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Journal Abstract Search


340 related items for PubMed ID: 2981158

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  • 2. Microsomal hydroxylation of 2- and 4-fluoroestradiol to catechol metabolites and their conversion to methyl ethers: catechol estrogens as possible mediators of hormonal carcinogenesis.
    Ashburn SP, Han X, Liehr JG.
    Mol Pharmacol; 1993 Apr; 43(4):534-41. PubMed ID: 8386306
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  • 7. Quercetin increases the severity of estradiol-induced tumorigenesis in hamster kidney.
    Zhu BT, Liehr JG.
    Toxicol Appl Pharmacol; 1994 Mar; 125(1):149-58. PubMed ID: 8128490
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  • 8. Estrogen metabolism in microsomal, cell, and tissue preparations of kidney and liver from Syrian hamsters.
    Beleh MA, Lin YC, Brueggemeier RW.
    J Steroid Biochem Mol Biol; 1995 May; 52(5):479-89. PubMed ID: 7748813
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  • 9. Catechol formation of fluoro- and bromo-substituted estradiols by hamster liver microsomes. Evidence for dehalogenation.
    Li JJ, Purdy RH, Appelman EH, Klicka JK, Li SA.
    Mol Pharmacol; 1985 May; 27(5):559-65. PubMed ID: 2985951
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  • 11. 17 beta-Estradiol metabolism by hamster hepatic microsomes. Implications for the catechol-O-methyl transferase-mediated detoxication of catechol estrogens.
    Butterworth M, Lau SS, Monks TJ.
    Drug Metab Dispos; 1996 May; 24(5):588-94. PubMed ID: 8723741
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  • 12. Estrogen carcinogenesis in Syrian hamster tissues: role of metabolism.
    Li JJ, Li SA.
    Fed Proc; 1987 Apr; 46(5):1858-63. PubMed ID: 3030825
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  • 13. Conversion of estrone to 2- and 4-hydroxyestrone by hamster kidney and liver microsomes: implications for the mechanism of estrogen-induced carcinogenesis.
    Zhu BT, Bui QD, Weisz J, Liehr JG.
    Endocrinology; 1994 Nov; 135(5):1772-9. PubMed ID: 7956900
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  • 14. Estrogen metabolism in primary kidney cell cultures from Syrian hamsters.
    Brueggemeier RW, Tseng K, Katlic NE, Beleh MA, Lin YC.
    J Steroid Biochem; 1990 Jul 04; 36(4):325-31. PubMed ID: 2391962
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  • 15. Metabolic deglucuronidation and demethylation of estrogen conjugates as a source of parent estrogens and catecholestrogen metabolites in Syrian hamster kidney, a target organ of estrogen-induced tumorigenesis.
    Zhu BT, Evaristus EN, Antoniak SK, Sarabia SF, Ricci MJ, Liehr JG.
    Toxicol Appl Pharmacol; 1996 Jan 04; 136(1):186-93. PubMed ID: 8560473
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  • 16. Elevated 4-hydroxylation of estradiol by hamster kidney microsomes: a potential pathway of metabolic activation of estrogens.
    Weisz J, Bui QD, Roy D, Liehr JG.
    Endocrinology; 1992 Aug 04; 131(2):655-61. PubMed ID: 1386303
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  • 17. 17 beta-estradiol metabolism by hamster hepatic microsomes: comparison of catechol estrogen O-methylation with catechol estrogen oxidation and glutathione conjugation.
    Butterworth M, Lau SS, Monks TJ.
    Chem Res Toxicol; 1996 Jun 04; 9(4):793-9. PubMed ID: 8831825
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  • 18. Localization of estrogen-induced DNA adducts and cytochrome P-450 activity at the site of renal carcinogenesis in the hamster kidney.
    Liehr JG, Hall ER, Avitts TA, Randerath E, Randerath K.
    Cancer Res; 1987 Apr 15; 47(8):2156-9. PubMed ID: 3103911
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  • 19. The carcinogenic activity of ethinyl estrogens is determined by both their hormonal characteristics and their conversion to catechol metabolites.
    Zhu BT, Roy D, Liehr JG.
    Endocrinology; 1993 Feb 15; 132(2):577-83. PubMed ID: 8381068
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