1018(1):86-96. PubMed ID: 15262209
    [Abstract] [Full Text] [Related]

  • 20. Alzheimer's disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression.
    Audrain M, Fol R, Dutar P, Potier B, Billard JM, Flament J, Alves S, Burlot MA, Dufayet-Chaffaud G, Bemelmans AP, Valette J, Hantraye P, Déglon N, Cartier N, Braudeau J.
    Mol Neurodegener; 2016 Jan 12; 11():5. PubMed ID: 26759118
    [Abstract] [Full Text] [Related]

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