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PUBMED FOR HANDHELDS

Journal Abstract Search


219 related items for PubMed ID: 29709016

  • 1. EBV persistence without its EBNA3A and 3C oncogenes in vivo.
    Murer A, McHugh D, Caduff N, Kalchschmidt J, Barros M, Zbinden A, Capaul R, Niedobitek G, Allday M, Chijioke O, Münz C.
    PLoS Pathog; 2018 Apr; 14(4):e1007039. PubMed ID: 29709016
    [Abstract] [Full Text] [Related]

  • 2. Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV.
    Bazot Q, Paschos K, Allday MJ.
    J Virol; 2018 Apr 01; 92(7):. PubMed ID: 29367247
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  • 3. An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse Model.
    Romero-Masters JC, Ohashi M, Djavadian R, Eichelberg MR, Hayes M, Zumwalde NA, Bristol JA, Nelson SE, Ma S, Ranheim EA, Gumperz JE, Johannsen EC, Kenney SC.
    J Virol; 2020 May 04; 94(10):. PubMed ID: 32132242
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  • 4. Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites.
    Wang A, Welch R, Zhao B, Ta T, Keleş S, Johannsen E.
    J Virol; 2015 Dec 30; 90(6):2906-19. PubMed ID: 26719268
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  • 5. Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes.
    Schmidt SC, Jiang S, Zhou H, Willox B, Holthaus AM, Kharchenko PV, Johannsen EC, Kieff E, Zhao B.
    Proc Natl Acad Sci U S A; 2015 Jan 13; 112(2):554-9. PubMed ID: 25540416
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  • 6. First Days in the Life of Naive Human B Lymphocytes Infected with Epstein-Barr Virus.
    Pich D, Mrozek-Gorska P, Bouvet M, Sugimoto A, Akidil E, Grundhoff A, Hamperl S, Ling PD, Hammerschmidt W.
    mBio; 2019 Sep 17; 10(5):. PubMed ID: 31530670
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  • 7. An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model.
    Romero-Masters JC, Ohashi M, Djavadian R, Eichelberg MR, Hayes M, Bristol JA, Ma S, Ranheim EA, Gumperz J, Johannsen EC, Kenney SC.
    PLoS Pathog; 2018 Aug 17; 14(8):e1007221. PubMed ID: 30125329
    [Abstract] [Full Text] [Related]

  • 8. EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.
    Styles CT, Bazot Q, Parker GA, White RE, Paschos K, Allday MJ.
    PLoS Biol; 2017 Aug 17; 15(8):e2001992. PubMed ID: 28771465
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  • 10. Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP.
    Skalska L, White RE, Franz M, Ruhmann M, Allday MJ.
    PLoS Pathog; 2010 Jun 10; 6(6):e1000951. PubMed ID: 20548956
    [Abstract] [Full Text] [Related]

  • 11. The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells.
    Allday MJ, Bazot Q, White RE.
    Curr Top Microbiol Immunol; 2015 Jun 10; 391():61-117. PubMed ID: 26428372
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  • 15. Epstein-Barr Virus Nuclear Antigen 3C Facilitates Cell Proliferation by Regulating Cyclin D2.
    Pei Y, Singh RK, Shukla SK, Lang F, Zhang S, Robertson ES.
    J Virol; 2018 Sep 15; 92(18):. PubMed ID: 29997218
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  • 18. Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2.
    Bazot Q, Paschos K, Skalska L, Kalchschmidt JS, Parker GA, Allday MJ.
    PLoS Pathog; 2015 Jul 15; 11(7):e1005031. PubMed ID: 26153983
    [Abstract] [Full Text] [Related]

  • 19. Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9.
    Harth-Hertle ML, Scholz BA, Erhard F, Glaser LV, Dölken L, Zimmer R, Kempkes B.
    PLoS Pathog; 2013 Sep 15; 9(9):e1003638. PubMed ID: 24068939
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  • 20. Experimental infection of NOD/SCID mice reconstituted with human CD34+ cells with Epstein-Barr virus.
    Islas-Ohlmayer M, Padgett-Thomas A, Domiati-Saad R, Melkus MW, Cravens PD, Martin Mdel P, Netto G, Garcia JV.
    J Virol; 2004 Dec 15; 78(24):13891-900. PubMed ID: 15564497
    [Abstract] [Full Text] [Related]


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