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Journal Abstract Search

314 related items for PubMed ID: 29843805

  • 1. Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.
    Mitsui S, Otomo A, Nozaki M, Ono S, Sato K, Shirakawa R, Adachi H, Aoki M, Sobue G, Shang HF, Hadano S.
    Mol Brain; 2018 May 29; 11(1):30. PubMed ID: 29843805
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  • 2. Functional links between SQSTM1 and ALS2 in the pathogenesis of ALS: cumulative impact on the protection against mutant SOD1-mediated motor dysfunction in mice.
    Hadano S, Mitsui S, Pan L, Otomo A, Kubo M, Sato K, Ono S, Onodera W, Abe K, Chen X, Koike M, Uchiyama Y, Aoki M, Warabi E, Yamamoto M, Ishii T, Yanagawa T, Shang HF, Yoshii F.
    Hum Mol Genet; 2016 Aug 01; 25(15):3321-3340. PubMed ID: 27439389
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  • 4. Loss of glial fibrillary acidic protein marginally accelerates disease progression in a SOD1(H46R) transgenic mouse model of ALS.
    Yoshii Y, Otomo A, Pan L, Ohtsuka M, Hadano S.
    Neurosci Res; 2011 Jul 01; 70(3):321-9. PubMed ID: 21453731
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  • 6. ALS-FTLD-linked mutations of SQSTM1/p62 disrupt selective autophagy and NFE2L2/NRF2 anti-oxidative stress pathway.
    Deng Z, Lim J, Wang Q, Purtell K, Wu S, Palomo GM, Tan H, Manfredi G, Zhao Y, Peng J, Hu B, Chen S, Yue Z.
    Autophagy; 2020 May 01; 16(5):917-931. PubMed ID: 31362587
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  • 7. Heat shock factor 1 over-expression protects against exposure of hydrophobic residues on mutant SOD1 and early mortality in a mouse model of amyotrophic lateral sclerosis.
    Lin PY, Simon SM, Koh WK, Folorunso O, Umbaugh CS, Pierce A.
    Mol Neurodegener; 2013 Nov 21; 8():43. PubMed ID: 24256636
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  • 8. A cysteine residue affects the conformational state and neuronal toxicity of mutant SOD1 in mice: relevance to the pathogenesis of ALS.
    Nagano S, Takahashi Y, Yamamoto K, Masutani H, Fujiwara N, Urushitani M, Araki T.
    Hum Mol Genet; 2015 Jun 15; 24(12):3427-39. PubMed ID: 25762155
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  • 10. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS, Shim YM, Lee DY, Kim JS, Kang M, Ahn SH, Shin JY, Geum D, Hong YH, Sung JJ.
    Mol Neurobiol; 2019 Mar 15; 56(3):2007-2021. PubMed ID: 29982983
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  • 12. In-vivo effects of knocking-down metabotropic glutamate receptor 5 in the SOD1G93A mouse model of amyotrophic lateral sclerosis.
    Bonifacino T, Cattaneo L, Gallia E, Puliti A, Melone M, Provenzano F, Bossi S, Musante I, Usai C, Conti F, Bonanno G, Milanese M.
    Neuropharmacology; 2017 Sep 01; 123():433-445. PubMed ID: 28645622
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  • 13. Trehalose delays the progression of amyotrophic lateral sclerosis by enhancing autophagy in motoneurons.
    Castillo K, Nassif M, Valenzuela V, Rojas F, Matus S, Mercado G, Court FA, van Zundert B, Hetz C.
    Autophagy; 2013 Sep 01; 9(9):1308-20. PubMed ID: 23851366
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  • 17. Rapamycin treatment augments motor neuron degeneration in SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Zhang X, Li L, Chen S, Yang D, Wang Y, Zhang X, Wang Z, Le W.
    Autophagy; 2011 Apr 01; 7(4):412-25. PubMed ID: 21193837
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  • 18. Microglia RAGE exacerbates the progression of neurodegeneration within the SOD1G93A murine model of amyotrophic lateral sclerosis in a sex-dependent manner.
    MacLean M, Juranek J, Cuddapah S, López-Díez R, Ruiz HH, Hu J, Frye L, Li H, Gugger PF, Schmidt AM.
    J Neuroinflammation; 2021 Jun 15; 18(1):139. PubMed ID: 34130712
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  • 19. Different human copper-zinc superoxide dismutase mutants, SOD1G93A and SOD1H46R, exert distinct harmful effects on gross phenotype in mice.
    Pan L, Yoshii Y, Otomo A, Ogawa H, Iwasaki Y, Shang HF, Hadano S.
    PLoS One; 2012 Jun 15; 7(3):e33409. PubMed ID: 22438926
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  • 20. Actions of the antihistaminergic clemastine on presymptomatic SOD1-G93A mice ameliorate ALS disease progression.
    Apolloni S, Fabbrizio P, Amadio S, Volonté C.
    J Neuroinflammation; 2016 Aug 22; 13(1):191. PubMed ID: 27549088
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