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Journal Abstract Search

737 related items for PubMed ID: 29699580

  • 1. Genetic deletion of muscle RANK or selective inhibition of RANKL is not as effective as full-length OPG-fc in mitigating muscular dystrophy.
    Dufresne SS, Boulanger-Piette A, Bossé S, Argaw A, Hamoudi D, Marcadet L, Gamu D, Fajardo VA, Yagita H, Penninger JM, Russell Tupling A, Frenette J.
    Acta Neuropathol Commun; 2018 Apr 24; 6(1):31. PubMed ID: 29699580
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  • 2. An anti-RANKL treatment reduces muscle inflammation and dysfunction and strengthens bone in dystrophic mice.
    Hamoudi D, Marcadet L, Piette Boulanger A, Yagita H, Bouredji Z, Argaw A, Frenette J.
    Hum Mol Genet; 2019 Sep 15; 28(18):3101-3112. PubMed ID: 31179501
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  • 3. Neopterin/7,8-dihydroneopterin is elevated in Duchenne muscular dystrophy patients and protects mdx skeletal muscle function.
    Lindsay A, Schmiechen A, Chamberlain CM, Ervasti JM, Lowe DA.
    Exp Physiol; 2018 Jul 15; 103(7):995-1009. PubMed ID: 29791760
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  • 4. Alterations in Notch signalling in skeletal muscles from mdx and dko dystrophic mice and patients with Duchenne muscular dystrophy.
    Church JE, Trieu J, Chee A, Naim T, Gehrig SM, Lamon S, Angelini C, Russell AP, Lynch GS.
    Exp Physiol; 2014 Apr 15; 99(4):675-87. PubMed ID: 24443351
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  • 5. Osteoprotegerin protects against muscular dystrophy.
    Dufresne SS, Dumont NA, Bouchard P, Lavergne É, Penninger JM, Frenette J.
    Am J Pathol; 2015 Apr 15; 185(4):920-6. PubMed ID: 25708645
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  • 8. Physiological role of receptor activator nuclear factor-kB (RANK) in denervation-induced muscle atrophy and dysfunction.
    Dufresne SS, Boulanger-Piette A, Bossé S, Frenette J.
    Receptors Clin Investig; 2016 May 30; 3(2):e13231-e13236. PubMed ID: 27547781
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  • 10. Muscle specific kinase protects dystrophic mdx mouse muscles from eccentric contraction-induced loss of force-producing capacity.
    Trajanovska S, Ban J, Huang J, Gregorevic P, Morsch M, Allen DG, Phillips WD.
    J Physiol; 2019 Sep 30; 597(18):4831-4850. PubMed ID: 31340406
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  • 13. Anti-RANKL Therapy Prevents Glucocorticoid-Induced Bone Loss and Promotes Muscle Function in a Mouse Model of Duchenne Muscular Dystrophy.
    Jayash SN, Hamoudi D, Stephen LA, Argaw A, Huesa C, Joseph S, Wong SC, Frenette J, Farquharson C.
    Calcif Tissue Int; 2023 Oct 30; 113(4):449-468. PubMed ID: 37470794
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  • 14. RANKL signaling drives skeletal muscle into the oxidative profile.
    Cavalcanti de Araújo PH, Cezine MER, Vulczak A, Vieira LC, Matsuo FS, Remoto JM, Santos ADR, Miyabara EH, Alberici LC, Osako MK.
    J Bone Miner Res; 2024 Jul 23; 39(6):753-764. PubMed ID: 38619281
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  • 15. Osteoprotegerin and β2-Agonists Mitigate Muscular Dystrophy in Slow- and Fast-Twitch Skeletal Muscles.
    Dufresne SS, Boulanger-Piette A, Frenette J.
    Am J Pathol; 2017 Mar 23; 187(3):498-504. PubMed ID: 28041995
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  • 18. Targeting the Muscle-Bone Unit: Filling Two Needs with One Deed in the Treatment of Duchenne Muscular Dystrophy.
    Boulanger Piette A, Hamoudi D, Marcadet L, Morin F, Argaw A, Ward L, Frenette J.
    Curr Osteoporos Rep; 2018 Oct 23; 16(5):541-553. PubMed ID: 30225627
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  • 19. An octaguanidine-morpholino oligo conjugate improves muscle function of mdx mice.
    Widrick JJ, Jiang S, Choi SJ, Knuth ST, Morcos PA.
    Muscle Nerve; 2011 Oct 23; 44(4):563-70. PubMed ID: 21922468
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  • 20. Hypochlorous acid exposure impairs skeletal muscle function and Ca2+ signalling: implications for Duchenne muscular dystrophy pathology.
    Lea TA, Panizza PM, Arthur PG, Bakker AJ, Pinniger GJ.
    J Physiol; 2023 Dec 23; 601(23):5257-5275. PubMed ID: 37864413
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