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Journal Abstract Search


138 related items for PubMed ID: 29642351

  • 1. Re: Plasma Oxalate in Relation to eGFR in Patients with Primary Hyperoxaluria, Enteric Hyperoxaluria and Urinary Stone Disease.
    Assimos DG.
    J Urol; 2018 Apr; 199(4):887. PubMed ID: 29642351
    [No Abstract] [Full Text] [Related]

  • 2. Plasma oxalate in relation to eGFR in patients with primary hyperoxaluria, enteric hyperoxaluria and urinary stone disease.
    Perinpam M, Enders FT, Mara KC, Vaughan LE, Mehta RA, Voskoboev N, Milliner DS, Lieske JC.
    Clin Biochem; 2017 Dec; 50(18):1014-1019. PubMed ID: 28764885
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  • 8. Direct correlation between hyperoxaluria/oxalate stone disease and the absence of the gastrointestinal tract-dwelling bacterium Oxalobacter formigenes: possible prevention by gut recolonization or enzyme replacement therapy.
    Sidhu H, Schmidt ME, Cornelius JG, Thamilselvan S, Khan SR, Hesse A, Peck AB.
    J Am Soc Nephrol; 1999 Nov; 10 Suppl 14():S334-40. PubMed ID: 10541258
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  • 9. Enteric hyperoxaluria secondary to small bowel resection: use of computer simulation to characterize urinary risk factors for stone formation and assess potential treatment protocols.
    Rodgers AL, Allie-Hamdulay S, Jackson GE, Sutton RA.
    J Endourol; 2014 Aug; 28(8):985-94. PubMed ID: 24773381
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  • 11. Primary hyperoxaluria.
    ARCHER HE, DORMER AE, SCOWEN EF, WATTS RW.
    Lancet; 1957 Aug 17; 273(6990):320-2. PubMed ID: 13464052
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  • 12. Efficacy and safety of Oxalobacter formigenes to reduce urinary oxalate in primary hyperoxaluria.
    Hoppe B, Groothoff JW, Hulton SA, Cochat P, Niaudet P, Kemper MJ, Deschênes G, Unwin R, Milliner D.
    Nephrol Dial Transplant; 2011 Nov 17; 26(11):3609-15. PubMed ID: 21460356
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  • 13. Absorptive hyperoxaluria leads to an increased risk for urolithiasis or nephrocalcinosis in cystic fibrosis.
    Hoppe B, von Unruh GE, Blank G, Rietschel E, Sidhu H, Laube N, Hesse A.
    Am J Kidney Dis; 2005 Sep 17; 46(3):440-5. PubMed ID: 16129205
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  • 14. Effect of dietary changes on urinary oxalate excretion and calcium oxalate supersaturation in patients with hyperoxaluric stone formation.
    Penniston KL, Nakada SY.
    Urology; 2009 Mar 17; 73(3):484-9. PubMed ID: 19118878
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  • 15. Potential mechanisms of marked hyperoxaluria not due to primary hyperoxaluria I or II.
    Monico CG, Persson M, Ford GC, Rumsby G, Milliner DS.
    Kidney Int; 2002 Aug 17; 62(2):392-400. PubMed ID: 12110000
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  • 16. Pyridoxamine lowers oxalate excretion and kidney crystals in experimental hyperoxaluria: a potential therapy for primary hyperoxaluria.
    Scheinman JI, Voziyan PA, Belmont JM, Chetyrkin SV, Kim D, Hudson BG.
    Urol Res; 2005 Nov 17; 33(5):368-71. PubMed ID: 16292584
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  • 17. Dietary risk factors for hyperoxaluria in calcium oxalate stone formers.
    Siener R, Ebert D, Nicolay C, Hesse A.
    Kidney Int; 2003 Mar 17; 63(3):1037-43. PubMed ID: 12631085
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  • 18. Atypical features of primary hyperoxaluria in end-stage renal disease.
    Absy MM.
    Am J Nephrol; 1991 Mar 17; 11(4):301-4. PubMed ID: 1799188
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  • 19. [Oxalate: a poorly soluble organic waste with consequences].
    Lu Y, Bonny O.
    Praxis (Bern 1994); 2015 Mar 25; 104(7):353-9. PubMed ID: 25804778
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  • 20. [24-hour urinary oxalate excretion in healthy children and in children with calcium urolithiasis].
    Sikora P, Bieniaś B, Wawrzyszuk M, Zajaczkowska M.
    Pol Merkur Lekarski; 2008 Mar 25; 24 Suppl 4():76-9. PubMed ID: 18924510
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